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Eosinophilic cell pneumonia _83490 by heku


									?Eosinophilic cell pneumonia


Eosinophilic cell pneumonia is a set of clear or has not been clear etiology
to Eosinophilic Infiltration to the characteristics, often accompanied by
peripheral blood eosinophils diseases. Sometimes called Eosinophilic cells
increase pulmonary infiltrates (PIE) syndrome. Wenzhou medical college
affiliated hospital pediatric respiratory medicine Sue Miller Award

Etiology and pathogenesis

Etiology including parasitic (such as ascariasis, bow Bronchioles and silk
worms), medications (e.g. penicillin, PAS, hydrazine benzene pyridazine,
nitrofurantoin, chlorpromazine, sulfanilamide preparations); chemical
allergy substances (such as steam inhalation of Nickel carbide form); and
fungi (Aspergillus fumigatus, causing allergic bronchopulmonary
aspergillosis). Although the suspected allergic mechanisms, but the majority
of Eosinophilic cells pneumonia etiology. Eosinophilic cells growing tips
for type I allergic reaction, other features of the syndrome (Vasculitis, round
cell infiltration) prompts for the type III may also be a section reaction.

Eosinophilic cell pneumonia often merge with bronchial asthma. Merge
asthma and etiology of eosinophilic pneumonia cases fall into three
categories: 1. Extrinsic asthma with PIE syndrome, in fact, often allergic
bronchopulmonary aspergillosis; 2. Intrinsic asthma with PIE
syndrome (Cell chronic eosinophilic pneumonia), chest x-ray, often
characteristic of pulmonary infiltrates the surrounding Department; 3.
Allergic Granulomatosis (Churg-Strauss Syndrome, nodular lesions of
various arthritis associated with lung). Simple Eosinophilic cell pneumonia
(L? ffler syndrome) pairs can be combined with asthma.

Not associated with asthma of Eosinophilic cell acute eosinophilic
pneumonia including cellular pneumonia, Eosinophilic cells muscular pain
syndrome and Eosinophilic cells increase syndrome. Cell acute
eosinophilic pneumonia is an unexplained different diseases, can cause
acute fever, severe hypoxemia, diffuse pulmonary infiltrates and
bronchoalveolar lavage of Eosinophilic cells proportional 25% following a
corticosteroid therapy can quickly, complete remission. Eosinophilic cells
muscular pain syndrome and eat a lot of as food additives of l-tryptophan-
related pulmonary infiltrates pairs can-with myalgia, muscle weakness, skin
rash and similar to Scleroderma soft tissue induration. Eosinophilic cells
increase syndrome diagnostic criteria for sustainability of Eosinophilic
cells increase ( 1500/mm3) for up to 6 months, a lack of other cause
Eosinophilic cells causes increased, with heart, liver, spleen, central nervous
system or lung, heart most frequently suffer. fever, weight loss and anemia
are common, and can often occur artery instead of venous thromboembolic

Characteristic changes include Eosinophilic cells, monocytes full of alveolar,
alveolar interval Eosinophilic cells, plasma cells and big, small monocyte
infiltration. may also have the Bronchioles in mucus filled and vascular

Symptoms and signs

Signs and symptoms can be mild, or life-threatening low grade can with
mild fever, (if any) respiratory symptoms, you can instantly restore. other
types of PIE syndrome, may have fever and asthma symptoms, including
coughing, wheezing in and resting time breathing difficulties. If not treated,
chronic eosinophilic pneumonia often progressive cell development and life-
threatening, similar acute eosinophilic pneumonia. often apparent increase
of Eosinophilic cells (20% ~ 40%, and sometimes higher)-chest x-ray
findings for different lung lobes are rapidly emerging and rapidly vanishing
of invasive disease (walk of infiltration).


According to patients living in geographic area for pathogenic helminths.
sputum can find parasites and Aspergillus fumigatus. required careful
medication history. Differential diagnosis including nodules, Sarcoidosis,
Hodgkin's disease and other Lymphoid Hyperplasia of the lung disease,
Eosinophilic cell Granuloma, desquamation of Interstitial pneumonia and
collagen-vascular diseases. Hypersensitivity Pneumonitis and Wegener's
Granulomatosis often not with eosinophilic cells increases.


The disease is self-limiting, is benign, but is not required for the treatment of
symptoms of severe, as the use of corticosteroids (see above prednisone
Hypersensitivity Pneumonitis) often have very good results; for cell acute
eosinophilic pneumonia and idiopathic chronic eosinophilic pneumonia, the
treatment can save lives as a bronchial asthma, use usually treatment
methods. A worm infection, you should use anthelmintic.

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