Interstitial Lung Disease

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Shared by: Amna Khan
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Interstitial Lung Disease Pulmonary interstitium  Alveolar lining cells (types 1 and 2) Thin elastin-rich connective component containing capillary blood vessels  Interstitial lung disease      Increase in interstitial tissue causing “stiffness” Physiological restrictive defect Alveolar-capillary interface is site of injury Acute and chronic disease Lots of causes/patterns! Interstitial lung disease   Early stage and acute is alveolitis (injury with inflammatory cell infiltration) Late stage characterised by fibrosis Clinical effects due to hypoxia (respiratory failure) and cardiac failure Causes….  Environmental (mineral dusts, drugs, radiation. Post-ARDS)  Hypersensitivity (mouldy hay, avian proteins)  Unknown (idiopathic) Connective tissue diseases  Idiopathic pulmonary fibrosis Diagnosis based on clinical features often with biopsy  Biopsy in interstitial lung disease…   Transbronchial biopsy – special forceps used at bronchoscopy Thoracoscopic biopsy – more invasive but more reliable and generates far more tissue ARDS (adult respiratory distress syndrome)    Classic model of acute interstitial injury Diffuse alveolar damage syndrome (DADS) Shock lung  Causes include sepsis, diffuse infection (virus, mycoplasma), severe trauma, oxygen Pathogenesis of ARDS Injury (eg bacterial endotoxin)  Injury to cell membranes (either alveolar epithelial cells or endothelial cells)  Infiltration of inflammatory cells  Cytokines  Oxygen free radicals Pathology of ARDS    Fibrinous exudate lining alveolar walls (hyaline membranes) Cellular regeneration Inflammation ARDS with hyaline membrane ARDS – cellular reaction Outcome of ARDS    Death Resolution Fibrosis (chronic restrictive lung disease) Neonatal RDS    Premature infants Deficient in surfactant (type 2 alveolar lining cells Increased effort in expanding lung physical damage to cells Chronic Interstitial Disease      Idiopathic pulmonary fibrosis (IPF) Sarcoidosis Extrinsic allergic alveolitis (hypersensitivity pneumonitis) Pneumoconiosis Connective tissue diseases IPF     aka cryptogenic fibrosing alveolitis (CFA), usual interstitial pneumonia (UIP) Progressive interstitial fibrosis of unknown cause Variable associated inflammation Finger clubbing Pathology    Subpleural and basal fibrosis Inflammatory component variable Terminally lung structure replaced by dilated spaces surrounded by fibrous walls Fibrosing alveolitis (early) IPF (late – honeycombing) Honeycomb lung   Subpleural “enlarged” spaces with fibrous walls A powerful “splint” effect – like a plaster cast Honeycombing – basal/subpleural (a splint!) Extrinsic allergic alveolitis (hypersensitivity pneumonitis)  Chronic inflammatory disease     Allergic origin  Small airways Interstitium Occasional granulomas  Type III hypersensitivity Type IV hypersensitivity Inflammation in hypersensitivity pneumonitis Causes of EAA    Thermophilic bacteria – Farmers lung Avian proteins – Bird fanciers lung Fungi – Malt workers lung Precipitins (antibodies) often detectable in serum. Unusual cases come to biopsy. Sarcoidosis     Multisystem granulomatous disorder of unknown cause (defined by histological means) Pulmonary involvement is common Most cases mild and self-limiting In US 10 times commoner in black vs. white Other manifestations of sarcoidosis     Uveitis (inflammation of iris) Erythema nodosum Lymphadenopathy Hypercalcaemia Transbronchial biopsy - sarcoidosis Sarcoidosis - granuloma Pulmonary involvement in connective tissue diseases    Interstitial fibrosis (milder than fibrosing alveolitis) Pleural effusions Rheumatoid nodules Pneumoconiosis Lung disease caused by mineral dust exposure • Asbestosis • Coal workers lung • Silicosis Thin whole mount section of a coal-workers lung (unstained) Coal miner with progressive massive fibrosis (unstained) Disease depends on….     Particle size (1-5mm) Reactivity of particle Clearance of particle Host response Asbestos    A silicate Serpentine (curved) asbestos fibres relatively safe Straight (amphibole) asbestos highly dangerous Asbestosis bodies (from human lung) Association of asbestos bodies with fibrosis (asbestosis) Pleural plaque on diaphragm Pulmonary eosinophilia    Alveolar infiltration by eosinophils: Tropical eosinophilia – associated with migration of microfilariae through the lung Other eosinophilic “pneumonias” – a range of immune responses Often acutely sensitive to treatment with steroids

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