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Abatacept for Rheumatoid Arthritis

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Abatacept (ORENCIA) for Rheumatoid Arthritis Biological License Application Arthritis Advisory Committee September 6, 2005 Abatacept  Proposed indications for abatacept:  For use in adult patients with moderately to severely active rheumatoid arthritis who have had an inadequate response to one or more biologic or non-biologic DMARDs Reducing signs and symptoms  Inducing major clinical response  Inhibiting the progression of structural damage  Improving physical function   Abatacept may be used as monotherapy or concomitantly with methotrexate or other nonbiologic DMARD therapy 2 Overview of FDA Presentation   Clinical Development Program and Study Design Efficacy Data Improvement of Signs and Symptoms  Improvement of Physical Function  Inhibition of Radiographic Progression    Safety Data Summary 3 Abatacept BLA CLINICAL DEVELOPMENT PROGRAM & STUDY DESIGN 4 Abatacept Clinical Trials Randomized, Double-Blind, Placebo-Controlled Cinical Trial IM101-100 (Phase II) 12-month doseranging study with concomitant DMARDs IM101-102 (Phase III) 12-month trial with concomitant MTX IM101-029 (Phase III) 6-month trial in TNF-blocker failures IM101-031 (Phase III) 12-month trial addon to standard of care IM103-002 (Phase II) 3-month doseranging monotherapy study IM101-101 (Phase II) 12-month trial with concomitant etanercept Total Control Subjects (n) Subjects Treated with Abatacept (n) 119 220 219 133 482 433 258 959 32 90 36 85 1021 2045 5 Study Design-Common Features   Randomized, double-blind, placebo-controlled studies Major Inclusion Criteria:   Diagnosis of RA (1987 ARA criteria) Active disease despite DMARD therapy at randomization    ≥ 10 swollen joints (66 joint count) ≥ 12 tender joints (68 joint count) CRP ≥ 1 mg/dL  Stable doses of prednisone and NSAIDs allowed  Abatacept Dosing: Week 0, 2, and 4, then Q4 weeks   Weight-based dosing Weight-Tiered-based dosing    <60kg: Abatacept 500 mg IV 60 kg to 100 kg: Abatacept 750 mg IV > 100 kg: Abatacept 1000 mg IV 6 Study Design-Common Features  Statistical Analyses Modified ITT efficacy analyses performed for all trials  Sequential testing for co-primary endpoints  Co-primary endpoint tested for significance only if preceding co-primary endpoint was statistically significant  Type I error rate of 5% maintained   Adjustment for multiple doses performed using global testing then pairwise comparisons for individual doses 7 Study Design-Common Features  Statistical Analyses  ACR and Health Assessment Questionnaire (HAQ) response rates Categorical Endpoints  Chi-square Test  Non-responder imputation for missing data   Radiographic Progression Genant-modified Sharp Score  Rank-based nonparametric ANCOVA model  Linear extrapolation for missing data  8 Study IM101-102: Concomitant MTX Study   12 month study Active RA despite MTX therapy  656 patients randomized 2:1   Weight-Tiered-dose Abatacept + MTX (n=433) Placebo + MTX (n=219)  Sequential Co-Primary Endpoints 1. 2. ACR 20 response at 6 months Improvement in Physical Function (HAQ) at 12 months 3. Inhibition of Radiographic Progression at 12 months 9 Study IM101-100: Dose-Ranging Study   12 month Study Active RA despite MTX therapy  339 patients randomized 1:1:1    Abatacept 10 mg/kg + MTX (n=115) Abatacept 2 mg/kg + MTX (n=105) Placebo + MTX (n=119)  Primary Endpoint  ACR 20 response at 6 months 10 Study IM101-029: TNF-Blocker Failure Study   6 month Study Active RA despite TNF-blocker therapy ± DMARD  Etanercept or Infliximab  Following drug-washout 393 patients with active RA randomized 2:1   Weight-Tiered-dose Abatacept + DMARD (n=258) Placebo + DMARD (n=133) ACR 20 response at 6 months Improvement in Physical Function (HAQ) at 6 months  Co-Primary Endpoints 1. 2. 11 Study IM101-031: Clinical Practice Study   12 month Study Active RA despite DMARD therapy  non-biologic and/or biologic DMARDs   Patients with co-morbid conditions permitted 1441 patients randomized 2:1   Baseline therapy + Weight-tiered dose Abatacept (n=959) Baseline therapy + Placebo (n=482) Safety Improvement in Physical Function (HAQ) at Day 365 12  Primary Objective   Exploratory Endpoint  Clinical Studies-Study Conduct  Baseline Patient Demographics (mean): 52 years of age  79% Female  85% White and 4% Black   Baseline Disease Activity (mean): 10 years RA duration  21 swollen joints  31 tender joints  79% RF(+)  MTX 16 mg Qweek  13 Abatacept BLA EFFICACY ANALYSES Signs and Symptoms 14 IM101-102: Concomitant MTX Study Signs & Symptoms: ACR Responses Abatacept + MTX (n=424) Day 169 ACR 20 ACR 50 ACR 70 Day 365 ACR 20 ACR 50 ACR 70 288 (68%)* 169 (40%)* 84 (20%)* Placebo + MTX (n=214) 85 (40%) 36 (17%) 14 (7%) 310 (73%)* 205 (48%)* 122 (29%)* *p<0.001 85 (40%) 39 (18%) 13 (6%) 15 IM101-102: Concomitant MTX Study Signs & Symptoms: ACR 20 Response Time Course 100 90 80 70 60 50 40 30 20 10 0 15 29 57 ACR 20 Responders (%) Abatacept + MTX Placebo + MTX 85 113 141 169 225 281 365 Study Day 16 IM101-102: Concomitant MTX Study Signs & Symptoms: Major Clinical Response Abatacept + MTX (n=424) Number of responders (%) Major Clinical Response p-value 60 (14%) <0.001 Placebo + MTX (n=214) 4 (2%) 17 IM101-102: Concomitant MTX Study Median % Improvement in ACR Components at Day 169 Median Baseline Score # Swollen Joints # Tender Joints Subject Pain Assessment Physical Function (HAQ) Subject Global Assessment Physician Global Assessment CRP 19 28 67 1.75 66 69 2.2 Abatacept + MTX (n=424) 74% 75% 45% 35% 56% 70% 59% Median Baseline Score 20 31 70 1.75 64 68 2.1 Placebo + MTX (n=24) 45% 55% 29% 21% 25% 41% 4% 18 IM101-102: Concomitant MTX Study DAS28 Response at Day 365 Abatacept + MTX (n=366) Baseline Mean Day 365 Patients with improvement (DAS28 change 1.2) Patients with EULARdefined low disease activity (DAS 3.2) Patients in EULAR- defined Remission (DAS <2.6) 6.8 4* 328 (88%) Placebo + MTX (n=179) 6.8 5.4 108 (59%) 103 (28%) 7 (4%) 65 (17%) *p<0.001 4 (2%) 19 IM101-100: Dose-Ranging Study Signs & Symptoms: ACR Responses Abatacept 10 mg/kg + MTX (n=115) Day 180 ACR 20 ACR 50 ACR 70 Day 360 ACR 20 ACR 50 ACR 70 70 (61%)* 42 (37%)* 19 (17%)* Abatacept 2 mg/kg + MTX (n=105) 44 (42%) 24 (23%)* 11 (11%)* Placebo + MTX (n=119) 42 (35%) 14 (12%) 2 (2%) 72 (63%)* 48 (42%)* 24 (21%)* 44 (42%) 24 (23%) 13 (12%) *p≤0.03 43 (36%) 24 (20%) 9 (8%) 20 IM101-029: TNF-Blocker Failure Study Signs & Symptoms: ACR Responses at Day 169 Abatacept (n=258) Day 169 ACR 20 ACR 50 ACR 70 129 (50%)* 52 (20%)* 26 (10%)* *p≤0.003 Placebo (n=133) 26 (20%) 5 (4%) 2 (2%) 21 IM101-029: TNF-Blocker Failure Study Signs & Symptoms: DAS28 Response at Day 169 Abatacept (n=366) Baseline Mean Day 169 Subjects with improvement (DAS28 change 1.2) Subjects with EULARdefined low disease activity (DAS 3.2) Subjects in EULAR- defined Remission (DAS <2.6) 6.9 4.9* 129 (71%) Placebo (n=179) 6.9 6.2 3 (2%) 30 (17%) 4 (4%) 19 (10%) 1 (1%) *p<0.001 22 Exploratory Analysis Criteria to Assess Very Low Disease Activity  EULAR-definition of remission is defined as a DAS28<2.6 However, patients with a EULAR definition of remission can still have several swollen or tender joints Alternative criterion for very low disease activity DAS28<2.6 AND ≤ 1 swollen and ≤1 tender joint   23 Criteria to Assess Very Low Disease Activity IM101-102: Concomitant MTX Study Abatacept + MTX N Day 169 Total Patients with EULAR-defined remission (DAS<2.6) Total Patients with EULAR-defined remission (DAS<2.6) AND ≤1 swollen and ≤1 tender joint N Day 365 Total Patients with EULAR-defined remission (DAS<2.6) Total Patients with EULAR-defined remission (DAS<2.6) AND ≤1 swollen and ≤1 tender joint 394 35 (9%) 17 (4%)* Placebo + MTX 195 1 (<1%) 1 (<1%) 406 65 (17%) 44 (11%)** 202 4 (2%) 2 (1%) *p<0.05; **p<0.001 24 Criteria to Assess Very Low Disease Activity IM101-029: TNF-Blocker Failure Study Abatacept + MTX 202 19 (9%) 10 (5%)* Total Patients with EULAR-defined remission (DAS<2.6) AND ≤1 swollen and ≤1 tender joint *p<0.05 Placebo + MTX 111 1 (<1%) 0 N Day 169 Total Patients with EULAR-defined remission (DAS<2.6) 25 Exploratory Analysis Weight-Tiered-Dosing : ACR20 Responders Abatacept Weight (kg) <50 50 to <60 60 to <70 70 to <80 80 to <90 90 to ≤100 101 to <110 >110 Approx. Abatacept mg/kg Placebo N 10 47 62 45 26 12 7 5 N(%) Response 5 (50%) 12 (26%) 28 (45%) 16 (36%) 16 (62%) 5 (42%) 2 (29%) 1 (20%) Est. of Diff. 13 39 20 36 14 38 44 24 N Dose N(%) Response 19 (63%) 50 (65%) 74 (66%) 62 (71%) 48 (75%) 19 (79%) 8 (73%) 8 (44%) <12.5 9.1 11.5 10 8.8 7.9 9.5 >8.3 30 77 113 87 64 24 11 18 26 Abatacept BLA EFFICACY ANALYSES Improvement in Physical Function 27 IM101-102: Concomitant MTX Study Improvement in HAQ score ≥ 0.3u at Day 365 Abatacept + MTX (n=424) HAQ Number of responders achieving 0.3 units p-value 270 (64%) Placebo + MTX (n=214) 84 (39%) <0.001 28 IM101-100: Dose-Ranging Study Improvement in HAQ score ≥ 0.3u at Day 360 Abatacept Abatacept 10 mg/kg + MTX 2 mg/kg + MTX Placebo + MTX (n=115) (n=105) (n=119) HAQ Number of responders achieving 0.3 units p-value 44 (38%) 31 (30%) 24 (20%) 0.002 0.104 - 29 IM101-100: Dose-Ranging Study Improvement in HAQ score ≥ 0.3u Open-Label Study 70 % Patients with Improvement 60 50 40 30 20 10 0 15 90 180 240 360 Abatacept 10 mg/kg Abatacept 2 mg/kg Placebo 450 630 720 810 900 1080 30 Study Day Abatacept BLA EFFICACY ANALYSES Inhibition of Radiographic Progression 31 IM101-102: Concomitant MTX Study Mean Change In Genant-Modified Sharp Scores at Day 365 Abatacept + MTX (n=391) Total Score Baseline mean ± SD Mean change from baseline (± SD) Median change from baseline (range) p-value Placebo + MTX (n=195) 44u ± 37 1.21u ± 2.94 0.25 (0-1.78) 0.012 45u ± 38 2.32u ± 5.04 0.53 (0-2.54) 32 IM101-102: Concomitant MTX Study Mean Change in Genant-Modified Sharp Score Components Abatacept + MTX (n=391) Erosion Score Baseline mean ± SD Mean change from baseline (± SD) Median change from baseline (range) p-value Joint Space Narrowing Baseline mean ± SD Mean change from baseline (± SD) Median change from baseline (range) p-value 23u ± 20 0.58u ± 1.54 0 (0-0.49) 0.009 33 Placebo + MTX (n=195) 22u ± 19 1.14u ± 2.81 0.27 (0-1.27) 22u ± 18 0.63u ± 1.77 0 (0-1.02) 0.029 23u ± 20 1.18u ± 2.58 0.27 (0-0.97) Study IM103-002: Monotherapy Study 3 month study  Active  112  RA despite DMARD therapy (n=90) patients randomized 0.5 mg/kg (n=26), 2 mg/kg (n=32), or 10 mg/kg (n=32)  Abatacept  Placebo  (n=32) Primary Endpoint  ACR 20 response at Day 85 34 Study IM103-002: Monotherapy Study ACR Responses at Day 85 Abatacept 0.5 mg/kg (n=26) Day 85 ACR 20 ACR 50 ACR 70 6 (23%) 0 0 4 (44%) 6 (19%) 4 (13%) 7 (53%) 5 (16%) 2 (6%) 10 (31%) 2 (6%) 0 Abatacept 2 mg/kg (n=32) Abatacept 10 mg/kg (n=32) Placebo (n=32) 35 Abatacept  Efficacy Analysis Subset Analyses by:  Baseline Demographics Age  Sex  Race  Weight   Baseline Disease Activity Disease Duration  Swollen & Tender Joints  CRP  Genant-modified Sharp Score  HAQ  36 Abatacept BLA SAFETY ANALYSES 37 Safety Analyses: Overview  Safety Assessment Based on 5 Studies: IM101100, IM101101, IM101102, IM101029, IM101031  Double-Blind Periods:  1955 Abatacept-treated patients (1688 person-years)  989 Placebo-treated patients (795 person-years)   Open-Label Periods + Double-Blind Periods:  2688 Abatacept-treated patients 38 Cumulative Extent of Exposure Number (%) of Subjects Abatacept 0.5 mg/kg (n=26) 7 (27%) 19 (73%) 0 0 0 0 0 4 Abatacept 2 mg/kg (n=222) 19 (8%) 203 (92%) 157 (71%) 89 (40%) 0 0 0 12 Abatacept 10 mg/kg or Tiered-dose (n=2638) 460 (17%) 2178 (83%) 1868 (71%) 1596 (61%) 263 (10%) 223 (8%) 66 (2%) 14 All Abatacept (n=2760) 483 (17%) 2277 (83%) 1908 (70%) 1622 (60%) 282 (11%) 248 (10%) 151 (6%) 14 Months <3 ≥3 ≥6 ≥12 ≥18 ≥24 36 Median (month) 39 Deaths  26 total deaths  16 patients died during the double-blind periods  10  (0.5%) abatacept-treated patients 4 died from cardiovascular disorders  3 found dead at home  2 died from malignancies  1 died from infection 6 (0.6%) placebo-treated patients 2 died from cardiovascular disorders  1 found dead at home  1 died from malignancy  2 died from infection  40 Deaths  Analysis of the individual deaths did not suggest a safety signal for any single type of AE 8 of the deaths in the Abatacept group occurred during a study that permitted enrollment of patients with co-morbidities  41 Serious Adverse Events  14% of Abatacept-treated patients had an SAE compared to 12% placebo-treated patients  3% of Abatacept-treated patients had an infectious SAE compared to 2% placebotreated patients 42 Malignancies: Double-Blind Periods Malignant Abatacept: 29 (1.5%) Placebo: 11 (1.1%%) Non-Melanoma Skin CA Abatacept: 15 (0.8%) Placebo: 6 (0.5%) Solid Organ CA Abatacept: 13 (0.7%) Placebo: 5 (0.5%) Hematologic Abatacept: 2 (0.1%) Placebo: 0 43 Solid Organ Tumors Abatacept-Treated Patients-Double Blind Periods Subject Age/Gender/Race 69/F/W 68/M/W 72/F/W 83/M/W 53/M/W 71/F/W 63/F/W 74/M/W 52/F/W 42/F/W 70/F/W 39/F/W No. of Infusions 3 5 13 13 8 14 4 5 6 7 6 5 Onset Day 29 100 320 332 203 349 42 97 115 237 85 69 Malignancy Lung Neoplasm Non-small cell lung CA Sq. Cell lung CA Renal Cell CA Lung Neoplasm Bladder CA Breast CA Ovarian CA Prostate CA Thyroid CA Breast CA Cholangiocarcinoma Cervical CA 44 Malignancies: Open-Label Periods  47 patients developed 52 neoplasms  26 malignancies  13  solid-organ tumors 4 lung cancers  2 ovarian cancers  2 endometrial cancers  1 case each of breast, prostate, melanoma, cervical, and rectal cancer 3 lymphomas 45 Most Frequently Observed vs. Expected Malignancies Malignancy Overall Lung Lymphoma Breast Prostate Thyroid Ovarian Endometrial Observed 26 8 4 2 2 2 2 2 Expected 30.2 4.0 1.1 7.7 3.2 0.6 0.7 1.5 SIR 0.9 2.0 3.7 0.3 0.6 3.5 2.7 1.3 SIR 95% CI 0.6-1.3 0.9-4.0 1.0-9.5 0-0.9 0.1-2.2 0.4-12.5 0.3-9.7 0.2-4.9 46 Incidence Rates of Malignancies N (rate/100 person-years) Days 1-180 Total Exposure (p-y) 1285 Days 181-360 1032 Days 361-540 795 Days 541-720 399 Days 721-900 117 Days 901-1080 198 Overall malignancies (excluding non-melanoma skin cancer) 3 (0.15) 0 2 (0.36) 1 (0.16) 1 (0.16) 1 (0.16) 47 Malignancies  3 potentially concerning malignancies  Lung Cancer  8 cases of lung cancer in patients receiving abatacept  Breast Cancer and Lymphoma Pre-clinical studies demonstrated increased rate of mammary tumors and lymphomas in mice that was believed to be secondary to abatacept-induced chronic immunosuppression and MMTV and MLV  Immunosuppression and RA both associated with increased risk of lymphoma  48 Lung Cancer Incidence Observed Events (95% CI) All Abatacept 8 (3.45, 15.76) Trial Expected Number of Events (95% CI) BC 9.95 (6.5, 16.3) NDB 3.58 (1.8, 8.3) NOAR 4.76 (1.1, 22.8) Expected Number of Events SEER 4 (0.9, 4.0) 49 Breast Cancer & Lymphoma  Breast Cancer  3 (0.1%) cases of breast cancer reported in abatacept-treated patients compared to 2 (0.2%) cases reported in placebo-treated patients  Current evidence does not suggest abatacept increases the rate of breast cancer  Lymphoma  4 cases of lymphoma reported in patients receiving abatacept  Approximately 4-fold higher than general US population  However, increased rate of lymphoma in RA patients, particularly those with high disease activity 50 Serious Infections Serious Infection Preferred term Infectious SAE (n) Pneumonia Cellulitis Urinary tract infection Bronchitis Diverticulitis Acute Pyelonephritis Localized infection Sinusitis Subcutaneous abscess Abatacept (n=1955) 58 (3%) 14 (0.7%) 5 (0.3%) 4 (0.2%) 4 (0.2%) 3 (0.2%) 3 (0.2%) 2 (0.1%) 2 (0.1%) 2 (0.1%) Placebo (n=989) 19 (2%) 5 (0.5%) 2 (0.2%) 1 (0.1%) 0 0 0 0 0 0 51 Infections of Special Interest Infection Preferred term Infection of Special Interest Total Subjects All Herpes Infections Pneumonia Opportunistic Infections Herpes Zoster Oral Fungal Infection Tuberculosis Aspergillosis 30 (2%) 3 (0.1%) 2 (0.1%) 1 (<0.1) 16 (2%) 2 (0.1%) 1 (0.1%) 0 52 Abatacept (n=1955) 187 (10%) 72 (4%) 40 (2%) Placebo (n=989) 70 (7%) 28 (3%) 8 (1%) AEs in Abatacept w/ Biologic RA Therapy  A total of 204 patients received abatacept and concomitant biologic RA therapy during the double-blind period representing 173 person-years of exposure  Majority of patients were from studies IM101-101 (n=85) and IM101-031 (n=103)  >90% TNF-blocker therapy  Study IM101-101 compared the combination of abatacept 2 mg/kg + etanercept to placebo + etanercept 53 AEs in Abatacept w/ Biologic RA Therapy Number (%) of Subjects Biologic RA Therapy Abatacept Placebo (n=204) (n=134) SAEs AEs 40 (20%) 192 (94%) 12 (9%) 113 (84%) Non-Biologic RA Therapy Abatacept Placebo (n=1751) (n=855) 226 (13%) 1544 (88%) 110 (13%) 727 (85%) 54 SAEs with Abatacept & Concomitant RA Therapy Study IM101031 Background RA Therapy Total in Biologic Subgroup Etanercept Infliximab Adalimumab Anakinra Total in Non-Biologic Subgroup MTX Hydroxycholoquine/Chloroquine Sulfasalazine Leflunomide 1 DMARD 2 DMARDs 3 DMARDs 4 DMARDs Number of Subjects (%; n of subgroup) Abatacept Placebo 23 (22%; n=103) 8 (13%; n=64) 17 (26%; n=66) 5 (12%; n=42) 3 (15%; n=20) 0 (0%; n=5) 3 (27%; n=11) 1 (10%; n=10) 2 (15%; n=13) 2 (20%; n=10) 100 (12%;n=856) 73 (11%; n=691) 23 (12%; n=194) 15 (11%; n=137) 25 (24%; n=106) 66 (11%; n=598) 27 (13%; n=202) 6 (13%; n=45) 1 (10%; n=10) 51 (12%; n=418) 37 (11%; n=336) 10 (8%; n=123) 9 (13%; n=72) 9 (15%; n=59) 36 (14%; n=257) 14 (11%; n=123) 0 (0%; n=31) 1 (17%; n=6) 55 Infusion Reactions  Infusion Reactions within 1 hour post-infusion  9% of abatacept-treated patients vs. 6% placebotreated patients  Infusion Reactions within 24 hours postinfusion  23% of abatacept-treated patients vs. 19% placebo-treated patients  2 cases of anaphylactic reactions in patients receiving abatacept 56 Immunogenicity & Clinical Laboratory Values  Immunogenicity of Abatacept  Overall, 1.6% of patients treated with abatacept developed antibodies to abatacept 5.8% of abatacept patients who had discontinued therapy for at least 56 days developed antibodies to abatacept   Changes in Clinical Laboratory Values  no clinically meaningful differences between patients treated with abatacept compared to placebo regarding blood chemistry and hematologic laboratory values 57 Autoimmune Symptoms & Disorders  3% of Abatacept-treated patients reported autoimmune-related AEs compared to 2% of placebotreated patients during the Double-Blind period  Most common symptoms were associated with RA  e.g., Keratoconjunctivitis sicca, Sjogren’s syndrome Psoriasis was more frequent in patients treated with abatacept compared to placebo (0.5% vs. 0.1%)  58 Autoimmune Symptoms & Disorders ANA and ds-DNA Antibody Formation at 12 months Anti-nuclear Antibody n (%) 93 (10%) 443 (11%) ds-DNA Antibody n (%) 29 (3%) 23 (5%) Abatacept Placebo N 962 575 N 1062 490 59 AEs in patients with Co-Morbidities Study IM101031    Study IM101-031 permitted patients with comorbidities including COPD, diabetes, asthma, and CHF Diabetes, asthma, or CHF: no apparent increase in AEs or SAEs COPD: AEs reported in 97% of abatacept-treated patients vs. 88% of placebo-treated patients    Respiratory AEs more common with abatacept (43% vs. 24%) SAEs more common with (27% vs. 6%) No reported deaths 60 Summary  The studies presented here show abatacept-treatment associated differences regarding    improvement in signs and symptoms improvement of physical function Inhibition of radiographic progression  There was a higher rate of serious infections in patients treated with abatacept, especially with patients receiving concomitant TNF-blocking agents Overall malignancy rates were not substantially different between abatacept (1.5%) and placebo (1.1%) treated patients   However, abatacept treated patients had more cases of lung cancer and the rate of lymphomas was higher than expected compared to the general US population 61 Summary  Infusion-related reactions were observed including hypersensitivity reactions and 2 cases of anaphylaxis  Patients with COPD treated with abatacept had a higher incidence of adverse events and serious adverse events, particularly respiratory disorders 62
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