Vitamins and Coenzymes

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TUMS









Azin Nowrouzi, PhD

Vitamin classification

Name(Letter) RDI

Retinol (A) 5000 IU

Calciferol (D) 400 IU

Tocopherol (E) 30 IU

Phylloquinone (K) 70 g

Fat-soluble Vitamins

• Absorbed with dietary fat in small intestine

• 40-90% absorption efficiency

• Absorption typically regulated by need

– need absorption

• Transported away from small intestine in

chylomicra via blood and lymph (depending on

size)

• They are not easily excreted through urine.

• Substantial amounts are stored in liver and fat

tissue.

• Therefore, toxicity is possible.

Vitamin A

Compounds with 20-carbon structure.

Contain a methyl substituted cyclohexenyl ring (-ionone ring),

and an isoprenoid side chain with either a hydroxyl group, and aldehyde

group, a carboxylic acid group, or an ester group (retinyl ester) at the

terminal C15.









All-trans-retinal 11-cis-retinal









Retinol Retinoic Acid

Can‟t be reduced to retinol or retinal in the body.

Vitamin A

1. Active vitamin A- Preformed vitamin A can be obtained

either directly from foods that are substantial in vitamin

A (beef liver, fish liver oils, egg yolks and butter)

• The active form of vitamin is retinol, an alcohol which can be

converted to other forms (e.g. vitamin A esters) for storage in liver

and tissues.

• much the body's vitamin A is stored in the liver as retinyl palmitate

2. Provitamin A- provitamins, substances that are

transformed into vitamins in the body

• Beta-carotene is the most abundant and widespread provitamin A.

• Beta-carotene comes from a group of compounds called the

"carotenoids (C40 polyisoprenoids).

• One need to eat approximately six times as much beta-carotene

to get the same amount of vitamin A as in retinol.

• Dark-green leafy vegetables (spinach) and yellow-orange

fruits (apricots and mango) and vegetables (carrots, yellow

squash and sweet potatoes) are high in beta-carotene and other

carotenoids (-carotene, -cryptoxathin, etc.)

• Carotenoids are not toxic even at high doses for long times.

Vitamin A: Absorption and transport

- Micelle formation

- Inside mucosal cell -carotene is

cleaved in the lumen of the intestine

by -carotene dioxygenase to yield

retinal.

- Retinal is reduced to retinol by

retinal reductase, an NADPH

requiring enzyme within the

intestines.

- Retinol is esterified to palmitic acid

and delivered to the liver with

chylomicrons via lymphatic system.

- Carotenoids which escape intestinal

conversion to retinol can be stored in

adipose tissue.

- Species in which this occurs include

humans, horses and cattle, which

have yellow-pigmented body fat when

carotenoid intake is high.

- Vitamin A absorption efficiency 70-

90%; carotenoids 9-22%.

Vitamin A: Biological functions

• Role in vision. Vitamin A (retinal) is an essential precursor for

formation of the visual pigment, rhodopsin, in the retina of the

eye. Retinal plays an important role in vision, especially night

vision. Lowers cataract incidence.



• Role in growth. Helps regulate cell development, cell

differentiation and cell division.



• Role in Bone and teeth formation. Promotes the proper growth

of bones and teeth. Bone cells (osteoblasts and osteoclasts)

depend on vitamin A for their normal functioning.

– Is important in the formation and maintenance of healthy hair, skin

and mucous membranes.



• Role in reproduction. Vitamin A holds an important place in

sexual reproduction. Adequate levels of vitamin A are needed

for normal sperm production. The female reproductive cycle

requires sufficient amounts of vitamin A.



• Boosts the body's immune system helping to increase body

resistance to infectious diseases.

Role of Vitamin A in Vision

(Wald‟s visual cycle)

1. When the rhodopsin is exposed to light it is bleached releasing the

11-cis-retinal from opsin.

2. Isomerization of the cis-isomer of retinal to all-trans-retinal, causes

conformational changes in rhodopsin, hyperpolarization of the retinal

rod cell, and extremely rapid transmission of electrical activity to the

brain via the optic nerve

3. Trans-retinal is isomerized to cis-retinal in the dark, which

associates with opsin to regenerate rhodopsin.



All trans retinol

= main circulating

form of Vit A









Visual Pigment

Vitamin A: Biological functions

(Cell growth and differentiation)

• Regulation of gene expression by retinoic acid,

the acid form of vitamin A

• Retinoic acid is formed within the target cell from

retinol transported in the bloodstream.

• Retinoic acid, combined with protein receptor,

acts as transcription regulator by binding to

specific sequences of DNA in the nucleus

• Affected cells include epithelium of numerous

tissues, including skin and gut, and bone, ovary

and testis.

• Retinoic acid is an especially important regulator

of embryonic cell differentiation.

Vitamin A: Deficiency symptoms

1. Night blindness" - lessened ability to see in dim light.

2. Increased susceptibility to infection and cancer and anemia

equivalent to iron deficient.

3. Prolonged lack of vitamin A

(keratinization of the cornea, a condition known as xerophthalmia).

4. Abnormal bone development in fetal and neonatal life.

5. Various congenital defects.



Retinol and its precursors are used as dietary supplements to prevent

the above symptoms.



Retinoic acid is used in dermatology to cure acne and psoriasis:

1. Mild acne (Darier‟s disease) and aging

Topical application of tretinoin (all-trans retinoic acid), along with

benzoyl peroxide and antibiotics.

2. severe recalcitrant cystic acne

Oral consumption of isotretinoin (13-cis retinoic acid). This drug is

teratogenic. Should not be prescribed during pregnancy. It may lead to

hyperlipidemia and increased LDL/HDL ratio (increased chance of

heart disease) in long term.

Vitamin A: Toxicity

• Skeletal malformations

• spontaneous fractures

• internal hemorrhages

• loss of appetite

• slow growth or weight loss.

• Hypervitaminosis A in humans

– Symptoms include dry, pruritic skin with excessive itching and

increase in intracranial pressure.

– is becoming an increasing problem in Western countries because of

self-medication and overprescription.



– has occurred through eating polar bear or seal liver by polar

explorers.

• Eat polar bear liver sparingly, 30 grams contain 450,000 IU of retinol!

Continued ingestion causes peeling of the skin from head to foot.



• It is possible that some early Arctic explorers died from eating their sled

dogs in an attempt to survive.

Vitamin D

• Vitamin D is a family of fat-soluble

sterol compounds:



• Vitamin D1 (lamisterol)

• Vitamin D2 (ergocalciferol)

• Vitamin D3 (cholecalciferol)

• Vitamin D4 (dihydroergocalciferol)

• Vitamin D5 (7-dehydrositosterol)

Vitamin D: Types and Sources

• For humans, the two most important forms of

vitamin D are:

– vitamin D2

– vitamin D3.

• Vitamin D2 (ergocalciferol) is derived from

plants and irradiated yeast and fungi.

• Vitamin D3 is synthesized in the body when

skin is exposed to sunlight

– Cholesterol + sunshine = Vitamin D3

– “sunshine vitamin” – UV-B rays (5-10 minutes arms

and legs, mid-day sun).

• Vitamin D3 can be obtained from foods like

milk, fortified cereals, tuna, salmon and fish

oils.

Sunlight

Vitamin D2

(Ergocalciferol)



Ergosterol

Diet

(in plants)









Sunlight

Vitamin D3

Cholecalciferol)

7-Dehydrocholesterol

Activation of Vitamin D

• Vitamin D2 and vitamin D3 are

biologically inactive but can

have equal biological activity:

• Both can be converted first to

calcifediol in the liver and then

to calcitriol, also known as

1,25-dihydroxycholecalciferol,

in the kidneys.



• Calcitriol, which is the most

active form of vitamin D3, is

then transported via a carrier

protein to the various sites in

the body where it is needed.

Calcitriol is also called

1,25-dihydroxy vitamin D3,

or (1,25-(OH)2D3.

1,25-dihydroxyvitamin D3

Conversion of 25-(OH)D3 to its

biologically active form, calcitriol,

occurs through the activity of a

specific D3-1-hydroxylase present

in the proximal convoluted tubules

of the kidneys, and in bone and

placenta. Cytochrome P450, O2

and NADPH are needed.









25-hydroxyvitamin D3

In the liver cholecalciferol is

hydroxylated at the 25 position

by a specific D3-25-hydroxylase

generating 25-hydroxy-D3 [25-

(OH)D3] which is the major

circulating form of vitamin D.

Vitamin D Functions:

Hormone for Calcium and Phosphate regulation

• Nerves and muscles must function properly; calcium is

vital for nerve cell transmissions and muscle fiber

contractions.

• Calcitriol functions in concert with parathyroid hormone

(PTH) and calcitonin to regulate serum calcium and

phosphorous levels by:

1. Increasing dietary calcium absorption from the small intestine.

2. Decreasing the urinary calcium excretion (increasing renal

reabsorption).

3. Stimulating resorption of calcium from bone

• Instrumental in the growth, hardening and repair of bones.

• Too much vitamin D, however, can increase calcium losses from bone

• There is evidence that vitamin D (specifically, vitamin D3)

is involved in regulation of the body's immune system.

• Vitamin D is essential for normal insulin secretion by the

pancreas and therefore control of blood sugar levels.

When vitamin D goes abnormal

• Too little (northern latitudes) leads to Vit D

deficiency:

– Rickets (bone deformities in children)

– Osteoporosis

– Osteomalacia (weak bones)

• Demineralization of bones

• Too much (5 times the RDA, chronically): It

is most toxic of the vitamins.

– Nausea, thirst, loss of appetite, stupor

– Hypercalcemia: calcium gets deposited in soft

tissues, Arteries and kidneys.

Vitamin E

A family of eight different molecules. Four of the eight vitamin E molecules

are called tocopherols (alpha, beta, gamma and delta) [also known as

“Natural Tocopherols”] while the remaining four are called tocotrienols

(alpha, beta, gamma and delta). D-alpha-tocopherol is the most biologically

active in humans.









-Tocopherol

is the most potent

of the tocopherols.

Functions

• Vitamin E in the form of d-alpha-tocopherol is an important fat-soluble

antioxidant, scavenging oxygen free radicals, lipid peroxy radicals and

singlet oxygen molecules before these radicals can do further harm to

cells. [Free radicals are very reactive atoms or molecules that typically

possess a single unpaired electron.]



• New studies have revealed that the tocotrienol forms of vitamin E are

even more potent antioxidants than the tocopherol isomers.



• Vitamin E helps maintain the structural integrity of cell membranes

throughout the body.



• d-alpha-tocopherol has been shown to inhibit the "clumping" of blood

platelets (thus helping to avoid blood clots) and enhancing vasodilation

(the opening of blood vessels).



• d-alpha-tocopherol protects the fat component in low-density

lipoproteins (LDLs) from oxidation and has shown moderate

cholesterol-lowering capabilities. Studies have shown that gamma-

and delta-tocotrienols may be better suited than the tocopherols at

inhibiting the manufacture of cholesterol in the liver and hence in

contributing to a greater cholesterol lowering effect.

Vitamin E (deficiency)

• Deficiency: rare in adults usually due to impaired fat absorption or

transport; seen usually in children (anemia, edema in infants)

– Muscular dystrophy

– Hemolytic anemia

– Dietary hepatic necrosis



• Excess: very safe below 800 IU/day



• Source:

– Vitamin E is present in animal fats, meat, green vegetables, nuts/seeds.

– Alpha-tocopherol is found in a number of vegetable oils, including safflower

and sunflower. It is also found in wheat germ. Soybean and corn oils contain

mainly gamma-tocopherol.



• The major site of vitamin E storage is in adipose tissue.

• Estimated requirements: 5mg/day = 0.6mg/day of unsaturated fat.

• Uses:

– Hemolytic anemia in premature infants, unresponsive to B12, Fe and folic acids.

– Macrocytic megaloblastic anemia seen in children with severe protein-calorie

malnutrition.

Free Radicals - the Metabolic Oxidizers





Free radical = unpaired electron very reactive





. OH

OH

. OH

OH



Oxygen radicals: Hydroxy (HO•) / Peroxy (HOO•)

An antioxidant is a chemical so easily

oxidized itself that it protects others from oxidation.



OH



and / or







Double Bond Phenol

eg. Vitamin A eg. Vitamin E or C

Antioxidant





The antioxidant

neutralizes a free

radical by donating an

electron.

Vitamin K

• The "K" in vitamin K comes from the German

word "koagulation," which refers to blood

clotting (coagulation).



• Vitamin K is essential for the functioning of

several proteins involved in normal blood

clotting.



• There are several forms K1-K7

Vitamin K1 (Phylloquinone)

• Naturally occurring vitamin K is absorbed from the

intestines only in the presence of bile salts and other lipids

through interaction with chylomicrons. Therefore, fat

malabsorptive diseases can result in vitamin K deficiency.

• Present in green leafy vegetables like lettuce, parsley,

spinach and various greens (beet and mustard). Broccoli

and certain vegetable oils (soybean, cottonseed, and olive).

are also a good source of vitamin K1.



__

Vitamin K2 (Menaquinone)

• Vitamin K2 is a group of compounds called the "menaquinones."

• Synthesized by intestinal bacteria "n" can be 6, 7 or 9 isoprenoid

groups.

– The number of isoprene residues in the side chain of the vitamin

K2 molecule designate the individual menaquinone compounds,

e.g., menaquinone-4 (MK-4) has four isoprene units in the side

chain of vitamin K2; menaquinone-7 (MK-7) has seven isoprene

units in the side chain.

• Vitamin K2, which is the most biologically active form of vitamin K, is

found in egg yolks, butter, liver, cheddar cheese and yogurt.



• It has been suggested that products like yogurt, may help to increase

the functioning of these useful bacteria.

Vitamin K3 (Menadione)

• The synthetic (man-made)

vitamin K3 is water soluble

and absorbed irrespective

of the presence of

intestinal lipids and bile.





Uses : essential cofactor in blood clotting.



Excess: Dangerous if taking anti-coagulants.

Hemolytic anemia, jaundice in infants.

Functions

1. Blood coagulation

• Post-translational carboxylation of some glutamate

residues in blood factors.

• Vitamin K is needed for the body to make four of the

blood's coagulation factors, including prothrombin

(also known as factor II), proconvertin (factor VII),

Christmas factor (factor IX) and the Stuart-Power

factor (factor X).

– It occurs in the liver

– Vitamin K has to change to Hydroquinoine to function

as coenzyme for carboxylase.



2. Calcium binding proteins

– Carboxylation of specific glutamate residues in

calcium binding proteins.

3. Role in oxidative phosphorylation

Prothrombin -Carboxylation

Vitamin K role in -Carboxylation

Deficiency

1. Prolonged use of antibiotics

2. Malabsorption and biliary tract obstruction

3. Spoilt sweet-clover hay

– Contains dicumarol (vit K antagonist)

4. Short circuiting of the bowel

5. In immediate post-natal infants

• (bruising/bleeding in infants).

– Sterile bowels (no intestinal microflora)

– Low Vit K in mothers milk

– Hemorrhagic disease of the newborn.

Too Much Broccoli !

Two women with diseases characterized by abnormal blood

clotting did not improve when they were given Warfarin.

When questioned about their diets, one woman reported

that she ate at least a pound of broccoli every day, and the

other ate broccoli soup and a broccoli salad every day.

When broccoli was removed from their diets, warfarin

became effective in preventing the abnormal clotting of their

blood.

Because broccoli is high in vitamin K, these patients had

been getting enough dietary vitamin K to compete with the

drug, thereby making the drug ineffective.

What do vitamins do?

• Metabolically they have diverse functions as:

• Coenzymes (B vitamins, vitamin K)

• Hormones (retinoic acid, vitamin D)

• Modulators or regulators of growth and

development (retinoic acid, folic acid)

• (apparently non-specific) antioxidants

(Vitamins C and E)

Cofactors and coenzymes

Other coenzymes

Coenzyme Q = Ubiquinone

• A lipid in inner membrane of mitochondria.

• Moves freely within membrane.

• Carries electrons.

• Coenzyme Q10 (CoQ 10) or ubiquinone is essentially a vitamin or

vitamin-like substance.

– Found in small amounts in a wide variety of foods.

– It is synthesized in all tissues.

– The biosynthesis of CoQ10 from the amino acid tyrosine requires at least

eight vitamins and several trace elements.

• Coenzyme Q10 is the coenzyme for at least three mitochondrial

enzymes (complexes I, II and III) as well as enzymes in other parts

of the cell.

• CoQ10 has been studied in its reduced form as a potent antioxidant.





2H++2e



Coenzyme Q

Coenzyme Q

Coenzyme Q - 'promoted' to a Vitamin?

O

CH3O CH3





CH3

CH3O

C

O H2 C C H

H H2

10



A vital 'transport agent' in the use of oxygen to generate

energy, especially in the heart muscle.

Supplements do aid in many forms of heart disease,

gum disease, BUT >> slows Parkinsons/Alzheimers/

aging, relieves allergies, boosts athletic performance

and immunity, aids weight loss ?!

Causes of Deficiency

• Significantly decreased levels of CoQ10 have

been noted in a wide variety of diseases in

both animal and human studies.

• CoQ10 deficiency may be caused by

1. Insufficient dietary CoQ10.

2. Impairment in CoQ10 biosynthesis.

3. Excessive utilization of CoQ10 by the body

• Increased body consumption of CoQ10 is the presumed

cause of low blood CoQ10 levels seen in excessive

exertion, hypermetabolism, and acute shock states.

4. or any combination of the three.

5. Drugs can cause CoQ deficiency

• HMG-CoA reductase inhibitors used to treat elevated blood cholesterol levels by

blocking cholesterol biosynthesis also block CoQ10 biosynthesis.

• The resulting lowering of blood CoQ10 level is due to the partially shared

biosynthetic pathway of CoQ10 and cholesterol.

Phytochemicals

Non-nutrient compound

Found in plant-derived foods and have biological activity

in the body.



Some categories:

Antioxidants (anti-aging/cancer/heart disease?) -

catechins (berries, green/black tea), lycopene

(tomatoes,watermelon), bioflavanoids (citrus

fruit, grapes), coenzyme Q (nuts,oils).

Estrogen mimics (cancer inhibitors?) -

indoles (broccoli, cauliflower), isoflavones

(legumes), lignans & omega-3- fatty acids

(canola, flaxseed), genistein (soy).

Lycopene = red pigment in tomatoes, red

grapefruit, watermelon

Plant Pigments = Potent Antioxidants



Almost 2000 known plant pigments. >800 flavonoids,

~450 carotenoids and ~150 anthocyanins.

Red (lycopene) – tomatoes, pink/red grapefruit,

watermelon, guava, red peppers.

Red/Blue/Purple (anthocyanins) – red/blue grapes,

blue-/straw-/raspberries, beets, cherries,

egg plant, plums, red cabbage.

Orange (carotene) – squash, pumpkin, yams,

carrots,

mango, cantaloupe.

Plant Pigments = Phytochemicals

Orange/Yellow (cryptoxanthin) – peach, orange,

nectarine, papaya.

Yellow/Green (lutein), zeaxanthin =

especially good for health of eyes) –

corn, cucumber skins, yellow/green

peppers, green beans/peas, spinach,

honeydew melon, kiwi, romaine lettuce,

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Check out the „Net‟ under „vitamin O‟

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