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Respiratory1 NSC

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Shared by: Marie Ruby
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Pulmonary Diseases & Disorders: Assessment EMS Professions Temple College Pulmonary Diseases & Disorders  Epidemiology  28% of all EMS Chief Complaints in the US  >200,000 deaths annually due to respiratory emergencies Pulmonary Diseases & Disorders  Many, many pulmonary diseases  Difficult to learn all pathophysiologies  All can be categorized as affecting:  Ventilation  Diffusion (Respiration)  Perfusion  Treatment can be focused on identifying and treating source of ventilatory/respiratory impairment Sources of Pulmonary Impairment    Pulmonary Diseases Disorders of the Pulmonary System Non-Pulmonary Disorders/Disease Impairing Ventilation or Respiration What examples can you list for each of these?  Sources of Pulmonary Impairment  Ventilation  Upper Airway Trauma  Epiglottitis  FBAO  Inflammation of tonsils   Lower Airway Trauma  Obstructive lung disease  Mucous accumulation  Smooth muscle spasm  Airway edema  Sources of Pulmonary Impairment  Ventilation  Chest Wall Impairment Trauma  Hemothorax  Pneumothorax  Empyema  Pleural inflammation  Neuromuscular diseases   Neurologic Control Brainstem dysfunction  Phrenic or spinal nerve dysfunction  Sources of Pulmonary Impairment  Diffusion Inadequate FiO2  Diseased alveoli   Interstitial space disease High pressure pulmonary edema  High permeability pulmonary edema  asbestosis  COPD  inhalation injury   Capillary bed disease  atherosclerosis Sources of Pulmonary Impairment  Perfusion  Inadequate blood  Impaired blood flow  pulmonary embolus volume or hemoblogin  Capillary wall  hypovolemia pathology  anemia  trauma Risk Factors for Pulmonary Disease  Intrinsic Risk Factors  Genetic predisposition  asthma  COPD  carcinoma  Cardiac or Circulatory pathologies for pulmonary edema  Source for pulmonary emboli  Stress  Source Risk Factors for Pulmonary Disease  Extrinsic Factors  Smoking  prevalence of COPD & carcinomas   severity of pulmonary disease  Environmental  Factors prevalence of COPD & asthma   severity of all obstructive disorders Function of the Pulmonary System  Gas Exchange System  ~10,000 liters of air are filtered, warmed and humidified daily  Oxygen diffused into blood  Carbon dioxide excreted from the body Function of the Pulmonary System  Physiology of Ventilation  Requires neurologic initiation (brainstem)  Nerve conduction pathways between brainstem and muscles of respiration  Intact & patent Upper and Lower airways  Intact & non-collapsed alveoli Function of the Pulmonary System  Physiology of Respiration  Simple diffusion process at the pulmonarycapillary bed  Diffusion Requirements  Intact, non-thickened alveolar walls  Minimal interstitial space & without additional fluid  Intact, non-thickened capillary walls Function of the Pulmonary System  Physiology of Perfusion  Process of circulating blood through the capillary bed  Perfusion Requirements  Adequate blood volume  Adequate hemoglobin  Intact, non-occluded pulmonary capillaries  Functioning Left Heart Control of Ventilation  Control ventilation in response to physiologic needs  Driven  2° 1° by pH of CSF largely by PaCO2  influenced drive = PaCO2  3° drive = PaO2 detected by chemoreceptors  very small population with severe COPD Nervous System Effect on Ventilation  Medulla  Stimulation to initiate ventilation of the diaphragm of intercostal muscles  Phrenic Nerve  Innervation  Spinal Nerves at Thoracic levels  Innervation  Hering-Breuer reflex  Prevents overinflation General Assessment  Size-Up  Environment  Airborne Hazards  Number of patients  Needs • Specialized rescue equipment • Protective equipment  Is the environment creating or exacerbating the pulmonary condition? General Assessment  Initial Goal  Identify potentially life-threatening pulmonary conditions  Perform minimal PE & Hx  Initiate immediate & appropriate therapies  Then, continue PE & Hx  Try to determine if origin is ventilation, diffusion, perfusion or combination General Assessment  Signs of potentially life-threatening pulmonary condition  altered mental status  absent signs of ventilation  Audible stridor or wheezing  Able to speak in short phrases only  Sustained Tachycardia  Pallor / Diaphoresis  Accessory muscle use / Retractions Assessment: H&P  Present History (focused hx)  Chief Complaint  Dyspnea • “Subjective sensation that breathing is excessive, difficult or uncomfortable  CP  Cough, Hemoptysis  Associated  Fever, Symptoms Chills   sputum production  Fatigue Assessment: H&P  Present History (focused hx)  Sputum  Findings amount of sputum  infection  Thick green or brown  pneumonia or infection  Yellow or gray  allergic or inflammatory response  Hemoptysis  tuberculosis or carcinoma  Pink, frothy  severe pulmonary edema Assessment: H&P  HX of Present Illness  How long has dyspnea been present?  Gradual or sudden onset?  What aggravates or alleviates?  Hx of orthopnea?  Coughing?  Productive cough?  What does sputum look/smell like?  Pain?  What does the pain feel like? Assessment: H&P  Listen - To Pt. Breathe or Talk Breathing is Obstructed Breathing  Not All Obstructed Breathing is Noisy  Snoring - Tongue Blocking Airway  Stridor - “Tight” Upper Airway from Partial Obstruction  Noisy  Observe Breathing  Tachypnea  Bradypnea Assessment: H&P  Observe  Body Positioning in dependent position  Tripod  Legs  Mental Status  Ventilatory Effort muscle use / retractions  Abdominal muscle use  Chest wall expansion  Nasal flaring, pursed lips  Accessory Assessment: H&P  Physical Exam of the Chest  Increased A-P Diameter  Lung Sounds  Abnormal: stridor, wheezing, rhonchi, rales, pleural rub expansion  Symmetrical Findings  Evidence of Trauma  Chest Assessment: H&P  Physical Exam  Cyanosis?  Late, unreliable sign of Hypoxia  Oxygenate  Immediately! Especially If: Decreased LOC  Possible Shock  Possible Severe Hemorrhage  Chest Pain  Chest Trauma  Respiratory distress or dyspnea  HX of any Kind of Hypoxia Assessment: H&P  Physical Exam  Vital Signs  Skin Color, Temp & Moisture  Respiratory Rate • No an accurate lone indicator of respiratory status unless very slow  Respiratory  Pulse Rhythm/Pattern • Bradycardia vs Tachycardia  Blood Pressure Assessment: H&P  Physical Exam - Circulatory assessment  Is the heart beating?  Is there major external hemorrhage?  Is the Pt. Perfusing vital organs?  Effects of hypoxia:  Early in adults - Tachycardia  Late in adults - Bradycardia  Children - Bradycardia Assessment: H&P   Don’t let respiratory failure distract you from assessing for circulatory failure. Vascular Access Assessment: H&P  Physical Exam  Extremities  Peripheral  Clubbing Cyanosis spasm  Peripheral edema  Carpopedal Assessment: H&P  Diagnostic Testing  Pulse oximetry Saturation  Inaccuracies & Disadvantages   Peak Flow Meter Baseline measurement for obstructive lung disease  Often available from patient   Capnometry real-time assessment of endotracheal tube placement  quantitative vs qualitative  Assessment: H&P  Past History  Similar Episodes in Past  Patient’s description of acuity  “What happened last time you had an episode this bad?”  Chronic Symptoms  Acute, Seasonal SOB episodes  Seasonal Allergies  Chronic cough  Recurrent flu, pulmonary infection or SOB Assessment: H&P  Past History  Known diagnosis  Does the present H&P correlate with this past history? • CHF • Hypertension • Renal Failure intubation or hospitalization  Aggravating Factors (e.g. smoking)  Previous Assessment: H&P  Past History  Medications  Class, Route, Frequency of Use  Pulmonary • • • • Sympathomimetics Corticosteroids MAST Cell Stabilizer Methylxanthines  Cardiovascular • Diuretics • Antihypertensives • Cardiac glycosides Assessment: H&P  Disability  Restlessness, anxiety, combativeness = HYPOXIA Until Proven Otherwise  Drowsiness, lethargy = HYPERCARBIA When the patient stops fighting, he is not necessarily getting Better!! Other Adventitious Sounds  Cough  Forced exhalation against partially closed glottis  Reflex response to mucosa irritation  Determine circumstances At work  Postural changes  Lying down   Productive vs non-productive Other Adventitious Sounds  Sneeze exhalation via nasal route  Clears nasal passages  Reflex response to mucosa irritation  Forced  Sigh  Slow, deep inspiration - Prolonged, audible exhalation  Reexpands areas of atelectasis Other Adventitious Sounds  Hiccough  Hiccups, singultus  Spasm of diaphragm followed by glottic closure  No useful purpose  Benign, transient
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