Hematologic Emergencies

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Hematologic Emergencies Sickle Cell Anemia  Pathophysiology – Genetic abnormality causes red cells to contain hemoglobin S rather than hemoblobin A – At low oxygen tensions hemoglobin S forms long, rigid rods – RBCs become distorted, sickle-shaped Sickle Cell Anemia  Pathophysiology – Sickled RBCs cannot pass through small vessels »Thrombosis »Infarction – Sickled RBCs are fragile »Hemolysis Sickle Cell Anemia  Incidence – Gene present in 10% of black population (sickle cell trait) – One in every 400 to 600 blacks has sickle cell anemia Sickle Cell Anemia  Incidence – Also occurs in populations from » Puerto Rico » Turkey » India » Middle East » Southern Europe » North Africa Sickle Cell Anemia  Incidence – Sickle cell gene survives by providing resistance to malaria » Two normal genes = malaria = death » Two sickle-cell genes = sickle cell anemia = death » One normal gene/one sickle-cell gene = survival – Persons with sickle cell trait survive, pass gene to offspring Sickle Cell Anemia  Thrombotic crisis (vasoocclusive-infarctive) – Precipitated by: » Cold exposure » High altitude » Dehydration » Infections – Gnawing pain in bones (tibias, back), joints, muscles, and abdomen Sickle Cell Anemia  Hemolytic Crisis – Rupture of sickled cells – Fall in hematocrit with jaundice Sickle Cell Anemia  Aplastic Crisis – Suppression of RBC production – Life threatening decrease in hematocrit Sickle Cell Anemia  Complications – Acute Respiratory Insufficiency »Thrombus formation »Pneumonia – CNS Involvement »Vascular occlusion »TIA, CVA Sickle Cell Anemia  Complications – Arthropathy »Aseptic necrosis »Hemarthosis »Septic arthritis – Priapism Sickle Cell Anemia  Complications – Renal infarction »Flank pain »Hematuria – Narcotic drug addiction Sickle Cell Anemia  Management – Oxygen – IV fluids – Analgesia Hemophilia  Cause – Inherited, sex linked recessive gene – Produces clotting factor deficiencies »Hemophilia A (Factor VIII) »Hemophilia B (Factor IX) Hemophilia  Signs – Swollen, painful joints – Local bleeding out of proportion to injury – Subcutaneous bleeding – Bleeding from mucous membranes – Abdominal pain, distension – Hematemesis, melena Hemophilia  Management – Standard techniques for hemorrhage/ hypovolemia – Apply ice packs to hemarthoses/ bleeding sites – In hospital, administration of »clotting factor »cryoprecipitate, or »fresh frozen plasma Disseminated Intravascular Coagulation (DIC)  Pathophysiology – Damage tissue, blood vessels, blood cells – Clotting mechanism activation – Excess clotting »Diffuse microthrombi »Consumption of clotting factors DIC  Pathophysiology – Split fibrin products from clots inhibit clotting – Clotting factor consumption, split fibrin products lead to: »Hemorrhage »Shock DIC  Causes – Gram negative sepsis – Abruptio placentae/missed abortions – Burns – Heat Stroke – Shock – Transfusion reaction with hemolysis DIC  Signs/Symptoms – Petechiae and ecchymosis – Bleeding from mucus membranes, orifices, venipuncture sites – Blood in urine, stool, vomitus, sputum – Signs/Symptoms of hypovolemia DIC All patients with predisposing conditions or who are critically ill should be monitored for signs of prolonged clotting or of bleeding DIC  Management – Control bleeding – Treat hypovolemia – Fresh frozen plasma or cryoprecipitate to inhibit bleeding, activate normal clotting – Identify and treat cause DIC  Management – Heparin to inhibit additional clotting (controversial) – Antifibrinolytic Agents (Amicar) to inhibit fibrinolysis and formation of split fibrin products (controversial) Anticoagulant Use with Hemorrhage    Control bleeding/treat hypovolemia with standard techniques Use ice packs to supplement pressure dressings Antidotes – Coumadin » Vitamin K, fresh frozen plasma – Heparin » Protamine sulfate Leukemia  Definition – Neoplastic disorder of blood cell forming tissues – Causes uncontrolled production of white cells Leukemia  Clinical manifestations – Decreased RBCs – Bleeding (decreased platelets) – Infection »WBC immaturity »Chemotherapy Leukemia  Clinical manifestations – Bone pain – Liver, spleen, lymph node enlargement – Anorexia Leukemia  Management – Oxygen – IV – Analgesia – Infection control procedures

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