Neurological Emergencies
Coma, Seizures, Syncope, Stroke
Temple College EMS Professions
Coma
State of unconsciousness from which patient cannot be aroused
Coma
Unconsciousness = Immediate Life Threat
Loss of airway Aspiration
Coma
Management of ABC’s must come before investigation of cause
Airway
Open, clear, maintain If trauma present or no history available, immediately control C-spine
Breathing
Assess presence, adequacy High concentration O2 immediately on all patients with decreased LOC Assist if respiratory rate, tidal volume inadequate
Circulation
Pulses? Perfusion?
After ABC’s stabilized. . .
Quickly investigate cause DERM
D = Depth of coma
What does patient respond to? How does he respond?
E = Eyes
Pupils equal, dilated, constricted, Responsive to light? How?
R = Respiratory pattern
Rate? Unusually deep or shallow? Altered pattern?
M = Motor Function
Evidence of paralysis? Movement on stimulation? How?
Vital Signs
Shock? Increased ICP? Arrhythmias?
Head to Toe Survey
Injuries causing coma? Injuries caused by fall? What do the scene, bystanders tell you?
Possible Causes
Not enough oxygen Not enough sugar Not enough blood flow to deliver O2, sugar Direct brain injury
Structural (trauma) Metabolic (toxins, infections, temperature)
Possible Causes
Alcohol Epilepsy Insulin Overdose Uremia (and other metabolic causes)
Trauma Infection Psychiatric Stroke, syncope
Management
Secure airway Protective reflexes may be lost Immobilize spine unless absolutely certain injury not present Spinal injury not suspected - patient on left side
Management
High concentration O2 Assist ventilation as needed Monitor neurological/vital signs every 5 minutes
Management
Protect patient’s eyes on long transports (tape shut, moist pads) Patient may hear, understand even though unable to respond Treat, reassure accordingly
Seizures
Episodes of uncoordinated electrical activity in brain Signs/symptoms depend on area involved
Epilepsy
Tendency to have repeated episodes of seizure activity
Seizure Types
Grand mal (major motor) Petit mal (absence) Focal motor (simple partial) Psychomotor (complex partial)
Grand Mal Seizure
Aura
Sensation coming before convulsion Patient may recognize as sign of impending seizure May help locate origin of seizure in brain
Grand Mal Seizure
Convulsion
Loss of consciousness Tonic phase - rigidity Clonic phase - rhythmic jerking, incontinence, ineffective breathing
Grand Mal Seizure
Post-ictal Phase
Exhaustion Drowsiness Headache Possible hemiparesis (Todd’s paralysis)
Petit Mal Seizure
Loss of consciousness No loss of postural tone More common in children
Focal Motor Seizure
Rhythmic jerking of limb, one side of body No loss of consciousness
Psychomotor Seizure
Loss of consciousness Sterotyped movements (automatisms)
May look purposeful, but aren’t Lip smacking, movements of hands
May be called in as “drunk”, “O.D.”, “psych patient”
Generalized Seizure Management
During seizure
Remove from potential harm Do not forcibly restrain Roll on side Avoid putting anything in mouth
Generalized Seizure Management
After seizure ends
Assess ABC’s Clear airway Most common cause of seizure deaths is post-ictal airway loss
Generalized Seizure Management
High concentration O2 - immediately!! Assist breathing if ventilation inadequate
Generalized Seizure Management
Obtain history/physical
Trauma that could have caused, been caused by seizure Anti-seizure medications
Neuro/vital signs every 5 minutes If patient ventilating adequately, transport on left side
Seizures
Anything that injures brain can cause seizures (AEIOU/TIPS) Do not assume seizures are due to idiopathic epilepsy until proven otherwise
Status Epilepticus
> 2 seizures without intervening conscious period Immediate Life Threat Management
Secure airway Assist breathing with O2 Transport Request ALS intercept
Syncope
Fainting Sudden, temporary loss of consciousness Caused by lack of blood flow to brain
Causes
Stress, fright, pain (vasovagal syncope)
Orthostatic hypotension (BP fall on standing)
Decreased blood volume Increased size of vascular space
Decreased cardiac output Prolonged forceful coughing
Management
ABCs Keep
patient supine, elevate lower extremities Oxygen Assess underlying cause
CVA
Cerebrovascular Stroke
accident
CVA
Damage of portion of brain due to interruption of blood supply Mechanisms
Thrombosis Hemorrhage Embolism
Thrombosis
Blockage of vessel by thrombus Usually forms at area narrowed by atherosclerosis Typically in older persons Frequently occurs during sleep
Hemorrhage
Vessel ruptures Associated with hypertension, aneurysms of cerebral blood vessels Usually characterized by
Sudden onset Severe signs, symptoms
Embolism
Blood clots, plaque fragments travel through vessel; lodge, block flow Often associated with:
Atherosclerosis of carotids Chronic atrial fibrillation
Signs/Symptoms
Alterations in consciousness
Altered affect Confusion Dizziness Coma
Signs/Symptoms
Localizing signs
Paralysis Loss of sensation Loss of speech Unilateral blindness Loss of vision in half of visual field of both eyes Unequal pupils
Signs/Symptoms
Seizures Headache Stiff neck
Transient Ischemic Attacks
TIAs “Little strokes” Produce deficits that resolve completely in <24 hours Frequently precede CVA
Management
Assess ABC’s Protect airway High concentration O2 Vital signs every 5-10 minutes Note increased BP, irregular pulse
Management
Nothing by mouth Avoid rough handling Transport paralyzed side down Guard your conversation Patients who cannot speak may still understand!
Management
CVAs caused by thrombus, embolus may be reversible with thrombolytics (clot busters) Early recognition, rapid transport to appropriate facility is critical