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Electrolyte Emergencies

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Electrolyte Emergencies Hyponatremia Decreased serum sodium Na+ <135 mEq/L Hyponatremia  Causes Loss of salt and water Diuretics Addison’s disease Osmotic diuresis (DKA) Vomiting Diarrhea Diaphoresis Hyponatremia  Causes Increase in body water (dilutional hyponatremia) Excess water intake (water intoxication) CHF Renal failure Cirrhosis Hyponatremia  Signs/Symptoms apathy secondary to cerebral edema Headache Muscle twitching, cramps Seizures Na+ < 110 mEq/L Lethargy, Hyponatremia  Treatment NS infused at a rate to restore normal sodium over 48 hours Hypernatremia Elevated serum sodium Na+ >150 mEq/L Hypernatremia  Causes water loss Diabetes insipidus Pure solute gain NaHCO3 administration Pure Hypernatremia  Causes Water loss > Solute loss Diarrhea Vomiting Sweating Osmotic diuresis Hypernatremia  Signs/Symptoms Irritability, Coma Intracranial restlessness hemorrhage Blood sludging - thrombosis Seizures Thirst, dry mucus membranes, poor skin turgor Hypernatremia  Treatment Infusion of D5W (free water) to correct water deficit over 48 hours Hypokalemia Decreased serum potassium K+ < 3.5 mEq/L Hypokalemia  Causes Gastrointestinal Diarrhea losses Vomiting suction Urinary losses Diuretics Osmotic diuresis NG Hypokalemia  Causes Intracellular Alkalosis shifts agents Theophylline Inadequate intake  Hypokalemia  Signs/Symptoms hyporeflexia, paresthesias Decreased GI motility Nausea, vomiting Increased sensitivity to digitalis Weakness, Hypokalemia  Signs/Symptoms changes Flat to inverted T waves U waves ST segment depression Cardiac arrhythmias Ventricular ectopy V-Fib EKG Hypokalemia  Treatment underlying cause 10 mEq KCL IV over 1 hour, repeated until serum K+ > 3.5 mEq/L Correct Hyperkalemia Elevated serum potassium K+ > 5.5 mEq/L K+ > 6 mEq/L - Emergency K+ > 7 mEq/L - Life Threat Hyperkalemia  Causes Decreased excretion Renal failure Addison’s disease  - blockers Hyperglycemia Acidosis Hyperkalemia  Causes Increased intake Crush injury, burns, sepsis Massive hemolysis Blood transfusion High-dose penicillin Increased oral intake Hyperkalemia  Signs/Symptoms weakness Numbness, tingling EKG changes peaked T waves Broadening of QRS; decreased P wave size “Sine wave” leading to V-Fib then asystole Tall, Muscle Hyperkalemia  Treatment - 0.5 - 1.0g Antagonizes cardiac toxicity NaHCO3 - 50 mEq Drives K+ into cells D50W (25g)/insulin (10 units regular) Drives K+ into cells Calcium Hypocalcemia Decreased serum calcium Ca2+ < 8.5 mg/100ml Hypocalcemia  Causes Hypoparathyroidism D deficiency Decreased dietary intake Alkalosis Massive blood transfusion Citrate toxicity Vitamin Hypocalcemia  Signs/Symptoms Paresthesias Hyperreflexia, carpopedal spasm, tetany Seizures Trousseau’s sign: Carpopedal spasm following BP cuff compression of arm Hypocalcemia  Signs/Symptoms sign Contraction of facial muscles when face is tapped at angle of jaw Laryngospasms Prolonged QT interval Increased ventricular irritability Chvostek’s Hypocalcemia  Management chloride 1g over 10 - 20 minutes Overly rapid administration of calcium can cause bradycardia, hypotension, and cardiac arrest Calcium Hypercalcemia Increased serum calcium Ca2+ > 10.5 mg/100ml Hypercalcemia  Causes Malignancies breast, lungs Hyperparathyroidism Vitamin D toxicity Addison’s disease Milk/alkali syndrome Skeletal, Hypercalcemia  Signs/Symptoms fatigue, depression, coma Muscle weakness Hypertension Anorexia, nausea, abdominal pain Apathy, Hypercalcemia  Signs/Symptoms stones Short QT interval, heart block, cardiac arrest “Bones, Stones, Hypertones, Abdominal moans” Kidney Hypercalcemia  Treatment - 2 liters NS over 1 hour Decrease Ca2+ reabsorption Furosemide 40 mg IV Inhibits Ca2+ reabsorption Calcitonin Directly lowers Ca2+ 1

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