The CHEST
Emergency Medical Conditions
MI Zucker, MD
A dr Z Lecture
The WHAT:
• • • • • Normal chest films Abnormal patterns Atelectasis Infection Obstructive airway diseases • • • • • Heart failure Noncardiac edema Pulmonary embolism Aortic dissection …and a few others
The WHY:
• Give you a basic approach to acute medical diseases emphasizing CHEST Radiographs • Show you the most COMMON diseases • Show you commonly MISSED findings
The NORMAL Chest
PA/lateral Portable AP
The PA (adult and kid) and Lateral
• Check list • Commonly overlooked areas
The PORTABLE AP
• The “bottom feeder” of chest radiology
Pitfalls in Chest Radiology
• • • • Phase of respiration Position Comparison films Portables
Poor Inspiration
The PATTERNS
Too dense Too lucent
Location, Location, Location
Lung? Chest wall? Mediastinum? Pleura?
Lung: Too DENSE
• Alveolar pattern • Interstitial pattern • Masses
Lung: Alveolar Pattern
• Something of unit/soft tissue/water density replaces the air in the alveolar ducts, alveolar sacs and the alveoli
Alveolar Lung Pattern: causes
• • • • • • PUS WATER BLOOD Lymphoma BAC Alveolar proteinosis
Alveolar Lung Pattern: findings
• • • • Increased density Confluence Ill defined margins Air bronchograms
Lung: Interstitial Pattern
Something thickens the interstitium of the lung parenchyma
What?
• • • • • Edema Inflammatory cells RBC’s Malignant cells Fibrosis
How?
All of them ADD tissue to the peripheral and axial interstitium of the secondary pulmonary lobule
Secondary Pulmonary Lobules
Who?
• Many, many diseases present with the same interstitial patterns • You need history, lab, and frequently biopsy to make a specific diagnosis
A Memory Aid
“I Munch Ice Chips In Places Called Igloos”
Interstitial diseases
• Idiopathic
UIP DIP LIP BOOP LAM PEG sarcoid
• Malignancy
Metastases, lymphoma
• Infection
Viral, PCP, mycoplasma. Fungi, TB, MAC
Interstitial diseases
• Congenital NF TS CF
• Iatrogenic
Drugs, radiation
• Pulmonary edema
Cardiogenic, renal, noncardiogenic
Interstitial diseases
• Collagen-vascular RA, SLE, scleroderma, AS
Allergic alveolitis, noxious gases, pneumoconiosis
• Inhalational
The Interstitial Patterns
• • • • Lines: fine, medium, or coarse Nodules: tiny to 3 cm Reticular: network of crossing lines Reticular-nodular: lines and nodules
Lines
Nodules
Reticular pattern
Lines and nodules
What do they mean?
• Coarse lines mean fibrosis, also called “honeycomb” pattern • The other patterns usually mean more active disease, but aren’t specific
Kerley lines, A and B
• Thickened secondary lobule septae • Often, but not always due to CHF • Basically, they are just a slightly specialized intertstitial linear pattern • A and B differ only by location
INFECTION
The pneumonias
BACTERIAL PNEUMONIAS
Pyogenic
The Silhouette Sign
• If two adjacent structures have the same density, the border between them is not visible. Replace air with an alveolar process and the border between the involved lung and the heart, or diaphragm, or aorta disappears
The Spine Sign
• On the lateral view the spine normally progressively looks darker caudally. If it looks whiter, there is an alveolar process in one of the lower lobes.
Atypical Pneumonias
• Mycoplasma • Chlamydia
Patterns
• Diffuse bilateral patchy opacities • Diffuse interstitial linear opacities
Viral Pneumonias
• Air-trapping • Mucus plugs and atelectasis • Diffuse interstitial linear and nodular opacities • Findings more pronounced in kids
Pneumocystis carnii Pneumonia
• Interstitial linear nodular pattern, usually bilateral • Followed by diffuse alveolar pattern • Early, 10% of CXR’s in PCP can be negative. Later, atypical patterns are fairly common.
PCP
Tuberculosis
• Primary • Post-primary
TB: primary
TB: postprimary, early
TB: postprimary, cavitary
A few more Infections
• Lung abscess • Empyema • Fungus
Lung abscess
Empyema: Hydro-pneumothorax
Coccidioidomycosis
ATELECTASIS
Loss of Lung Volume
Atelectasis: types
• • • • • Obstructive Passive Compressive Cicatricial Adhesive
Atelectasis: signs
• • • • • • • Increased density Shift of fissure Elevation of diaphragm Shift of mediastinum Shift of heart Shift of hilum Compensatory hyperinflation
Subsegmental
Right upper lobe
Lower lobes
Right middle lobe
Left upper lobe
ATX: entire lung
Edema
Cardiogenic Renal Noncardiogenic
Edema: pathogenesis
• Cardiogenic: increased hydrostatic pressure • Noncardiogenic: increased alveolarcapillary membrane permeability • Renal: multiple factors
Cardiogenic
Congestive heart failure
CHF
• Cephalization 12 wedge pressure • Interstitial edema 20 • Alveolar edema 25
• Cardiomegaly, pleural effusions
CHF: cephalization
CHF: interstitial edema
CHF: alveolar edema
Renal related
• Fluid overload • Increased permeability • CHF
Edema: renal
Noncardiogenic edema
• • • • • • Near drowning High altitude Drugs Inhalation Hypoxia (ARDS)
Noncardiogenic edema
Obstructive lung disease
Asthma COPD
Asthma
• • • • Hyperinflation Mucus plugs/atelectasis Interstitial inflammation Barotrauma
Asthma: kid
Asthma: adult
COPD
• • • • • Hyperinflation Flat diaphragm Increased retrosternum air space Pulmonary arterial hypertension Look for pneumonia as cause of exacerbation
COPD
Pulmonary embolism
PE: diagnosis
• • • • • • • Clinical: dyspnea, chest pain, increased RR & PR D-dimer Doppler ultrasound *CXR *CTPA Lung scan Pulmonary angiography
Chest film
• • • • • Subsegmental atelectasis Small pleural effusion Elevated diaphragm Westermark’s (rare) Hampton’s (rare)
PE: CXR
PE: CTPA
Aortic Dissection
Aortic Dissection
• • • • HYPERTENSION Marfans Coarctation Turners, SLE, pregnancy
Aortic Dissection
• Type A: more common, ascending aorta, surgery • Type B: descending aorta, trial of medical management
AD: imaging
• • • • • CXR CTA MRI TEE Angiography
AD: CXR
• Mediastinum contour abnormality: abnormal shape or width • A change in contour from previous film
AD: CXR
Sensitivity: 80%
AD: CXR
AD: CXR
AD: CTA axial
AD: CTA reformat
…and a few more
Sickle Cell Disease Cystic Fibrosis
Sickle Cell Disease
Cystic Fibrosis
Goodbye
• Copyright 2004 MI Zucker, MD
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