Presentation, Heritability, and Genome-Wide Linkage Analysis of the Midchildhood Growth Spurt in Healthy Children from the Fels Longitudinal Study

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Presentation, Heritability, and Genome-Wide Linkage Analysis of the Midchildhood Growth Spurt in Healthy Children from the Fels Longitudinal Study
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Presentation, Heritability, and Genome-Wide Linkage Analysis of

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the Midchildhood Growth Spurt in Healthy Children from the

4 Fels Longitudinal Study

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6 bradford towne, 1,2 kimberly d. williams, 1 john blangero, 3 stefan a.

7 czerwinski, 1 ellen w. demerath, 4 ramzi w. nahhas, 1 thomas d. dyer, 3

8 shelley a. cole, 3 miryoung lee, 1 audrey c. choh, 1 dana l. duren, 1,5

9 richard j. sherwood, 1,6 william cameron chumlea, 1,2 and roger m.

10 siervogel 1,2

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12 Abstract Growth is a complex process composed of distinct phases over the

course of childhood. Although the pubertal growth spurt has received the most [First Page]

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attention from auxologists and pediatricians, the midchildhood growth spurt

14 [623], (1)

has been less well studied. The midchildhood growth spurt refers to a rela-

15 tively small increase in growth velocity observed in some, but not necessarily

16 all, children in early to middle childhood. If present, the midchildhood growth

17 spurt typically occurs sometime between the ages of 4 and 8 years, well be- Lines: 0 to 42

18 fore the onset of the far more pronounced pubertal growth spurt. In this study ———

19 we used a triple logistic curve-fitting method to fit individual growth curves 0.89745pt PgVar

20 to serial stature data from 579 healthy participants in the Fels Longitudinal ———

21 Study, 479 of whom have been genotyped for about 400 short tandem repeat Long Page

22 (STR) markers spanning the genome. We categorized individuals according to

the presence or absence of a midchildhood growth spurt and then conducted PgEnds: TEX

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heritability and genome-wide linkage analyses on the dichotomous trait. In

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the total sample of 579 individuals, 336 (58%) were found to have evidence

25 of having had a midchildhood growth spurt. There was a marked sex differ-

[623], (1)

26 ence in presence of the midchildhood growth spurt, however, with 232 of the

27 293 males (79%) having had a midchildhood growth spurt but just 104 of

28 the 286 females (36%) having had one. Presence of a midchildhood growth

29 spurt was found to have a significant heritability of 0.37 ± 0.14 ( p = 0.003).

30 Two quantitative trait loci with suggestive LOD scores were found: one at 12

31 cM on chromosome 17p13.2 (LOD = 2.13) between markers D17S831 and

32 D17S938 and one at 85 cM on chromosome 12q14 (LOD = 2.06) between

33 markers D12S83 and D12S326.

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Lifespan Health Research Center, Department of Community Health, Boonshoft School of Medicine,

35 Wright State University, 3171 Research Blvd., Dayton, OH 45420.

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Department of Pediatrics, Boonshoft School of Medicine, Wright State University, Dayton, OH.

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