Nursing care and interventions for Neurological Disorders
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Powerpoint for Nursing care and interventions for Neurological Disorders.
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Neurological Disorders
Adult Health I
Seizure Disorders
Episodes of abnormal motor, sensory,
autonomic or psychic activity ( or any
combination) that result from sudden
excessive discharge from cerebral neurons
May be brought on by part of the brain or
all of the brain may be involved
Causes of Seizures
Idiopathic
Cerebrovascular disease
Hypoxemia
Fever (childhood)
Hypertension
Head injury Acquired
CNS infections
Metabolic and toxic conditions
Brain Tumor
Drug / alcohol withdrawal
Allergies
Types of Seizures
Simple Partial
– No LOC
– May produce abnormal sensations, such as an
unpleasant smell, or a motor movement, such
as jerking of the arm
Complex Partial
– Likely has impairment of consciousness
Partial Seizures Secondarily Generalized
Types of Seizures
Generalized
– LOC usual
– Sx bilaterally symmetric
– Absence: Previously known as petit mal, triggers a
short lapse in consciousness. Most often seen in
children
– Atonic: Previously known as a drop attack, causes a
complete loss of muscle control and results in
collapse
– Myoclonic: Triggers sudden jerking in the muscles,
often in the arms and legs
– Tonic-clonic: Triggers a fall to the ground (tonic
phase) followed by jerking movements (clonic phase)
Absence Seizure
http://video.google.com/videoplay?docid=2673074961833568919
&total=35&start=10&num=10&so=0&type=search&plindex=2
Medical Management
Pharmacologic Therapy
– May need more than 1 medication to control
– Some drugs more effective for certain types
of seizures
– Can control up to 70-80% of all seizures
– Meds may need to be adjusted during illness,
weight changes or increases in stress
– Side effects are problematic and can be
frequent
Medical Management
Tegretol
Lamictal
Dilantin
Phenobarbital
Keppra
Topamax
Depakote
Neurontin
Ativan
Klonopin
Medical Management
Common side effects of anti-seizure
medications
– Sedation, dizziness, fatigue, gi complaints,
vision disturbances, weight gain, gait
disturbances
Toxic effects of anti-seizure medications
– Blood dyscrasias
– Hepatitis and/or hepatotoxicity
– Skin rashes
– Nephrolithiasis
Surgical Management
Excision of lesion
causing seizure
Vagus Nerve
Stimulator-
implantation of
generator to deliver
electrical impulses to
the brain
Nursing Care During a Seizure
Observe and record findings
– Circumstances prior to seizure
– Aura
– Description of behaviors (gaze, movements, head and
body position, etc)
– Pupillary response
– Presence/absence of automatisms
– Incontinence
– Unconsciousness
– Cognitive function
– Weakness after seizure
– Sleeping after seizure
Nursing Care During a Seizure
Prevent complications during seizure
– Turn the patient to the side
– Do not force anything into the mouth
– Suction if needed
– Pad siderails
– Bed in low position
– Loosen constrictive clothing
– Provide privacy
Nursing Care During a Seizure
Documentation example:
11/6/07 3:15pm – Pt reported hearing buzzing in right ear 30
seconds prior to onset of seizure. During seizure, pt was
unconscious, head turned toward the right with bilateral tonic-
clonic movements of upper and lower extremities and lip
smacking. Eyes open and deviated to the right. Pupils fixed at
6mm bilaterally. Pt was incontinent of urine. Seizure lasted
approximately 2 minutes. Vital signs at termination of seizure: p-
100, r-22, bp-100/68. Pt lethargic, speech garbled at termination
of seizure and pt fell asleep. Safety measures implemented.
Nurse remained with pt for duration of seizure with no evidence
of injury. Dr Jones notified of events. No new orders received.
Joann Paoletti, RN.
4:00pm- pt awake, alert, oriented x3. Has no recollection of events
occurring after aura. Joann Paoletti, RN.
Nursing Management
Pt / family education
– Medication use, dose, side effects
– What to do in event of seizure
– Avoidance of triggers
Lifestyle changes
Coping skills and stress management
Monitoring for complications
Stroke
“Brain Attack”
Main Types:
– Ischemic
Sudden loss of function due to disrupted blood flow to the
brain leading to brain cell injury and death
Caused by: thrombus, embolus, atherosclerosis
– Hemorrhagic
Sudden loss of function due to rupture of cerebral vessel,
thus reducing blood flow
Caused by: uncontrolled hypertension or aneurysm
– Stroke cause by other problems
Illicit drug use, migraine, coagulopathies, etc
Transient Ischemic Attack (TIA) – stroke sx
which resolve <24h
Clinical Manifestations
Numbness or weakness of the face, arm
or leg, esp on 1 side of the body
Confusion or change in mental status
Trouble speaking or understanding speech
Visual disturbances
Difficulty in walking, dizziness, or loss of
balance or coordination
Sudden severe headache
Types of Deficits
Motor Deficits
Hemiparesis- weakness of 1 side of the body
Hemiplegia- paralysis of 1 side of the body
Ataxia- gait disturbance
Dysarthria- difficulty in speaking
Dysphagia- difficulty in swallowing
Sensory Deficits
Parasthesia- numbness or tingling
Types of Deficits
Verbal Deficits
Expressive aphasia- unable to form
words
Receptive aphasia- unable to
comprehend the spoken word
Global aphasia- mixed with expressive
and receptive features
Types of Deficits
Cognitive Deficits Emotional Deficits
Short and long term Loss of self-control
memory loss Emotional lability
Decreased attention Decreased tolerance
span to stress
Impaired ability to Depression
concentrate Withdrawal
Poor abstract
Fear, hostility, anger
reasoning
Feelings of isolation
Altered judgment
Comparison of Left
and Right Sided Strokes
Left Hemispheric Stroke
Right sided weakness
Right visual field deficit
Aphasia (expressive, receptive
or global)
Altered intellectual ability
Slow, cautious behavior
Right Hemispheric Stroke
Left sided weakness
Left visual field deficit
Spatial-perceptual difficulties
Increased distracability
Impulsive behavior and poor
judgement
Lack of awareness of deficits
Assessment and Diagnostics
Complete h/p with detailed neuro
assessment
Non-contrast CT of Head
12 lead ekg
Carotid ultrasound
Echocardiogram
MRI/MRA
Stroke Risk Factors
Modifiable Nonmodifiable
Hypertension Advanced age
Hyperlipidemia Male gender
Obesity African-American
Smoking ethnicity
Diabetes
Periodontal disease
Excessive alcohol use
Medical Management
Primary Prevention
– Identify those at risk and educating them about
lifestyle changes
– Low dose aspirin
Secondary Prevention
– Warfarin
Goal to keep INR 2-3
– Antiplatelet therapy
Plavix 75mg/day
– Statins
– HCTZ and ACEI for BP control
Medical Management
Thrombolytics to destroy clot
– Must be given within 3h of onset of sx
Carotid Endarterectomy
– Removal of atherosclerotic plaque
Carotid Stenting
– Stent placed in carotid artery to keep vessel open
Craniotomy with evacuation of bleed and repair
of aneurysm
Ventriculostomy
– Drain to manage ICP
Nursing Management
Improve joint mobility and prevent joint
deformities
– Establish exercise program
– Position arm to prevent shoulder adduction
– Position hand and fingers
– Change positions
– Prevent shoulder pain
Nursing Management
Enhance self-care
– Use assistive devices
Prepare for ambulation
– Work on sitting, then standing
– Position pt to use “good” side to help with transfer
Promote adequate nutrition
– Assist with feedings as needed
– Speech therapy referral
– Change consistency of food/fluids
– May need tube feedings if dysphagia severe
Nursing Management
Manage sensory-perceptual difficulties
– Use visual stimuli
– Keep stimuli on side where vision is intact
– Encourage involvement of affected side
Attain bowel/bladder control
– Toileting schedule
– Kegel exercises
– Intermittent cath
– High fiber diet with at least 2l fluid/day
Nursing Management
Improve thought processes
– Reality orientation
– Cueing
– Provide positive feedback
Improve communication
– Speak slowly and clearly
– Allow the patient to speak
– Use posted schedules to aid with daily events
– Repetition
– Use gestures, pictures, etc
– Give 1 instruction at a time
– Talk to the patient
Nursing Management
Maintain skin integrity
– Turn and position
– Pressure reducing devices
– Keep skin clean and dry
Improve patient/family coping
– Encourage verbalization of feelings
– Incorporate pt/family into care
Promote community based care
– Refer to home care agency
– Reinforce need for secondary prevention measures
Parkinson’s Disease
Slowly progressive neurological movement
disorder
There is decreased dopamine, thus allowing
acetylcholine to be unchecked resulting
imbalance which affects voluntary movements
Leads to disability
Sx usually appear in 50s or later
Affects more men than women
Cause unclear http://video.google.com/videoplay?docid=77
70259655554714650
Clinical Manifestations
Tremors
Rigidity
Bradykinesia
Postural instability
Shuffling gait
Micrographia
Dysphonia
Mask-like facies
Other Manifestations
Dementia
Dysphagia
Constipation
Urinary retention
Sleep disturbances
Depression
Psychosis
Assessment and Diagnostics
Dx based on clinical presentation with at
least 2 of the following:
– tremor
– Rigidity
– Bradykinesia
– Postural changes
PET scan may be helpful
Medical Management
Anti-Parkinson meds
– Replace dopamine (Sinemet)
– ? Whether or not they advance disease and promote dyskinesia
and “on-off” effects
Anticholinergic meds
– Cogentin
– Can be used with dopamine agents
Antiviral meds
– Symmetrel
– Exact mechanism unknown
Dopamine Agonists
– Parlodel, Requip, Mirapex, etc
– Work by prolonging availability of dopamine by stimulating
dopamine receptors
Medical Management
Monoamine Oxidase (MAO) Inhibitors
– Eldepryl
– Inhibits dopamine breakdown
– Drug-food interactions
COMT Inhibitors
– Comtan
– Useful adjunct medication
– Blocks enzyme that breaks down levodopa
Antidepressants
– TCAs (Elavil) often used due to anticholinergic and
antidepressant effects
– Some SSRIs can aggravate Parkinson’s
Antihistamines
– Used for anticholinergic and sedative effects
Medical Management
Sterotactic Surgery
– Part of nerve pathway permanently destroyed, thus
alleviating sx
Neural Transplantation
– Stem cell transplantation
http://stemcells.nih.gov/info/basics/defaultpage.asp
– Creates ethical issues
Deep Brain Stimulation
– Pacemaker like device surgically implanted
Nursing Management
Improve mobility
Enhance self-care activities
Improve bowel elimination
Improve nutrition
Enhance swallowing
Improve communication
Support coping abilities
Promote use of community resources
Multiple Sclerosis
Immune mediated progressive
demyelinating disease of the CNS
Destruction of myelin sheath interferes
with transmission of nerve impulses
More women affected
Dx between ages 20-50
Clinical Manifestations
Fatigue
Depression
Loss of balance
Pain
Weakness
Numbness
Poor coordination
Blurred or double vision
Spasticity
Other Manifestations
Bladder dysfunction
Bowel dysfunction
Pressure ulcers
Dysphagia
Dysphonia
Sexual dysfunction
Depression
Types of MS
Assessment and Diagnostics
Complete h/p with detailed neuro hx
MRI
Spinal tap
Medical Management
MS Drugs
– Rebif, Betaseron
– Copaxone, Avonex
– Solumedrol
– Novantrone
Symptom Management
– Baclofen
– Zanaflex
– Benzodiazepines
Nursing Management
Promote physical mobility
– Exercise
– Minimize spasticity and contractures
– Rest periods
Prevent injury
Enhance bowel and bladder control
– Routine toileting
– Intermittent catheterization
– Bowel training program
Nursing Management
Enhance communication and swallowing problems
– Speech therapy referral
– Alter consistency of food/fluids
– Assist with meals
– Monitor for aspiration
Improve sensory/cognitive function
– Eye patch for double vision
– Prism glasses
– Provide support and assist with coping skills
Improve home management
– Environment handicap equipped
– Assistive devices
Case Study
BD is a 70y AAF who lives in an assisted living facility. She
has a hx of HTN and CAD. She woke up in the morning
with right sided weakness and slurred speech. On
admission to the hospital, cardiac rhythm- atrial
fibrillation. P- 98, R- 20, BP 136/88. She is awake and
alert. Speech is garbled and she cannot follow one step
directions. She has paralysis of her right side.
1. What diagnostic tests might be done?
2. What is likely going on?
3. What medical treatments might be appropriate?
4. What nursing interventions would be appropriate?
5. Can this patient return to the ALF after discharge?
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