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Surgical management of Hydrocephalus

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Shared by: Sachin Kandhari
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9/12/2008
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Surgical management of Hydrocephalus Historical Background      The first shunt was a nail of glass wool communicating the lateral ventricle and the subgaleal space. Cushing – transvetebral cannulae draining the lumbar theca into the peritoneum Dandy- open third ventriculostomy. Yorkildsen- 1939: ventriculocisternostomy John Holter -1955: slit  Ventriculoperitoneal shunt: Scott (1955) Alternatives  Excision of the obstructive lesion Posterior fossa tumour  Thalamic tumours  Choroid plexus tumours  Congenital lesions:   Aqueductal  ACM stenosis cyst  Dandy walker  Arachnoid  CSF diversion Ventriculoperitoneal  Ventriculo atrial  Ventriculo pleural  Theco peritoneal  Endoscopic third ventriculostomy    Hydrocephalus should be treated if it is progressive or symptomatic Contraindications of shunt : Ventriculitis  Other inadequately treated infections  Acute intraventricular hemorrhage  Ventriculomegaly without elevated intracranial pressure  Extreme brain pathology( hydrancephaly)  V-P shunt  Contraindications : Active abdominal infections  Diffuse peritoneal adhesions  Ventriculo pleural shunt  Absolute contraindications: Active pulmonary infections  Preexisting pleural effusions or adhesions   Relative contraindications: Infancy  Latent disease of pleura  CRF,CHF, Connective tissue disorder  Ventriculo atrial  Not favoured : Technically more demanding  Revision difficult and more often required  Complications more serious  Lumbo peritoneal  Indications : Communicating hydrocephalus  Pseudotumour cerebri  CSF fistula  Shunt systems  System characteristics that minimize dhunt complications( Saint rose et al ): One piece system  Nonflanged ventricular catheter  Proximal nonslit valve  Open ended distal tubing  Valves   Differential pressure valves Variable resistance flow regulated valve. Differential pressure valve    Low pressure : 20-40mm H2O Medium pressure : 40-70mm High pressure : 80-100mm Four designs:      Slit valve : Ball in cone : Diaphragm: Miter valve: Holter Hakim Pudenz Mishler Variable resistance constant flow valves  F = DP/R ( supine position ) F:  DP:  R:  csf flow differential pressure valve resistence  F = DP +H/R ( Upright position )  H: height Shunt complications  Obstruction Proximal end  Distal end  Disconnection      Wound complications Infections – 7-10% incidence Seizures -5-48% Extracerebral fluid collection       Subdural hematoma Spontaneous pneumocephalus Overdrainage syndrome Ascites Bowel perforation Slit ventricle syndrome  Vent –pleural : Pneumocephalus  Pleural effusion   Ventriculo atrial : Infection  Shunt nephritis  Endocarditis  Superior vena cava syndrome   Lumbo peritoneal Radicular pain  Scoliosis  Chiari type I  ETV  Third ventriculostomy is intended to treat non communicating hydrocephalus with patent subarachnoid spaces and adequate CSF absorption. Success rates  High success rates : (75%) Acquired aqueductal stenosis  Tumour obstructing ventriculat outflow   Tectal  Pineal  Thalamic  Intraventricular  Intermediate success rates (5070%) Myelomeningocele (previously shunted, Older patients)  Congenital aqueductal stenosis  Cystic abnormalities  cyst  Dandy walker   Arachnoid Previously shunted patients with malfunction  Slit ventricle syndrome  Recurrent shunt infection or  Low success rates (<50%) Myelomeningocele( unshunted, neonates )  Posthemorrhagic hydrocephalus  Post infectious hydrocephalus  Indications  Clinical Causes of hydrocephalus in high or intermediate success group  Age > 6 months  No prior radiation therapy  No history of hemorrhage or meningitis  Patient previously shunted   Radiographic  Evidence of ventricular non communication  Obstructive pattern of HCP  Aqueductal anatomic obstruction  Lack of aqueductal flow void on T2 MRI  Favourable third ventricular anatomy  Width and foramen monro sufficient to accommodate endoscope • Rigid >7mm • Flexible>4mm • Thinned floor of third ventricle  Down ward bulge floor, draped over clivus  Basilar posterior to mamillary bodies  Absence of structural anomalies AVM or tumour obscuring third ventricular floor  Insufficient space between mamillary bodies and basilar and clivus  Basilar artery ectasia  Outcome    Morbidity- 3-12% Success rates – 60-70% Mortality – negligible Complications      Hypothalamic injury Fever- irritation of ependyma from blood Cranial nerve paresis – III and VI Injury to fornix, caudate nucleus Basilar artery THANKS

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