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Biochemistry of Neurotransmitters

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Biochemistry of

Neurotransmitters



Dr. Abdullah A. Osman

Objectives

 To define the chemical synapses

 To discuss the major classes of molecules that function

as neurotransmitters at chemical synapses

 To discuss the structure, origin, function of

neurotransmitters and their receptors

 To discuss the effects of neurotransmitters on

postsynaptic cells

 To define synaptic transmission

 To know the principal action of cholinergic agonists and

antagonists

Neurons and Central nervous System



 The nervous system regulates all aspects of bodily

function

 The human brain-control center that stores, computates

and transmits information

 The human brain contains~ 1012 neurons (nerve cells)-

each forming as many as thousands connections with

other neurons

 Millions of neurons regulates contraction of muscle and

secretion of hormones

 Function of neurons-to communicate information by two

methods:

“Continued”

 A) Electrical signals: process and conduct information

within a cell

 B) Chemical signals: transmit information between cells

 Type of neurons:

 A) Motor neurons-carry information and innervate

muscle cell via axon terminals

 B) Sensory neurons-have specialized receptors that

convert stimuli into electrical impulse which passes down

to the cell body, and then to spinal cord or brain

Synapses

 Are specialized sites where neurons communicate with

other cells

 They transmit signals in only one direction (from axon

terminal to postsynaptic cell)

 Two types:

1) Electrical synapses-rare and fast

2) Chemical synapses-common, slow and release

neurotransmitters

 The neuromuscular junctions-are junction between the

axon terminal of a neuron and the receiving cell (muscle

cells) and usually called synapse

Typical Mammalian Neuron Structure

 Neurons are specialized to produce and utilize different

neurotransmitters-chemicals that act in a paracrine fashion

Membrane Potential

 Action potential: neurons and muscle cells generate

electrical signals that are conducted rapidly over long

distances

 Resting membrane potential: action potential produced when

there is no active ionic conduction-an equilibrium state exists

 Membrane depolarization: a reduction in membrane potential

due to flow of cations into cells

 Membrane hyperpolarization: an increase in membrane

potetial due to influx of anions from outside the cells

 Exictatory synapse: represents membrane depolarization

 Inhibitory synapse: represents membrane hyperpolarization

Neurotransmitters

Transmitter Derived From Site of Synthesis

Molecule

Acetylcholine Choline CNS,

Parasympathetic

nerves

Serotonin Tryptophan CNS

5-hydroxytryptamine

GABA (gamma- Glutamate CNS

amino butyric acid)

Glutamate CNS

Aspartate CNS

Glycine Spinal cord

Histamine Histidine Hypothalamus

Epinephrine Tyrosine Adrenal medulla,

(adrenalin) some CNS cells

Norepinephrine Tyrosine CNS, sympathetic

(noradrenalin) nerves

Dopamine Tyrosine CNS

Adenosine ATP CNS, Peripheral

nerves

Acetylcholine (Ach)

 Used within the peripheral nervous system

 Synthesized from choline and acetyl CoA in the

axon terminals

 Neurons that secrete Ach and receptors that

bind Ach are described as cholinergic

 Ach is excitatory at neuromuscular junctions

(NMJs), where it acts directly to open ligand-

gated cation channels

 Ach is inhibitory in the autonomic

parasympathetic division, e.g. vagus nerve to

inhibit heart rate

Acetylcholine Biosynthesis

Acetylcholine-Chemical Synapse

Transmission

Acetylcholine (Ach) “continued”

 Ach binds to two types of cholinergic receptors: are

ligand-gated cation channels (Na+ and K+)

1) cholinergic nicotinic receptors:

a) found at the NMJs of skeletal muscle cells (only)

b) found on the post-gnglionic neurons of

parasympathetic nervous system and many

neurons in the brain

c) nicotine is an agonist ( hence the name)

d) curare is antagonist (its ability to paralyze skeletal

muscles)

e) when cations rush in-depolarization of synaptic cell

f) fast, short-acting synaptic potentials

Nicotinic and Muscarinic Acetylcholine

Receptors

Acetylcholine, “Continued”



2) Cholinergic muscarinic receptors (one of five types):

a) found at the NMJs of cardiac and smooth muscle

cells and glands

b) found on the post-gnglionic neurons of sympathetic

nervous system

c) muscarine (a toxin produced by certain mushrooms)

is an agonist ( hence the name)

d) atropine (sarin) is antagonist (its use in

acetylcholinesterase poisoning)-biological weapon

that

inhibits AChE

e) slow, long-acting synaptic potentials

Acetylcholine Receptor Signaling

“Continued”

Adrenergic Receptor Signaling

“Continued”

“Continued”

Ach-Receptor Agonists and Antagonists

Amine Neurotransmitters--derived from

a single amino acid

Catecholamines (CAMs):

 Derived from tyrosine-examples are:

Dopamine (DA), Norepinephrine (NE), Epinephrine (E)

 Cells that secrete NE are termed adrenergic neurons

 Bind to adrenergic receptors which are either alpha (α)

or beta (β) type

 Linked to G-proteins and initiate second messenger

cascade

 NE in the CNS is related to arousal, dreaming and

regulating mood

 DA in CNS neurons is related to emotion, skeletal

muscle tone and movement

Ctecholamine Biosynthetic Pathway

Release of Epinephrine at a Synaptic

Neuron

5-hydroxytryptamine

Turning Synapses Off

 Once its job is done, the neurotransmitter must be

removed from synaptic cleft to prepare the synapse for

the arrival of the next action potential:

 A) reuptake by synaptic knob of the presynaptic neuron

by active transport-all neurotransmitters use this method

except acetylcholine

 B) Ach is rapidly inactivated by acetylcholinesterase

(AChE) enzyme which splits Ach into acetate and to

choline. Choline is actively taken up by presynaptic

neurons and used to make more Ach

 C) Catecholamines (NE)-inactivated by monoamine

oxidase (MAO) action after active reuptake in the

presynaptic axon terminal

 * MAO inhibitors are used in the treatment of depression

and anxiety



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