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9. Aggressive lymphoma







557 558



PROGNOSTIC FACTORS FOB ADVANCED STAGE. INTERMEDIATE

CLINICO-PATHOLOGIC FEATURES OF K M POSITIVE, ANAPLASTIC LARGE AND HIGH GRADE NON-HODGKIN LYMPIIOMAS.

CELL LYMPHOMA IN EGYPT.

DA Gribabh, ThP VraOakopoulo*, VA Boimtotlj. F Komopldou, Ch Kltlas. GA

F-Abot-Ba. H.Khated, H.Taha, M.N.B-Bolkainy, R.Gaafar, MJUidei-Mooti, B.Bedair ; Pangafa. Hm Dipartment of Internal M«Bdn*, National and KapodJariaii

Pathology and Medical Oncology Depti., National Cancer Institute, Cairo, Egypt Unhvntfy o( Athens, Lalkoa Gsxral Hospital, Alhem, Greece.

K H positive, Anaptestic Large Cell Lymphoma (ALCL), has distinct cflnlco-parthotoglc Advanced a m (IIVTV) autnntdlati (K3> and high qrada (HG) non-Hodgklii

features which can resemble many other conditions. In this study, retrospective tymphomat (NHL) arc not curable wUh the convmUonaJ chemotherapy regimens.

analysis of 2 groups of patients was done. The first group included children and young In about the half of the patients (pts). Several rlafflflmtlnn s^wtems based on

adults presenting wfth lymphadenopathy and primarily diagnosed as metastaUc prognostic factors (PF) have been developed In order to distinguish subgroups of

patients utih poor prognosis, who are probably cfigfUe to participate In aggressive

undifferentiated carcinoma, while the second group consisted of patients diagnosed as chemotherapy protocols, followed by peripheral blood stem cell rescue or bone

high great lymphoma. Cases were selected on basis of peculiar involvement known marrow transplantation. In the present study we analyzed 174 pts wtth stage III or

for ALCL K M positive lymphoma. Twenty-six cases in each group were studied by IV, )G or HGNHL (etcept of lympboblastlc and dlfiuse small non-ckaved cell

NHL), aiming to identify prognostic factors (PF) for achievement of complete

immunophenotypic markers Including LCA, UCHL1, Leu 4, EMA, Cytokarattn and Ber remission (CR) and survtvaL The parameters studied were age ( s 6 0 vs >60

H2. Among the first group 8 cases showed the profile of ALCL K M positive years), gender, performance status (PS: 0-1 vs 2-4), stage, B-eymptoms, botic

lymphoma. Their ages ranged between 13 and 40 years, with a median of 29 years, 5 marrow Involvement (BMI), number of Involved cxtranodal sites (O-l vs i2>,

hWologlc subtype, serum LDH levels (nomud vs Increased), ESR (4JO, 21-50,

males, 6 presented with localized and 2 with generalized lymphadenopathy. >50) and serum albumin levels (2

group received regular treatment protocols and 4 received local radiotherapy to sites cxtranodal sites, m inulilvariale analysis. Moreover, In ntulltvariaia analysis of PF

for OS, only age >60 years had Independent prognostic significance. Whh respect

of disease. Complete remission was achieved in 8 out of the 15 K M postttve cases, to the modmed IPI, no differences were observed for CR rales. Patients wllh only 1

with a median overal survival of 16 months. Durable second remissions were also adverse PF had better DFS than those with 3-4 |p=0.02) and better OS than both

achieved In some of the relapsed patients. So, In conclusion, apart from the role of W- those with 2 (p-0.08 borderline) and 3-4 (p-0 03) adverse PF. Our results are

generally consistent with recent targe mulllcenler studies and confirm the abfllty to

1 antigen in differentiating this type of tymphoma from morphologically similar dlsartmljuue pts with advanced IG or 1-tG NHL with poor prognosis, m order lo be

conditions as undifferentiated metastatic carcinoma, K M positive NHL should receive treated more aggressively.

more intensive chemotherapy protocols to improve their CR rates and to benefit from

their tendency to attain durable second CR.









559 560



IS THE DOSE INTENSITY AN INDEPENDENT ANALYSIS OF PROGNOSTIC VALUE OF P53 AND PCNA EXPRESSION A D N

TRANSLOCATION t ( 1 4 ; 1 8 ) IN HIGH-GRADE NON-HODGKIN'S L M IO A Y PI M

PROGNOSTIC FACTOR FOR SURVIVAL IN HIGH-GRADE 1 2 1

A. Fossa ", K.A. Metz *, R. S i e b e r t * , C.P. W l l l e r s \ U. Noser 2 ,

MALIGNANT NON-HODGKIN LYMPHOMAS? B. Opalka 1 , S. Seeber1, M.E. Nowrouslan 1 ('contributed

equally).

0 . Sezer, K. Taghizadeh and H. Rasche. 1 2

Departments of Medical Oncology and Pathology , University of

Department of Internal Medicine I, Zentralkrankenhaus Essen, Essen, PRO

St.-JQrgen-StraBe, 28205 Bremen, Germany Molecular and cytogenetic findings have been reported to be

associated with clinical features and treatment response in

150 patients with high-grade malignant non-Hodgkin's non-Hodgkin's lyaphoaa (NHL). He studied p53 and PCNA

lymphoma were treated in a single centre with CABOPP, a expression by iuunohistochealatry and the translocation

modified COPBLAM protocol ( cyclophosphamide 500 mg/m2 t(14;18) by PCH in 36 patients (pts) with de novo high-grade

non-lymphoblastic NHL according to the Kiel classification. All

(iv) dl, doxorubicin 50 mg/m2 (iv) dl, bleomycin 3x1 mg (sc) pts were uniformly treated with an anthracycline-containing

dl-5, vincristine 1 mg/m2 (max. 2 mg, iv) dl+8, procarbazine •ultiagent cheaotherapeutic regimen.



100 mg/m2 (po) dl-10, prednisone 50 mg/m2 (po) dl-10 ). In The aedian age was 48 years (19-66). 5 pts had clinical stage

(CS) I, 17 CS II, 5 CS III and 9 CS IV disease. LDH > 240 U/l

patients older than 70 years the dose of cyclophosphamide and was present in 20 pts, B-syaptoas in 18 pts, and extranodal

doxorubicin were reduced to 300 mg/m1 and 30 mg/m2, Involvement in IS pts.

693 (22/36) of the pts achieved coaplete remission (CR) and 31X

respectively. The median age was 56 (range: 19 - 90). 72 % of (11/36) failed to respond or died during therapy. With a eedian

the patients achieved a complete remission with CABOPP. follow up of 9,5 aonths, the projected survival for the whole

group of pts is 58X at 34 months. 80X of the responding pts are

The chemotherapy associated mortality was 2 %. The overall predicted to readln free froa progression at 32 months.

survival rates were 65 % at 3 years and 45 % at 5 years. By p53 expression defined as positive staining in i. 40X of tuaour

multivariate analysis the major risk factors for the overall cells was found in 17/36 pts. In 24/36 pts i. 80X of tuaour

cells stained positive for PCNA. Molecular evidence for

survival were performance status, age and serum lactate t( 14; 18) was found in 22/34 cases. Multivariate analysis

dehydrogenase concentration. Neither the received total dose confirned CS as the sole prognostic factor for achleveuent of

CR and for survival. Other clinical parameters, expression of

of chemotherapeutic drugs nor the relative dose intensity p53 and PCNA and occurrence of t(14;18) did not appear to

calculated by incorporating the duration of the actual correlate with clinical outcome.

treatment courses were independent prognostic factors for the

survival in the multivariate analysis.







9. Aggressive lymphoma 151

561 562



PROGNOSTIC FACTORS IN PATUMTS WITH OBTUSE IAHGE CHI NON- PROGNOSTIC SIGNinCANCE OF IMMUNOPHENOTYPE IN

HOOGKNS LYMPHOMAS (NHL). INTERMIDIATE AND HIGH GRADE NON HODGKINTS LYMPHOMAS.

A HaiB*IC CO-OPttATTrt ONCCXOGY GKXJP STUDY

CMcoUd.1, CFourtrfcs. KLZouoboi. D.Sldfcii. P. Koiakii. K. Stefanoudaki-Soflanatou1*, P. Repouslsl*, Th. Papadaki 2 , M. Bakiri1,

AJCaraUS., TKOamaUdi, ASymmidl, LEtommou, KPovW.. M. Niklforakis1, D. Anagnostou 2 , A. Papayiannis 1 .

hWCOG OHlc, M.«>glon M. IB 27 Alt-ra, G , « c .

Haemntology 1 and Haematopathology 2 Departments,

Aim oiW NW«rkitr In Ihb retrotptcthre nufelurtric tfudy t U paiilJ* Evangelismos General Hospital, Athens - Creece.

prognostic voxiobfai for tumor r*tpora«, (ftMai* Inm curvtvd and ovaroQ ''Present address; HaematologyDptofSt.Anargyri Cancer Hospital, Athens-Greece.

•urvfvd w.r» anojywd in 215 conMcullvWr (rooted pollonts wth dtfh/u lorga

c*0 NH. (WF uttypn G and r". Sixty it* pollonU |3I%| praMrttd w*ti

primary •Jdranodol i t w m . Two hundred ihra* pdkrti (94.4%| w«r« Ireohd

Controversy exists regarding the prognostic significance of

wth adrknydn baud hi, 2nd and 3rd gmroikai rogknoni. M«Jon lolow up immunophenotype in intermldiate and high grade (I-HG) non Hodgkin's

w n 18 months. Lymphomas (NHL). To evaluate the significance of B JO mrVK oburah i3-So/ slgnllcont voriabbi. comparison between curves were based on the log-rank test. Statistical

(c]For dlMci* frw turvfvd both In th* untvarlot* anotysb ai w«l oi h analysis used the X2 test and Fisher's exact test for comparisons of groups.

awtlpU Cox analyiii PS23 and a>aU gmaW wrf. l U iwgoliv* proa^oiHc Cox's proportional hazards model was used to assess the prognostic value

ladori. of factors on survival and DPS in a multivariate framework. 68 cases (75%)

(d) In tK« unfvoriol« anatyill voriabUt prsdkling turvival w«r«- PS, slog*. were classified as Bronodol dU«OM, unlnvolvad bon* marrow, ipU«n invoryw—nt, tn« (ALCL). There was an even distribution of clinical characteristics

nnb*f ol .xlronodd tilai ol dluow, fi-rympleani, IDH, obumkv htnuuloHn between the two groups except for B symptoms which had a high









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and ESI In th. mMpU Cox oWy.ii PS 23 (p-O.OOOO) stag. Ill-TV (p-0O24a) incidence on T-cell lymphoma pts (p 07). The pts with T-cell lymphoma

and ESB >50 (p-00071) w«r« ubct«d ai prognodk lodo»i showed worse DFS comparing to those of B-cell (27% v 65%) at 5 yrs with a

{•) No staHdkal aW«nnc* wai o l u m d ilhif h conpUl* rctpou* rat« median of 23 m for the first group, median for the second group of pts not

|p-0-2509) nor In ovtral •urvt«al |pHDJ3V5) bdwmi Ir«atmn4 gmranon reached yet. Overall survival was 21% for T cell vs 42% for B cell

lymphoma at 5 yrs (p.064). Multivariate analysis revealed age, Karnofsky

status, LDH and initial response to treatment as the most important factors

affecting survival for both groups of pts. We conclude from our study that

more aggressive front line treatment should be considered as therapy for at

least some pts with T-cell NHL, I and HG malignancy.









563 564



DETECTION OF IgH AND IgL REARRANGEMENTS IN INTERMEDIATE Impact of Prognostic Factors and Long Term Results of BECOP

AND HIGH GRADE B-CELL NON HODGKIN'S LYMPHOMAS Regimen in Aggressive Non HodfWn Lymphoma.



Hussein M. Khaled ,N.G«d El-Mawla, M.R.Hamm, M.N.El-Bolkairry,

Nico van Belzen' lJ , Pauline E. Hupkes 11 , Marianne Hoogeveen 12 ,

R. Gaafar, N.Mokhtar ,1. El-Attmr, and I. Magrath • . * National Cancer

Lambert C.J. Dorssers1, and Mars B. van 't Veer'. 'Dept. Hematology,

Institute, Bethesda, MD ,USA , and National Cancer Institute, Cairo .Egypt

Dr. Daniel den Hoed Cancer Center, P.O.Box 5 2 0 1 , 3008 AE

Rotterdam; and 2 Dept. Hematology, Erasmus University, Rotterdam, Malignant Lymphomasrankthird (- 10 %) among patients presenting

The Netherlands. to the NCI of Egypt, with a higherfrequencyof aggressive subtypes and

advanced disease stages at presentation.

Rearranged IgH genes provide unique clonal markers for B cells, and In this study, a modified BECOP regimen were given every 3 weeks

allow sensitive detection of low levels of neoplastic B cells by PCR. rather than a 4 week period with a 25 % increase in dose intensity more than

However, cloning of the rearranged gene and proving that the cloned the standard BECOP combination , i.e. vincristine 1.4 mg/sq.m.,epinibicin

sequence is indeed derived from tumor cells appears to be more 40 mg/sq.m., cyclophosphamide 650 mg/sq.m., all IV days I & 8 .bleomycin

lOu/sq.m. IV day 15 , and prednisone 60 mg/sq.m. POdays 1 5 - 2 1 .

problematic in non Hodgkin's lymphoma (NHL) than in other B cell

One hundred and eight adult NHL patients were included in the study .

malignancies. We have therefore used a comprehensive PCR primer set

Seventy six were males .and 32 were females, with a M/F ratio of 2.4/1.

and stringent selection criteria to identify tumor-specific rearranged IgH

Their ages ranged between 16 and 70 yean (median 42 years ). Eight)'

genes. Lymphoma DNA was subjected to PCR using consensus JH 1

two(76%) patients had grade II and 26 (24%) had grade 1 1 pathologic

primers and either consensus VH or VH-femily specific primers. The subtype. Eleven (10%) patients presented with stage 1, 19 (18%) with stage

cloned IgH rearrangements are considered to be of tumor origin if the II. 64 (59%) with stage 111, and 14 (13%) with stage IV disease. The number

PCR products are predominantly monoclonal (as determined by direct of courses given ranged between I and 9 (median 6 ). Complete and partial

sequencing or denaturing gradient gel electrophoresis, DGGE), and if remissions were achieved in 79 (78%) and 10 (10%) out of 101 evaluabie

the same sequence is amplified with two independent VH primers. Of patients. Acturial 3-year overall and disease-free turvival rates were 55 %

13 intermediate and high grade malignant NHL, 12 yielded a PCR and 50 % respectively. Toxicities were mostly of grade 1 and II, with 5 %

product with two independent primers; 11 of these are considered mortality of treatment complications.

tumor-derived. The rearranged sequences showed 83-99% homology Response rates and survival data of this group of patients were analysed in

with their VH germline counterparts. In some cases clonal sequence relation to age, LDH level, rumor burden, and disease stage at presentation,

heterogeneity was observed. as well ts to the international index and the received dose intensity .

While this more dose intesive BECOP regimen has achieved the best

Using a patient-specific primer chosen in the CDR3 region of the results reported for treatment of adult NHL patients in Egypt, an ongoing

rearranged IgH gene, minimal residual disease was detected in study is evaluating the role of continuous infusion chemotherapy ( EPOCH

peripheral blood samples of a lymphoma patient. regimen) as front line therapy in previously untreated NHL patients.

Since accuracy is of utmost importance in diagnosis, rearranged IgL

genes are being cloned from lymphoma DNA to use as a second

marker for minimal residual disease.









152 9. Aggressive lymphoma

USE OF THE INTERNATIONAL PROGNOSTIC INDEX CENTRAL NERVOUS SYSTEM (CNS) RELAPSE IN NHL: INCIDENCE, RISK

(IPI) AND THE TUMOR SCORE (TS) TO DETECT POOR- FACTORS AND VALUE OF PROPHYLAXIS.

RISK PATIENTS WITH PRIMARY MEDIASTINAL JC Kluin-Nekmans, E Bollen 1 , RE Brouwer, S Hamers1, PM Kluin1, J.Hermans'. Dcpu of

LARGE B-CELL LYMPHOMA (PMLCL): A STUDY WITH Hcmuology, 'Neurology. 'Pathology and 'Medical Statistics, Leiden University Hospital.

37 PREVIOUSLY UNTREATED PATIENTS. J.E. Romaguera, Leiden, The Netherlands.

J. Rodriguez Diaz-Pavon, LCarias, F.B. Hagcmeister, P. McLaughlin, ID a retrospective analysis, the incidence, risk factors and potential role of prophylaxis

M.A. Rodriguez, A.H. Sarris, A. Younes, A. Preti, Edio LJerena, and of secundary CNS relapse were studied in 553 patients (307d ;2469. median age 59.2 yr) with

Fernando Cabanillas. The University of Texas M. D. Anderson Cancer NHL seen in our institution between 1983 and 1990.

Center, Department of Hematology, Lymphoma Section, Houston, Tx, Methods: Patients with AIDS. CLL and ALL were excluded. Diagnosis of CNS involvement

U.S.A. The best therapeutic approach and prognosis for patients with was made on spinal fluid analysis, mydofraphy, CT or MRI scans, brain biopsies, or a

PMLCL is undetermined. New prognostic models devised for combination of rhese. Risk factors assessed were: age. sex, malignancy grade (according to

intermediate grade h/mphomas might not apply to PMLCL in view of its the Working Formulation), stage, localizatioa, response to initial therapy. CNS prophylaxis

different biological features [e.g. their B2 mkroglobulin (b2M) is consisted of MTX i.L, high dose ARA-C consisting regimens, or both. CNS relapses were

consistently lower than other types]. Patients and Methods: We tested treated by MTX i.t. and whole brain radiotherapy with deiamcthasone in most ewes.

the age-adjusted IPI (aalPI) and a modified TS (MTS) in which the Results: Out of 553 patients, 21 bad primary cerebral NHL and were excluded. Eleven patients

prognostic threshold value of b2M was lowered to 1.6 mg/dl which was presented at start with both CNS and systemic NHL. With a median follow-up of survivors

the median value of our studied population. Thirty-seven consecutive of 30 months, another 55 patients developed CNS relapse, making up for a total cumulative

untreated patients diagnosed with PMLCL and treated from January risk of 19% at 4 year. Of these 55, 9 piescutcd initially with low grade (4 of whom had

1985 to January 1994 at our institution were included in the analysis. progressed to a higher grade), 33 wirh intermediate grade, 9 with high grade and for 4 cases

Results: Thirty-two percent of the patients fell in the low-risk category no WF grading was available. CNS relapses were localized lcptomeningeal ( n - 2 6 ) ,

according to the aalPI and 43% of the patients presented with a low parenchyma] ( n - 5 ) or both (n—21). Median tune between initial NHL diagnosis and CNS

score according to the MTS. Only the MTS predicted for achievement of relapse was 9 months (1-97), with all high grade cases having their relapse within the first 24

complete response (93% versus 60% for low and high scores, months; in the others relapses were seen up to 6-8 years after diagnosis. Risk factors for CNS

respectively; p = 0.02). Both models defined a group of patients at high relapse were malignancy grade, age e was

for refusal). Results: The complete response (CR) rate was 98% (49/50); 1 (2%) reported as definitely T cell in 11, possibly T cell in a farther 4, I) cell In 2 and w n

patient showed a progressive disease to CNS during CHOP chemotherapy and died indeterminate in Ibc remainder.

for lymphoma. Six patients (12%) in partial remission after CHOP achieved CR

following involved Odd radiotherapy. After a median follow-up of 39 months, 4/49 Seven children did not achieve a complete rcmistioo on this regimen, five of whom have

patients have relapsed, three of these patients relapsed in the she of previous died (two responded when changed to a Hodgkin'i type regimen). Of the remaining 27

radiation therapy. The actuarial 4 years overall survrtal and event free survival was children who did respond In initial treatment 19 remain in complete remission with a

respectively 88% and 81%. The treatment was well totalled without significant median followup of Iwcoly four months (range 3 - 5 2 moaths). Seven children have









Downloaded from http://annonc.oxfordjournals.org/ by guest on January 2, 2012

usddty. Conclusions :Short course of CHOP chemotherapy followed by involved rdapscd of whom six have died. One child died of secondary leukaemia.

field radiation therapy is a safe and effective treatment for patients with aggressive

NHL Stage ME. Although the role of adiuvant radiotherapy in this setting of There Is no consensus on (be optima] treatment for children with large cell anaplaslic

patients is fMMilfd. at the present time this approach maybe considered the standard histology. Wilb this regimen Ibe event free survival was jutt les> than 6 0 1 ' in

therapyforthese pnHf^t cotnparisoa lo that seen for children with malnrc 1) cell lympumna which ii KHT It

remains to be seen whether a hybrid treatment utilising a combination of H cell NIIL and

Ilodgkin'slypc treatment with procarbazine and cblorambocfl will produce better results.

DSV is supported by a fellowship from AIRC









571 572



WHICH TREATMENT IS PREFERABLE FOR ANAPLASTIC LARGE-CELL PRIMARY TREATMENT OF HIGH GRADE NIIL : FINAL

LYMPHOMA-HOOGKIN'S-UKE (ALCL-HL): MACOP-B OR ABVD? ANALYSIS OF A GERMAN MULTICENTER TRIAL

U. Kaiser, H.Koppler, I.Uebelacker, K-Havemann for the German

P.L Zinzani, for the Kalian Cooperative Study Group on Lymphoma. High Grade NHL study group. Zentrum Innere Medizln, Philipps-

Institute of Hematotogy "Seragnotf, University of Botogna, Italy Unlversltat Marburg, 3S033 Marburg, Germany



A randomized multlcenter trial comparing 4 cycles of CIIOEF with

From September 1988 to October 1993, 90 anaplastic large cell lymphoma

4 cycles of two alternating regimens, hCHOP and IVEP followed by

(ALCL) patient* were treated with third-generation chemotherapy regimens

Involved field (IF) radiation with a dose of 35 Gy for both

(F-MACHOP or MACOP-B) during the course of an Italian multicentric

treatment arms was conducted between 1986 and 1990. Included

randomized trial on high-grade non-Hodgkin's lymphomai (HG-NHL). In

were 175 patients with primary high grade (hg) NHL, stages II-

particular, 47 patients had ALCL of the common type (CT) and 43 had

IV. Treatment results after two years have been published (Ann

HodgHn's-like (HL) subtype. Complete remission was achieved in 66/90 Oncol, 1994) and have shown no benefit for the alternating

(73.5%) patients [33/47 (70%) with ALCL-CT and 33/43 (77%) with ALCL- regimen. We performed a final analysis after 5 years. Overall

HL]. The probability of relapse-free survival (RFS), projected at 63 months, survival was not significantly different in both treatment arms :

was 67% for ALCL-CT and 82% for ALCL-HL The data of the present study 58% for both groups combined. In a multivariate analysis pre

confirm that ALCL responds to third-generation chemotherapy regimens treatment LDH was the most valuable prognostic marker followed

slmaarty to other aggressive malignant lymphomas in terms of both CR and by stage. Among the 47 patients who relapsed 42 patients could be

RFS rates. further analyzed: median interval to relapse was 12.5 months (6-

As there is no proof from the literature that ALCL-HL cases actually 63 months). 4596 ( n - 1 9 ) of the patients relapsed exclusively

correspond to HodgkJn's disease (HD) with an unfavourable clinical course, outside of the prior radiation field, 29% (n-12) relapsed Inside and

outside, 19% (n-8) relapsed exclusively Inside the radiation field

and since variances still exist as to the optimal treatment of ALCL-HL we

and 7% (n-3) relapsed with an acute leukemia. Data underline the

have planned an ongoing randomized prospective muWcentric trial for

value of IF radiation therapy. In an ongoing study five cycles of

ALCL-HL, witri the aim of assessing the effectiveness of standard protocols

CHOEP are compared to three cycles of CHOEP followed by high

for HD (ABVD scheme) versus third-generauon chemotherapy regimens

dose chemotherapy and autologous stemm cell support for

(MACOP-B) as complete response (CR) and RFS rates. Since September

patients with high risk hg NHL In both arms IF radiatio is

1994 we have so far randomized 30 ALCL-HL (according to REAL

performed after chemotherapy. Currently 200 patients have been

classification) patients of whom 25 (10 for MACOP-B and 15 for ABVD) are

included demonstrating the feasibility of the study.

currently evaluate for the response rate. The two groups are fuBy

comparable In their main cfinteal features, without any significant difference.

So far, the CR rates have been 80% and 73% in MACOP-B- and ABVD-

treated patients, respectively.









154 9. Aggressive lymphoma

574



CHEMOTHERAPY FOR YOUNG PATIENTS BELOW 60 YEARS TREATMENT OF AGGRESSIVE NON-HODGKIN'S LYMPHOMA WITH A

WITH INTERMEDIATE GRADE (IG) NON-HODGKIN'S PROMACECytaBOM-UKE REGIMEN : IMPACT OP THE DELIVERED

RELATIVE DOSE INTENSITY FOR MITOXANTRONE AND

LYMPHOMA (NHL) USING FOUR DIFFERENT DOXORUBIC1N- CYCLOPHOSPHAM1DE ON THE SURVIVAL.

CONTAINING REGIMENS.

W.Fercmanj, P.Neve, L Marcdis, A Dewowere, M Morret-Rauis, JP Kadtu, H Boodue.

RXiang, D.Todd, Y.L.Kwong, C.S.Chim, C.K.Lcc, F.C.S.Ho*. D Brenez, D Brohee, JL Dtrgent, A Verhest d i m e of Haematology E n t i r e University

Hospital Brunei*; Belgian Lymphoma Group, Belgium.

Departments of Medicine and Pathology*, University of Hong Kong,

Queen Mary Hospital, Hong Kong. MitoxaiUioiie has been incorporated '

nan-Hodg)dn'( rymphoma (NHL) with a comparable efficacy tnd sometime* a tower

Doxonibicin-containing chemotberapeutic regimen, e.g. CHOP, is the todcity. Five j e a n ago, wt started a phase II study with a PROMNCECytabom regimen

corner-stone for treating IG NHL. This study compared the efficacy of 4 in aggrestive NHL. Miuxcantron* was given at 5 mg/m ] instead of 25 mg/m 2 of

doxorubidn; the othen drugs w e n ° - h —V"* according to the SWOG original regimen.

different regimens in a group of young patients below 60 years with IG

PATIENTS: 30 patients were eligible (M-17.F-13X mean age 54.2 y (17-69);

NHL. 405 patients below the age of 60 with IG NHL received four increased LDH and p2 mkroglobulin respectively in 50% and. 23%; 1WF D - 3 . P - 4 .

different doxorubicin containing regimens: 222 (55%) had CHOP, 87 G-14, H - 2 , > 3 , K l - I - 2 , other-2;atrioodal sites present in 73%; International Index

(21%) had BACOP, 72 (18%) had m-BACOD and 24 (6%) had Low (n—1IX Low-Interm. ( n - 9 ) , High-Interm. (n—5). High (n-5).

ProMACE-CytaBOM. There were 44% females and 56% males. The RESULTS: The response rate after 6 cycles was: CR-17 (57%), PR-7 (23%), primary

median age was 48 years. 71% had Stage I disease, 27% Stage II, 18% refractory 4 (13%) and early toxic deaths 2 (7%). The maximal tcoriciry grades (mean

of the highest WHO grade for each patient) were £ 1 except for Hb (1.4) and PMN (2 6)

Stage II and 48% Stage IV. 32% had B symptoms, 18% bulky disease and for the 167 cycles analysed. The DFS (CR patients) reached 66 % at 5 y (mean f-up- 31

48% a extranodal primary. For those who were immunophenotyped, 23% months). For the entire cohort, progression-free survival and overall survival (mean f-

were T-cell and 77% B-cell. Additional local radiotherapy was given to up: 28 months) were respectively o f 5 6 % a a d 55 % m 4 y. These resulu are dowry

43% of the patients following chemotherapy. For patients with Stage I/I! comparable to those of the others previously reported multictturic triali using

disease, 86% had a complete remission (CR) and the 5 year disease free PROMACECytaBOM or PROMACECytaBOM-like regimen*.

DOSE INTENSITY: Patients who had received the 6 cycles and more than SO % of

survival (DFS) was 76% for CR patients. For those with advanced Stage delivered RDI for N and CPM (full-dose cohort) were compared to the others (low-dose

III/IV disease, 57% had CR and the 5 year DFS for CR patients was 37%.









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cohort) in terms of PFS and OVS. The difference was highly significant in favor of the

There was no significant difference in CR rate, DFS and overall survival full-dew cohort m—«im PFS unreached v» 19.5 months (togrank p - 0.02), median OVS

for the four regimens. Significant independent variables affecting unreached vs 26 months (logrank p - 0.04). The 3y OVS for full-dote vs low-dose was

prognosis on multivariate analysis included clinical stage, B symptom and respectively 74 % vs 42 % (mean f-trp 33 months vs 30 months).

CONCLUSIONS: 1° PROMNCECytaBOM is an effective regimen with a moderate

serum lactate dehydrogenase level. We concluded that the newer

generations of doxorubicin containing regimens did not significantly toxiaty.

2° The use of colony-stimiilating factors is ttwtin to sustain the

improve the clinical outcome of these patients. delivered RDI in selected patients.









576



LONG-TERM FOLLOW-UP STUDY OF A COMPARISON OF CVU5RIAMYQN-0P (CHOP) C H O E P C H E M O T H E R A P Y IN I N T E R M E D I A T E A N D H I G H G R A D E

VS. C-EP1RUBICJN-0P (CEOPL WITH PHASE "B" BLEOMYDN. IN INTERMEDIATE AND NON-HODGKIN LYMPHOMAS

HIEH GRADE NON-HODGKIN'S LYMPHOMAS (NHL).

AOnOrk, E.O.K*ndemir, M.Yaylaci, N.Oskem, Department of Hematology-Oncology,

J. Otai Maquao*. E. Go/da VaW. f. Romafo Qards'. A AtMtf. U.J. Nambo*. JA. CMgado Lama*' OATA Haydarpaja lstanbul-TURKEY

^tdaOncotoglaCMNSiglo>0«.*\«S& Ma»fco Of ond 1>« C*»nto». MSS Guadatajatn. Mtofcn

Thirty-nine intermediate and high grade Non-Hodgkin rymphoma patients were treated

Recant obtatvebon* on rtaiiwt mufti of NHL he*« encourogadrenawadiraareM on O O P and with CHOEP prothocol (Cydophosphamide, 750 mg/m2 1st day, Adriamyrin, 50

CHOP-ik»reglmatAmong CHOP vena**, tw *ub»*Uton o) down*tc«i by 4-apiiubran (CHOP) may mg/m2ls day, Vlncristine, 2 mg 1st day, Etoposide, 100 mg/m2 on days 3-5 and

ha\»*padeiiniponAncac^ttl^tactlTeldo)stngot2Vrn(toxraxatalntaclnnaalwaalaVWan«l>.«MLauciMXinntsoja From July 11M7

year diwiTr free survival was 50 per cent and total survival was 60 per cent

ID Oacarabar 15.1991 3> paMnti ware indudad n a randomiiad way in asoh pnHocol oim. Mnoi

Grade II—III granulocytopenia, thrombocytopenia and grade IV alopecia were the most

\Mnnc*% raoanftig ega. MX liunjiugy and piijgnoJlc loctora ware obHfvad in botfi group*

encountered side effects due to the chemotherapy.

rendaring taut My compa/aUa. Ra«pon*a. madum disaaaa Iraa irtarvol (mOR) and madwn o^«fal

*orvk«l (mOS) ware anolytad accortjaig w(h t» Wamataial Propotaltotproflnottc danitcekon-In

penanttwUihigh 1/2 and high RtK. CHOP provad to b* more atcaoou* wtti a CR ol (3 CV « •

inondi*ofniOF)lotlwtawhoo6tair^CR44*monb*o(mOSenlSu^oldaahpa«ant*.vt oCRol

U.4X.13mona>*of mDH. 11 montw olmOS end SI SX oldaafc* In »ia CHOP group ButinpeMna

wtn low oi low 1/2 ri*k tare ware no tjgnacsnt dftaianca* batwaan Da t arm* « * 17.4/82 JH O\

65./SS- mOH 74>/72« mOS and 2V29.4X of daaha.totCHOP/CEOPretpactvalyThar, ware no

ikjn^coM uHltiaiir**regnrrfngfonciatogic natay b 10

cm) and i III intermediate or high grade (WF) NHL were included in the GOELAMS yet clarified. We refer the results of a cooperative randomized trial between two 12 weeks

07 protocol. This consisted of 4 courses of chemotherapy (ABAB or AAB8 by rr^imfn* ( VACOPB and V1COPB) different only for antracydine drug used

initial randomization). Course A • Epirubicin 100 mg/m2 IV d2, Prednisone 80 (Doxorubicine vs Idarubidne). Tbe purpose of tbe study was to test tbe efficacy and toxichy

mg/m2 (V d1 to d5, Cytoxan 1,2 p/m2 (V d2 and Ekfisine 3mg/m2 IV d2 and d5. of Idarubicine in DLCL.

Course B - Hosfamtde 1,5 g/m2 IV d1 to d3, Mitoxanlrone 10 mg/m2 IV d1-2, Patients and Methods Since June 1991 to December 1994 103 pts with DLCL in

Etoposide 100 mg/m2 IV d1 to d3, and Methotrexate 100 mg/m2 d1. Courses advanced stage, with age l, 31 pts B symptoms, 58 pu LDH level above normal ; 37

prognosis significance on SV and EFS of sex, age (>2) tcoddties occurred in 22 pur 2 cutaneous, 4 mucositis, 3 peripheral neuropathy, 3

hepatotoxicity, 3 infections, 1 diabetis, 2 pulmonary embolism , 1 paeumothorax and I

In univariate analysis (UVA) in the ITT pts the only factors influencing SV were LDH deep venous thrombosis. Mucosal toxidty was significantly lower in pu treated with









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level (>2N), PS (>1), BM involvement present at diagnosis and ASCT. Stage IV VICOPB (p-0.02). CR was obtained in 79% of pts treated with VACOPB and 56%

without BM involvement at diagnosis was not of adverse prognosis significance.

EFS was affected by the same factors. In a muttivariate analysis (Cox) if ASCT is (Idarubidne 8 mg/sqm ) and 75% (Idarubicine 10 mg/sqm) with VICOP ( p-n.i);

entered in the system only LDH level remain significant for survival and EFS. In prognostic factors that negatively affected CR were: stage IV and BM infiltration for both

ASCT, patients LDH and stage influenced SV in UVA and only LDH level in Cox, schedule. With a median FU of two years tbe OS and DFS were not significantly different

LDH only in Cox influenced EFS. The single factor influencing ASCT feasability between the two treatmem arms: VACOPB ( 73% and 70%) vs VICOP-B ( 56% and 60%

was BM involvement at diagnosis. p-n-s.)

Conclusions are that ASCT may abrogate the adverse effect of BM Involvement, Conclusion: Idarubicine is less toxic then doxorubicine and 10 mg/sqm is easily tolerated ;

high LDH level (>2 N) is the most adverse prognosis factor even for ASCT at 10 mg/sqm its shows the same efficacy of doxorubicine in DLCL, but this prdinunar)

patients. observation must be confirmed by In a larger series of pts.









579 580



ALTERNATING CEOP/CNOP WITH INCREASED DOSAGE OF AfTtsstve Don Hodgldn Lymphoma (NUL)i Experience wttfa the CHOP

ANTRACYCLINES IN INTERMEDIATE-HIGH GRADE NHL regimen In a single Institution.



CMonnent, J.Bauer and SXeyvnz. Centre plundisciplinaire d'Oncotogie,

F. Di Raimondo, G. Milonc, G.A. Palumbo, M. Pafumi, R. Giustolisi

CHUV, 1011 Lausanne, Switzerland.

Institute of Hematology - University of Catania - ITALY

In a recent piN'ori"". Fischer (NEJM 1993) have shown that 3 years survivil

In a previous itudy we have demonstrated that in a CHOP-like scheme (yj) of patients with intennodiate or high grade NHL treated either by

the substitution of Adriamycin in rotation with Epirubicin and standard CHOP chemotherapy or by more intensive regimen is equivalent. In

Mitoxantrone (CEOP/CNOP) does result in a better tolerabilily of other reports, Shipp (JCO 1990) demonstrated that high dose Methotrcxale

therapy with the same response rate as CHOP. In an attempt to increase (HDM) wti not better than low dose Methotrextte in preventing central

the percentage of response in the alternating CEOP/CNOP scheme, we nervous system (CNS) relapses.

We have retrospectively analyzed 129 of 173 patients with aggressive NHL

escalated the dosage of Epirubicin from 50 to 100 mg/m2 and that of

(IWF: E-J) treated in our institution between Juanary 1982 and July 1995 with

Mhoxantrone from 10 to 16 mg/m2, while keeping the standard dosage a standard CHOP (G.I) or CHOP with HDM (G.U). Preliminary results are

of CTX (750 mg/m2), VCR (1.4 mg/m2), and PDN (60 mg/m2). summarized in thi» following table:

Twenty consecutive patients affected by NHL from E to H of WF, were £4 mtmAa SIWIII rv B-IHWHH rsa-i £B a™rw.cn

enrolled in this study. Median age w u 48 ys. Nine patients were at initial OI Jl Hi 41H IMH 17% 67.1% 51%(4I 69%)

stage (I. It) of disease, while eleven were at advanced stage (III, IV). Oil 71 47 49% 11% S7S 11.7% 64%(5I_77%)

1

Complete remission (CR) was significantly better for G 1 (pO.001); however

Hematological toxichy was negligible and no patient required growth

5 yJ was identical in both groups. Moreover, three of the four CNS relapses

factors in order to maintain the 3 wits schedule No cardiac torichy has occur in group G U.

been encountered so far, but four patients experienced grade 3 GI 5 y j and CR were then estimated by grouping patient according to the

toxichy. Fifteen out of twenty patients (75 V.) obtained CR, two patients tnii-nmim.l Pronostic Index (Id) (NEJM, 1993) - PS; LDH levd;ige; stage;

(10 %) PR and three (15 %) showed progression of disease. When we extranodal /titM«*., Only 85 patients met these criteria :

examined our patients according to the International Prognostic Index in a in in

we found that CR was achieved in all the patients with low risk and in 3 L 31 SO% 17% 74%

U 15 72% M% U%

out of 5 in the low-intermediate category, but in none of the patients m 13 61% 55% 4J%

affected by high risk NHL. H 1 IP1 is comparable to the original results.

seem useful in high risk patients and may induce more cxtra- However, due to small number of patients and large confidence mtervill, it

bematological toxichy. does not reach statistical significance.









156 9. Aggressive lymphoma

ITALIAN NON HODGKIN'S LYMPHOMA COOPERATIVE STUDY GROUP Lymphoma of Burkitt and Burldtt-like type is curable with intense short-

(NHLCSG): PRELIMINARY RESULTS OF RECENT TRIALS FOR term chemotherapy.

DIFFUSE AGGRESSIVE NHL A Johnson, J Ltndenxh & E Cavtllin-Stihl. DcjX of Oncology, Univ Hosp or Lund, Sweden.

T.Crrisesi*. G.Santinl . PXeoni, AJorceuini, P.Coter, L.Salvagno, RXenturioni, Burkitt and Burkltt-like B-ceil lymphoma (BBL) is a rare (1%) but important lymphoma

MJtSertoli, C.Guarnaccia, L.Tedesdu, MCongju, M Vespignani* and VJtazoli subgroup to be recognized in young adults wilh rapidly progressive disease. Gross abdominal

•Department of Hematology, SS.Oiovarmi e Paolo Hospital, Venice, Italy tumour with ovarian or gut involvement and ascites is common. A large tumour burden and

high proliferation rate enhance the risk of the tumour lysis syndrome.

In 1992 the NHLCSO published the remit! of conventional chemotherapy (CCT) and Since 1985 we have treated all BBL patients under 40 yean of age wilh a short alternating

ABMT for offtniive NHL. The condusioni we drew were: a) second and third 3-month schedule according to the German NHL -BFM protocol 1986 and 1990. In recent

generation regimeni do not improve DFS and PFS, b) the role of ABMT in 1 CR wa« years support with G-CSF has made it possible to treat patients 40-50 years old.

Patients. Between 1985 and 1995, twelve patients, (10 males, 2 females, age 17 -50 years,

uncertain and randomized itudiet were required.

median 22) were diagnosed with BBL. The major presenting she was abdominal with bulky

Therefore we Parted 8 new trial in which a tenet of consecutive patients was divided

tumour or peritoneal spread (7) and ascites (3). Other sites w e n lung/mediastinum ( I ) and

in 3 groups according to age, Mage and negative prognostic factors at diagnosis.

extradural with medullary cord compression (I). Bone marrow involvement was present in

Up to June 1995, 206 new patient* (groupi F-O-H-K-/WF), wilh an age 15-59 yean only two and concomitant CNS involvement in one. N o patient was leukaemic. One patient

entered 3 different triali. An interim analyst ii presented. was stage I (cervkal node), two stage II (tonsil, sinus) and 9 stage IV. Three patients were

STUDY A: The objective wai to evaluate the impact of conventional CT on patienu severely ill • one moribund with ascites and pleiiral effusion, one uremtc and one

with low tumor burden. 41 patienu in stage II and III without advene prognostic paimpamic. LD was > 3 x normal value in 5 patients.

facton were treated with VACOP-B regimen- Three-year actuarial curvet thow a OS, Treatment Increased diuresis, alkalinUation and prednisone 30mg/m 1 d 1-7,

DFS and PFS of 81%, 77% and 73% respectively. cyclophosphamkJe (Ox) 200mg/m I (with menu) d 2-6 and methotrexate 12mg h d I.

STUDY B: The endpoint wai to evaluate the effectivenets of high dose Alternating Block I Dexatnethasone(Dxm)10mg/m J dl-7, C o 2 0 0 ™ ^ ' d 2 - 6 ,

chemotherapy (HDT) in increasing OS and DFS. 124 patienu in stage II-IV with one methotrexate (Mtx) MXhng/m1 23h infusion d 2 (leucovorin 30mg x 2 4!h and 54h after

or more adverse prognostic factors were randomly allocated to receive either start), Mtx 12 mg it d 2, tentposide \6$mgjm'd 6, cytarablne 300 mg/m 1 d 6 . After one week

rest alternating block II Dxm, Ctx, M a , Mtx it as in block I, doxombicin SOmg/m1 inf 2 h d

VACOP-B or VACOP-B+ABMT. Actuarial survival (67% in both arms) and DFS

6. Block I and II were repeated three times followed by CNS irradiation 24 Gy (not stage I).

(56% vs. 65%) curves at 3 years are similar, showing no advantage in adding HDT.

The present protocol NHL-BFM 1990 b augmented by Inclusion of Ifosfamide instead of

STUDY C: 41 patients in stage IV with bone marrow involvement were treated wilh

Ctx In one block and treatment further differentiated according to risk group. CNS irradiation









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VACOP-B until maximnm response followed by high dose CY+d/QM-CSF. is omitted and it profylaxis given with triple drug combination (Mtx 12rag, Cytanbine 30tng,

Peripheral progenitor cell rescue was given after Mebphalan+TBI. An interim analysis Prednisone lOmg d I in each block).

shows three-year OS and DFS curves of 67% and 49% respectively. Results. All patients obtained complete remission and remained disease free during a median

These preliminary retuhi allow us to draw some conclusions: 1) in the favorable follow up of 48 months (3 • 120) and normal physiologic function was restored in all.

group of patienu, the impact of conventional CT is reasonably satisfactory. 2) In Discussion. Standard chemotherapy for high grade lymphoma has produced poor results in

group B the intensification with HDT and ABMT teems not worthwhile, wilh no BBL and high dose therapy with stem cell support has been advocated. It is our experience

advantage in OS and DFS. For this group of patients a new approach with a different that BBL is cured in young adults with the use of short term intensive chemotherapy as has

rationale is needed. 3) Study C indicates that HDT has a good impact in patienu wilh been described in childhood BBL. The rapid clinical, pathological and immunological

BM involvement, independently from other prognostic factors recognition of this entity is decisive for the outcome.









E3CALATED DOSE EP1RUBIC1N (epi) - CYCLOPHOSPHAMIDE (cyclo) WITH COMPARISON OF THE ABILITY OF GM-CSF AND O-CSF TO

FILGRASTtM FOR PATIENTS (ptt) WITH AGGRESSIVE NON-HOOGKIN'S AMELIORATE TOXICITY FROM AN INTENSIVE TREATMENT

LYMPHOMA (NHL). PROTOCOL FOR NON-HODOKIN'S LYMPHOMAS. Adde M, Venzon

D, Horak I, Shad A, Arndt C, Gootenberg J, Seibel N,

A. Grigg, M. Wolf, J. Levi, J. Bartlett, M. Green. Royal Melbourne Hospital, Neely J, Nieder M, Owen H and Magrath I. National

Peter M i c C a l u m Cancer Institute & Royal North Shore Hospital, AustrsBa. Cancer Institute, USA.



Based on a phase I dose-escalation study of ept-cyclo with fUgrastlm support

In pts wSh solid tumours, a mutU centre phase II study in NHL was Initiated In We have treated 64 high risk patients with either SNCC

1963 using the maximal dose found to be deliverable over multiple (46) or LC (18) lymphoma with a chemotherapy protocol

treatment cycles. Epi was used Instead of doxoruWdn as i t equlmolar closes consisting of two regimens, A and B. Regimen A

It is less cardlotoxtc and less myelotoxic, allowing dose-escalation. Pts included CTX, ADR, VCR, and high dose MTX, and regimen

received IV epi 150 malm', cyclo 1500 mg/m 1 , vtneristine 2 mg on day 1 B: IFOS, VP16, and high dose ara-C. Two alternating

and oral prednlsolone 100 mg for 5 days, together with daily flkjrasilm 5 cycles of each regimen were given and all patients

ug/xg SC from d1 until W B O 1 0 x 1 0 / 1 Cycles were given at 21 day received IT ara-C and MTX. Patients enrolled on the

intervals. Twenty-two pis have been treated, with • median age of 44.5 (20- study through December, 1993 were randomized to receive

70) years. The underlying disease has been de novo Intermediate grade or GM-CSF (23) or no CSF (20). In randomized patients,

tmmunoblastlc NHL In the majority (18 pts); of these 39% had B symptoms,

GM-CSF administration began on day 13 in A cycles and

44% h i d elevated LOH, and IB had either stage 2-4. extranodal or bulky

disease. The regimen has been reasonably tolerated with febrile neulropenli day 7 in B cycles. EFS for patients enrolled through

(FN) the major toxtcly. occurring In 13/22 (59%) of first cycles but only 12/91 December, 1993 was 92%. A comparative analysis did not

(13%) of cycles 2-4. Tfw neutrophfl nadir occurred tattler and was longer In show statistically significant differences in duration

cycle 1 vs later cycles. Only 9 of 91 (10%) cycles 2-6 have been dose of neutropenia or the incidence of fever or infection

modified due to prolonged neulropenli (n«2) or platelets 1 year post

patients according to the administration schedule for

treatment have recovered their fertility; one woman b pregnant. One m a l e

remains azoospermtc at 13 months. Seventeen of me de novo pts are GM-CSF. Toxicity data from 19 G-CSF treated patients

evaluibte for response (1 too early). All 14 who achieved CR i r e currently was compared with the GM-CSF and control groups. In

•Bve In remission, with a median foflow-up of 23 (4-33) mths from diagnosis. regimen A cycles, the duration of neutropenia was not

The other 3 de novo pts filled to achieve CR and died of disease. These different among the three groups. However, the

promising preliminary results have led to the Initiation of a randomised study duration of neutropenia was shortened in B cycles for

In Intermediate grade NHL comparing this regimen with 'conventlonaC dose GCSF treated patients (median, 5 days compared to 10

(epi 75 mg/m 2 , cyclo 750 mg/rrr) treatment. days for both GM-CSF and control groups) . Although the

duration of neutropenia was shortened by G-CSF,

platelet recovery prevented earlier initiation of

subsequent chemotherapy cycles and higher dose

intensity. Patients in B cycles, however benefited

from the shorter duration of neutropenia.









9. Aggressive lymphoma 157

585 586



INFLUENCE OF G-CSF ON THE NEUTROPIDL RECOVERY THE PATTERN OF FIRST RELAPSE IN PATIENTS WITH

HISTOLOGICALLY AGGRESSIVE NON-HODGKIN'S

IN HIGH GRADE NON-HODGKIN LYMPHOMAS LYMPHOMA, PREVIOUSLY TREATED WITH ADRIAMYCIN-

CONTAINING COMBINATIONS

Gy. Varga. Z. Borbenyi, K. Piukovics, I. Marton

2nd. Dept. of Int. Mcd. Szcnt-GyOrgyi A. Med. Sch. Szeged. Hungary N. Hajm, A. Talmor, M. Ben-Shahar, M. Leviov, R. Epdbaum.

Granulocyte colony-stimulating factor (G-CSF) reduces the degree Department of Oncology, Rambam Medical Center and Technion - Israel

and duration of neutropenia in patients with high grade non-Hodgkin Institute of Technology. POB 9602, Haifa 31096, Israel

lymphoma after chemotherapy. The purpose of this study was to

determine the effect of G-CSF on the leukocyte recovery in patients The pattern of fint relapse was retrospectively analyzed in 71 patients

with high grade non-Hodgkin lymphoma. One group of the patients [pts] with histologically aggressive non-Hodgkin's lymphoma who had

received G-CSF (Neupogeo s u«/Vg) in 25 cycles of ProMace-MOPP been treated previously with adriamydn-containing chemotherapy,

on days 9-14 and in 32 cycles on days 18-27. The leukocyte count with/without radiotherapy. Relapse was limited to previously involved

increased from 6.8 ± 22xG/l to 14,4 ± 6.8xG/l in 96% of the

patients within 2-6 days. After day 14 of the cycle (Lv. Methotrexate) [old] sites in 11 [15%] pts only and to previously uninvotved [new] sites

the leukocyte count dropped to 32 ± \2xG/\ and G-CSF raised it in 22 [31%] pts. In 38 [54%] pts, relapse occurred in both old and new

to 15,7 ± 6,6xG/l within 2-6 days in 87% of the cycles. In a sites. In none of the pts was the relapse limited to a site of prior bulky

second group of patients Neupogen was given only in febrile disease. For the following analysis, bulky sites and sites that underwent

ncutropenic states during 21 cycles. The febrile neutropenia occurcd surgery or radiotherapy were excluded. Relapses in new sites were more

8 + 6.5 days after the cytostatic treatment. One part of patients frequent among extranodal [EN] than among nodal sites. Of 47 EN sites

received first line therapy, others were heavily pretreated. In both at the time of recurrence, 41 [87%] were new she*, compared to 99/178

group the initial WBC count was 1,0 + O^xG/1. In the former group [56%] nodal sites [p

were treated with atogeneic BMT. AS patients responded to ASHAP (13 CR, 3 PR). PMItCEBO was given for a total of two weeks (3 patterns) four weeks (3 patients) six

With a median follow-up of 24 months al CR pts except 1 have remained alive in weeks (2 patients) dgbt weeks (9 patients) and twelve weeks (2 patienu). Overall

remission, while the 3 PR pts have progressed and died. Sixty-four pts received response rate was 14 cat of 19 (74%) with 7 patients achieving complete clinical

ASHAP as a salvage regimen to be foBowed with HOC and APBSCT or ABMT for remission. Median duration of response was twelve months (1-27 months), actuarial

those responding pts under age 66. Their median age was 53 years (range 17-SO). mean survival from relapse was seven months with DO difference between histologtcal

The larger histologic subgroup was intermediate grade lymphoma Twenty-four had types. Two patienu died from mddental cardiac disease. There were ten episodes of

never attained a prior CR. A total of 52 (81%) pts responded (29 [45%] CRs. 23 neutropeula (total nentrophll coont less than 1.0) with three episodes of neuiropenic

(38%) PRs) and 34 of these pts received HDC for consolidation With a median sepsis. This hvltxirt one patient with bone marrow failure doe to diffuse Infiltration at

follow-up of 33 months, overall survival was 52% at 56 months Time to treatment relapse. In l-8%; elevated LDH=47%; stage III-IV-41%; tumor>IOcm-28%;

ranged between 16 and 78 (mean 42.5). They received 1-6 MICMA courses (median 3). Eleven extranodal site>l-21%; bone marrow involvcment=l3%.

achieved the CR, 18 a PR (reduction of rymphoma > 50%), 14 were NR. One patient is still

unvaluable. 38 patienu received G-CSF 5 ug/kg torecruitperipheral blood stem cells or in case The MAMI protocol (2 cycles every 21-2Sd) consisted in Mitoguazonc (400 mg/m2

of severe neutropenia (llll"#l of non cross rcsutant drugs, ind

only few other drugs can offer therapeutic chances for the NR p"i"«« Our MICMA regimen pts are surviving more than 2 yean after salvage treatment. Adverse prognostic

constsu of chemotherapeutic agems not administered before or employed with a different factors for survival at the time of MAMI for all pts were: refractory disease

schedule. The MICMA Is well tolerated overall if associated to the G-CSF, that allows also the (p 10cm (pO.OOl), and elevated LDH (p7S% reduction) (1 proceeded to BMT), 2

a PR and 3 showed no response. Median folow up I* 30 months (range 3-45), and

resistant HD in our small group of such pts.

6 lurvtve In CR, 3, 12, 16, 2 0 , 33 and 38 post chemo.

We beSevt thl* to be a promising treatment for difficult HD and toxidty ha* been









160 9. Aggressive lymphoma

597 598



PHASE II TRIAL OF VINORELBINE (VNB) IN PATIENTS WITH Vlnorelbine (Navelbine) as salvage therapy in heavily pre-treated patients

REFRACTORY LYMPHOMAS. with Hodgkln's and non-Hodgkln's Lymphoma.

V. Churion Sileni', A. Bononi', M. Rupolo1, F. Gaion', L. Sah/agno1, S.M.L. S Rule, M Tlghe, S Johnson, Department of Haematology, Taunton and

Aversa', M. Sorarii', M.V. Fiorentino'. Departments of Medical Oncology,

Somerset Hospital, Musgrove Park, Taunton, Somerset, UK

Hospitals of:' Podova, ' Rovlgo, ' Camposamptero; Italy



The purpose of this phase II study was to evaluate the activity and the toxicity of Thirteen heavily pre-treated patients with relapsed Hodgkin's disease (6

VNB tartrate (a novel semisintetic vinca alkaloid) in pts withrefractorylympbomas. patients) and high or intermediate grade non-Hodgkin's Lymphoma (7

Patients and Methods: From 1993, 17 patierts (pts) were enrolled. VNB wai patients) were treated with the semi-synthetic vinca-alkaloid Navelbine as

administered as a single agent (without steroids) at die dose of 30 mg/Tn'/wk i.v., in single agent salvage therapy. The median age of the patient group was 47

250 ml 0.9 % sodium chloridrc and infused in 30-43 min. Treatment was continued (range 24 to 78 years). The median number of prior treatment schedules was

until progression of disease. Median age was 67.5 (range 17 to 87), 4 pts were males 2 (range 1 to 8) of which 6 patients had had a prior autograft procedure and

and 13 females; median PS was 60 (40-100). Four patients had low grade rymphoma, one patient a prior allogeneic bone marrow transplant. At treatment with

11 had intermediate or high grade and 2 had Hodgldn'i disease 5 pts had B Navelbine, the stage of disease was as follows: 7 patients stage IV, 2 stage

symptoms and 7 had bulky disease. Extranoda) involvement was present in 5 pts and HI, 3 stage II and one stage I. Navelbine was given at 25 mg/m2 IV on days

bone marrow (BM) infiltration in 2. The median number of prior chemotherapy 1 and 8, each course repeated every 21 days. The median treatment cycles

regimens was 2 (range 1-5); prior radiotherapy in 5 (31%). 2 pts underwent also

received was 3 (range 1 - 9). The dose of Navelbine was reduced in 4

ABMT and 5 Interferon. A total of 125 cycles of VNB were administered.

Results: 16 pts are evatuable. Seven pts (44%)responded:complete remission (CR) m

patients due to neutropenia and treatment delayed in 5 patients following

2, partial remission in 5; stable disease in 7 (44%). One completerespondcrunderwent infectious complications,in 2 patients febrile neutropenia, one pneumonia and

ABMT and is in continuous CR (7+ mo.) The median response duration was 3 mo one shingles. The only other side effects were severe phlebitis and

(range: 1-5). The major dose-limiting toxicity was hacmatologic. Dose reductions were constipation seen in one patient. There was little nausea or vomiting and no

necessary in 5 pts (31%). A delay (median duration: 1 wk) was required in 12 pts (48 alopecia seen.

cycles, 38%). G3 neutropenia occurcd in 3 pts, G3 anemia in 2 pts; thrombocytopenia

was only mild. Non-hacrnatologic toxicity: G3 constipation in 1 pt, G3 fever in 1 pt,

Of the 13 patients, 6 progressed on treatment, one had stable disease for 4









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G3 nausea and vomiting in 1, G2 alopecia in 1. Five episodes of chemical phlebitis

occured. No severe peripheral ncurotoxicity was observed.

months, 5 achieved partial remission of median duration of 9 weeks (3 weeks

Three pts, who started VNB with platelets (ph) . had CNS chemotherapy In this patients was COP-BLAM, CHOP-based, Dexa-

progression after four cycles. No toxic death occurred. Grade 4 neutropenia and Ihrombo- BEAM, A-SHAP, EDAP, Chtorambucil + Predniso)one(Chl+Pred). The

cytopenia occurred in 50% and 6% of administered cycles, respectively Median absolute results of treatment Oex areCR-2, PR-3, partial fesponse-5, no

neutrophil and platelet counts at nadir were 500/uL (range:100-1,300) and I27,0UVUL

response-1, progression-1. Overal 3-years survivals after AutoBMT

(range: 19,000-183,000), respectively. The nemrophil nadir usually occurred between day 9

and 12. AU patients whh grade 4 neutropenia at nadir rccovcicd lo>300/uL ANC within 2-3 and/or PBSCT are 55% in HD, 47% in NHL

days. No transfusional support was required.CKhcr side effects were grade i-2 anemia (46% of Dex treatment Is effective modality for resistant/relapsed

cycles), grade 1-2 mucontis (36%), grade 1-2 infection in absence of neutropenia (15%) and low/Intermediate malignancies. It should used as a nontoxic therapy in

grade 2-3 SGOT/GPT increase (4%). One paiient suffered from grade 3 febrile neutropenic elder patients or as preface for further intensificationn including HSCT.

pneumonia. Hematologic toxicity never persisted at the ume of recycling. Delays or dose

HSCT in patients suffering of high grade lymphomas or HD is

reductions were needed only in seven (14%) cycles, due to earahemaological causes.

Conctashns. The CEVOP-B program whh E and V given as Cl is feasible, wdl tolerated and effective If transplantation has done in CR or PR.

sufBcicnuy active. The study is ongoing.









9. Aggressive lymphoma 161

601 602



TREATMENT OF RELAPSED AND RESISTANT LYMPHOMA WITH UPOSOMAL ANTHRACYCUNES: FEATURES ENSURING A FUTURE ROLE IN

LIPOSOMAL DAUNORUBICIN (DAONOXOMB™, NEXSTAR) THE TREATMENT OF LYMPHOMAS



F.M Muoola and P. GDI, USC - Norria Cancer Center. Lot Angeles. CA 9 0 0 3 3

D.S.Richardson'. M.Tighe'.S.AJohnfon'.S.M.Kdiey'.A C.NevHand1

Departments of Haematology, Royal London Hospital1, London, El IBB, UK DaunoXome and Doxil have recently become available for trsatmint of

&. Taunton and Somerset Hospital2, Taunton, TA1 5DA, UK. AlDS-related Kaposl's Sarcoma. Their toxicotogic and pharmacologic

properties have b m n defined (1,2), but the spectrum of antltumor activity it

16 patients with relapsed or resistant lymphoma (14 with NHL, 2 with HD) only recently being explored. Phase II studies in malignant lymphoma a t

were treated with liposoma) daunorubicin between September 1993 and single agents are ongoing with both DaunoXoma and Doxil. DaunoXom* a t

November 1995. Patients were heavily pre-treated (mean number of regimem weB a t Doxil may selectively be taken up by tumor tissue. Both have

- 3.6,range 1-8). 6 had received high dose chemotherapy with ABMT/PBSCT attenuated subjective toxictties and lesser propensity to cirdiotoxichty.

and one, a sibling allogeneic transplant. 9 patients received 1-4 cydes and one We have found DoxJ active against ovarian cancer with

myeJosuppreuion occurring exceptionally (confined to patients who have

patient 24 cydes at a dose schedule of 40mg/mJ q 14days No patient in this

had total abdominal radiation or prior mhomycin therapy.) Experience in 4

group achieved an objective response; 7 have died (5 from disease) and 3

patients with radiation during Doxfl treatment indicant treatment Is tolerated

remain alive, 2 with progressive disease at S months, one has proceeded to

without undue skin toxidty. Phase I study of Doxil with paclitaxel it

PBSCT and is in dinical CR at 4 months post transplant. The patient who ongoing. DaunoXoma and Doxil may contribute to future treatment of

received 24 cydes of treatment, remains alive and achieved excellent rdief maBgnant lymphoma: selective localization, no dose Bmrting

from bone pain which had been refractory to previous therapy. 6 patients myelosuppresskxi, and tolerance with radiation are among the attractive

received 1-3 cydes at 120mg/m2 q 21 days; one patient achieved a short CR features to be explored.

(4 weeks) and another a partial response after cyde I and is currently Doxfl and DaunoXome have been studied most extensively in AIDS-

receiving further treatment. A further patient achieved a minor response related KS. Both agents are weD tolerated with reduced frequency of heir

(duration 4 months from start of therapy) although cydophospharrude was loss, mucositis. Accumulative doses of 1,000-15,000 m g / m 1 of

also administered for PBSCH during this period. 3 patients did not respond DaunoXome In nearly 2 0 patients have not shown any evidence of cardiac

and 4 have died from their disease Toxiaty, judged according to WHO disease cfinlcally or by repeated echocardiography. Doxil hat also been

criteria, was predominantly haematological and dose dependent, with grade 4 shown to be safe but the cumulative doses in excess of 6 0 0 m g / m 1 have









Downloaded from http://annonc.oxfordjournals.org/ by guest on January 2, 2012

only been reached in a smaD number of the cases thus far. DaunoXome hi a

neutropenia or thrombocytopenia affecting 8 patients (SOS). Infection was

phase III trial was found to be at effective a t a combination of doxorubicin,

common, affecting 10 patients, although in all but 2, episodes were estimated

Adriamydn, Weomycin and vincristint and thus w a t recommended for

to be grade 2 or less in severity. Non-haematological toxiaty was mild, in

approval at first line treatment for KS. Doxil h a t been approved for use in

particular, no patient in the higher dose group snowed any deterioration in AIDS-KS as second line therapy after failure of other chemotherapeutlc

cardiac function. Liposoma] daunorubicin at 120mg/m2 appears to have some agents.

activity against refractory lymphoma and toxiaty is acceptable. We suggest

that further studies with this agent are required 1. GB PS et at.. JCJin QnscJ 13(41:996-1003, 1 9 9 5 .

2. Uziely B et al J CJin Qn£oj 13(7): 1 7 7 7 - 1 7 8 5 , 1 9 9 5 .









603 604



RANDOMIZED PHASE III STUDY O F INTERLEUKIN-3 (rhlL-3) AS ADJUVANT When a malignant lymphoma patient is "elderly"?

T O IEV-CHEMOTHERAPY FOR RELAPSING AGGRESSIVE LYMPHOMAS:

H.H.Gerhartz, F.Mandelll, T.Phllip, S.Tura, R.Helru:, R-Greil, H.J.Sem, C-Huber. Busetto M', Sarvagno L 2 , Guglielmi R B \ Giuslo M \ Coghetto F 5 , Bononi A 6 , Mandolin

W.BJau. LFarber, I.SkJenar, U.Haus, KDnikum Kalkweg, Med. Dep. 47O55 G 7 , Radio E 1 , Anloncllo M 1 , Endrted L», Rosa Bun A 1 0 , Manenlc P " . Fcrraoi E*,

Dulsburg. Puccctli C , Balli M 3 , Zoral PL 5 , Polico C 7 , Bassan F 1 0 , Fiorentino MV 2 and Puii GB1

Patients (pts.) relapsing with aggressive lymphoma loUowIng intensive

combination chemotherapy ( c t x ) usually have a poor prognosis due to tumor

resistance and a bad hematopoietlc reserve. In a imitlcenler trial 179 pts. (48

with HodgWrfs disease (HD) and 131 with non-Hodgktrfs lymphoma (NHL) were

treated with the IEV regimen (Ifosfamtde 2,5 g / m 2 l.v. day 1-3, Epirubictne 100

m g / m 2 l.v. day 1 , Etooostd 150 m g / m 2 Lv. day 1-3) followed by either IL-3 (10 Non Hodgkin'i rymphomas are stcadry increasing among people aged 60 and more, and

pg/kg body weight) or placebo s.c. day 4-15. Seventy-five (42 %) pts. were in first everybody knows (hat elderly people has a reduced tolerance lo oncologic treatments. Bm

relapse, 56 (32 % ) were primary progressive and 48 (27 %) had their second or when a patient may be defined 'old'7 There b not a umvocal definition Age is a continuous

subsequent relapse. The mean age was 53 years, 118 were male, 61 female. quantity, without steps, nevertheless we need a coJpoirt after which the chemotherapy

Beside one or more ctx. regimen 27 % had also previously received radiotherapy scheme should be less besny and dosa of cbemotberapy should be reduced to rvxrid an

One hundred and seventy nine pts. entered cyde 1, 143 pts. received a 2nd e n c s s of toxidty. Time ago the cutpoim was around 60 >t)L; 50-39 (I63JJ); 60-69 (243,32); 70-79 (191,13), 80 and over

groups CR rates were 37 % lor 1st relapse, and 18 % for 2nd or subsequent (71,5).

relapse or for primary progression. The number of Infections and transfusiora as As n-c expected, a highly significant difference in tun-hil among NHL and HD patients

well as the response rates were not significantly influenced by treatment with IL-3 was found (both pO.OOOl). The plotted actuarial J-j-ean OTCTIM mr\ rval probabilm- (5-ATI

versus placebo, Indicating that IL-3 Is Ineffective in the setting of Intensive rescue OS) and hi SE remain stable from the younger age interval (10-19) and s u m worsening

around SOies, with a sleeper decrease for NHL. For NHL the 5-\TI OS % ij 75 0 (± 15.81) at

chemotherapy: Survival probatility at 1 year was 55 % for NHL and 70 % for HD .

10-19 y n , 67.77 (± 7.05) at 30-39 j-rs, 78.47 (± 4 13) al 50-59 'yn, 48 03 (± 4 64) at 6U-69,

Conclusion: IEV chemotherapie proved to be a very effective and tolerable

38.0 (± 5.17) it 70-79 y n and 30.71 (± 9.0) m-er. F o r H D t h e 5 - > n O S % i s 92.59 (± 5 0)

treatment In both relapsing and primary progressive lymphomas. The Insufficient

at 10-19, 92.94 (± 3.65) at 30-39 yn, 83.86 (± 9.46) at 50-59 yrs. 73.95 (± 13 85) at 60-69

dinical effects of IL-3 alone might be overcome by continued application together y n , 67.31 (± 13.62) at 70-79 y n and 31.65 (± 18.19) over. Further anahscs are needed, but

with G-CSF or GM-CSF. these results should be considered in therapy planning.









162 9. Aggressive lymphoma

PROGNOSTIC FACTORS FOR MALICNANT LYMPHOMAS OF THE ELDERLY. PROGNOSIS OF ALL PATIENTS AGED 70 YEARS A N D OVER WITH

A REPORT OF THE 'GRUPPO V E N E T O LlNFOMl'. INTERMEDIATE OR HIGH GRADE NON-HODGKIN ' S LYMPHOMA

Busetlo M*. Ciusto M", Bononi AM, Police R \ Ruppolo M°,Endri«i LA, Gaton F+. R o n D I A G N O S E D BETWEEN 1988 A N D 1992 IN NOTTINGHAMSHIRE

Bias Afii, Maneole P+. Fcrrazzi E#, Puccetli C , B a t o n F(S\ Avcrsa S*, Pizri GB*

RM.BesseIL AJ.Motoney. JJlOcher. J.Tbomlcy,

•Dtrkkn. * laSoKnpb t O u h f a - U o m (VE); TXvUoo. d Ouu-t^i. r«luv»: . D n « ,

C«idn«neo « Oaafafi^b—ao (VI); >D>vn, Department of Clinical Oncology. Nottingham City Hospital N H S Trust,

United Kingdom



Malignant non-Hodgkin ryrrrphomas are becoming more and more Crcqueni world-wide, A uniform treatment policy was used for elderly patients ( > 7 0 years) with

especially among elderly. The Vcnclo region teems to be al first places in Ilflly Tor morulit)'

intermediate and high grade non-Hodgkin ' s lympboma. During the period

sad In"^"™ Since 1991 a rympboraa surveillance project, the 'Rcgutro Veneto LrnfamP.

was established among some oncologic centers of this Region. Data from this register are 1988-1992 3 0 7 palienu were diagnosed in Nottinghamshire (served population

I.I million) as having intermediate and high grade non-Hodgkin i lymphoma.

On September, 1995 there were 1029 registered cases of malignant lymphoma, 509 of 120 of these patients were aged 70 years and over al diagnosis. 12 patients with

whom aged 60 or older. These latter were included in this study. Four hundred! and sixty- non-bulky ( 'cUne-containuig

chemothcrapaJtic combination for the treatment o f elderly patients with

In the U s yean Kvenl reporu regirdal the tratmeat of NHL in elderly Since 1989 in our hiitologicaily aggressive NHL was evaluated. Between January 1988 to november

Division vrc utilized • protocol ipecifkalry denied for elderly puienU (65 y o n or older), MiCEP 1992, fift>-one previously untreated elderly patients (median age : 79 years : range

protocol »tjch w u utilized for the tratment of intermediate or hifb-frade NHL. We have recemery

70 to 90) stage III (19 patients) or IV (31 patients) intermediate or high-grade

reporud nUeroting untie imtinitioc results mint MiCEP protocol (Leukemii md Lymphnmi.

N H L were treated. All patients received cydophosphamide 750 mg/m' IV day I,

1995). We report now Ibe renhi of a nmrlnmirrd tral (MiCEP n P-VABEC) in • mihimito-

vtndesine 3 mg/m 2 IV day I and prednisolone 5 0 mg/rrr1 orally day 1 to day 5

study. Tbe centers were Haematotogy Depoiement of Florence and penfene Diviskns of General

(CEP regimen), every 14 days for 6 months. Patients were classified according to

Medicme.

the risk group defined by the international index as low intermediate (14 patients),

Dct^mi jane 1993 >nd december 1995 54 rl^i^nl" over 65 yean were randomized: 28 entered

MiCEP (Mitonmtrone, Ciclopbupharaidc, VPI6, Predabcoe) arm (A) and 26 entered P-VABEC high intermediate (16 patients) and high risk group (21 patients). Among the 51

(Prebisone, Vincristin, Adnamkin, Blroiririn. VP16, Cictopbosphimide) inn (B). The eligibility patients, 17 achieved a complete remission and 9 had a partial response, with a

requtremenu were F.O.H tuttotype according to Wortinf Formulation, P^. o now only

Age Is an Important prognostic parameter, especially In patients with anecdotic*! report* war* tvalatot* in th* currant Utrature aa to th* natural history and

treatment resuts of tiddly pt* affected by Bl_ Between 1988 and 1955, more than 1000

advanced high-grade non-Hodgkin's lymphoma (HG-NHL) who require more ca**a of non-Hodgkin's lymphoma* among adult HIV-ntgative pU ware ob*«rv*d at our

Intensive therapy. W e Investigated the potential of G-CSF for reducing Institution. BL wa* Diagnosed In 30 u u t and among them 11 pt* (36.6%) wtre older than

myetotoxicity, which Is the most important dose-limiting factor for 60 yrs (median ag* 68 yra; rang* 60-84 yr»). CMcal leaturt* at diagnosis wtre a* follow*:

chemotherapy. Between March 1993 and June 1995, 158 previously mate/lemal* ratio 6/S; stagkx) according to NCI paSotric branch: AR-B (1-5), C O (2-3);

untreated patienu 60 years and older with HG-NHL were included in a staging according to Ann Arbor II (2 pt*), IV (9 pts); B symptom*: 3/11: bufcy dl**aa*: 6/11;

ejUiamxIal diMa**: 8/11 (abdominal organ*, 6 pt»; (kin, 1 pt; t*stid*, 1 pt); bon* marrow

cooperative randomized comparative trial and treated with a combination

Involvement: 4/1 1; CNS Invotvemenl: 0/11; lncrea**d wrum LDH level* (> 500 LM.): 7/11;

therapy including cyclophosphamide, mitoxantrone, vfneristine, etoposkJe, reduced absosrla CD4+ T-cel court* (n(CR) wa* achieved In 8 out of 11 pt* (73%), wilh

response (CR) In 59%: 60% In the VNCOP-B plus G-CSF group, and 58% in a madlan CR duration of 35.5' month* (rang* 3*-91 •). Four pt* ar* maim lining a comlnuou*

the VNCOP group. At 30 months (median 24 months), 68% of all CRs were CR by longtr than 64 month*. Median overal survival lor th* whol* group of 11 pt* wa* of

alive without disease In the G-CSF group and 65% in the control group. 15 month* (ranga 1 -91 •), whfl* nwtSan survival ofpt* achieving CR wa* of 37* monlh* (rang*

Neutropenia occurred In 18/77 (23%) of the G-CSF treated patients and In 6*-91•). Tht 3 non-CR pt* (1 PR and 2 ptogreulons) dbplayed an overafl *urvlval of 11

40772 (55.5%) of the controls (P*= 0.00005). Clinically relevant infections month* (ranga 1-15). No Uwnpy-rdaled toxic death* were obttrved In tha whol* group of

pt*. Intertttlngly, txlranodal tltau** (In particular tkki) repr*«*nl*d th* u*u*l *ttt* o) both

occurred In 4/77 (5%) of the G-CSF group and In 15/72 (21%) of the

(fit**** progrenion and relap**. Our data IndkMle that b I* pottM* to obtain a CR in an

controls (P= 0.004). The delivered dose intensity was higher In patients •tovated fraction of BL» alao In paHerU over 60 yr* of eg*, and that a durable CR can b*

receiving G-CSF (95% vs 85%) but the difference was not statistically achieved In th**t tubject*. d**pt* th* advanced stage 60 years with a mean age of xx years.There

1986) and 16 with Magrath protocol ( Magrath IT Uppicot 1988) after this time . Median were 289 men and 248 women.

age was 40 y n (IJ-69),19 were mates and 12 females; according to Ann Arbor criteria 7

According to the Arm Arbor staging system 103 patients

pts had in localized stage, 24 advanced stage m or IV; 11 bone marrow involvement and 4

were In stage I, 86 In stage II, 48 In stage III and 300 In

CNS localization. LDH level was above normal value in 17 pts. The clinical characteristics

stage IV. I89 pts. had B symptoms. Extranodal disease was

were similar in the two groups except for stage that is more advanced in p u treated with

Magrath protocol (p<0.01). ATI p u with limited stage received radiotherapy « found In 257 cases. The moat common htstopathotogteal

consolidation after induction chemotherapy. types were centroblastlc and Immunoblastlc h/mphomas.

Results, i ) Stanford mdtnen: all p u with localized stage achieved CR and only one During first line treatment 491 pts. redeved combination

relapsed. OS was 86% with a median FU of 70 months. Only one pt received BMT. Of the chemotherapy ( CHOP/CHOPIDce 377 cases, COP 86, other

8 p u in advanced stage, only one achieved CR and subsequently relapsed. All pu died 28) and 248 pts. had Involved field radiation. Complete

within two years, b) Mapath regimen: only one pu with localized stage was treated and remission was obtained In 292 (56%) of the cases. At 5

achieved a CR. Fifteen p u in advanced stage were treated and CR was achieved in 12 years 158 (29%) pts. are afive without disease, 32 (6%) pts.

(80%), 3 p u did not respond and died within i months. Nine CR's were given BMT (8 are with lymphoma. The survival of pts. with stage I and I I









Downloaded from http://annonc.oxfordjournals.org/ by guest on January 2, 2012

autologous and 1 allogenic) as consolidation treatment. Six are in cominous CR, one died was statistically significant better than pts. with stage III and

of transplantation toxicity and two relapsed. Three CR's did not receive any consolidation IV disease. The causes of death could not be established in

therapy and are still in CR. With a median FU of 18 months OS for p u with advanced all of the cases; 224 (42%) pts. died from rymphoma, 27

stage was 4 3 % (5%) pts. died from other diseases/ other cancers, 7 (1%)

Conclusion. For localized disease the results of two scheme are similar. For advanced stage pts. died from complications and 23 (4%) pts. from

the Magrath protocol obtained better results. The original Magrath protocol was proposed unknown causes.

without any consolidation. Our experience with Stanford schedule suggested us to use BMT

as consolidation b m t t e r o k of BMT Ucomroversial. Randomized protocol are warranted

to clarified this question.









SECOND LINE CHEMOTHERAPY IN HIV-RELATED NON-HODGKIN'S EPIRUBICIN, BLEOMYCrN, VINBLASTINE AND P R E D N I S O N E (EBVP)

LYMPHOMA: EVIDENCE OF ACTIVITY OF A COMBINATION OF VP16, CHEMOTHERAPY (CT) IN COMBINATION WITH ANTIRETROVIRAL THERAPY

AND PRIMARY USE OF G-CSF FOR PATIENTS (PTS) WITH HODGKIN'S DISEASE

MITOXANTRONE AND PREDNIMUSTINE IN RELAPSED PATIENTS

AND HIV INFECTION (HD-HIV).

D. Emmie*.M.Spin**,S. Sandri*.R.Gastaldi0, E. Nigraj,AM. Nosarr\G. Magnani#,

U.TtonkD.Errarae,CGisseIbrechUP.MaroUcttu, YKemcis,A. Ridolfo.M.Mazzetli, A. Lcvis,

E Vaccher* and U. Tirelli*. D. Venai, G. Rossi, M. Span, E. Vaccher for the European Imergroup Study HD-HIV; Aviano

• Aviano Cancer Center, Italy. * University "La Sapienza", Rome - Italy. 5 Hospital Cancer Center-Italy.

"Amedeo di Savoia", Turin - Italy.A Hospital "Niguarda Ca Granda", Milan - Italy. #

Objective: The optimal therapeutic approach lor pts with HD-HIV is unknown. In an attempt lo

University of Parma - Italy.

improve the results that we obtained in a previous prospective study with EBV without G-CSF

Purpose: To evaluate the feasibility and tcti vilyof a second line chemotherapy regimen (Cancer, 73:437-44,1994), mJmuary 1993 we sanedasecond trial consisting of CT.concomitani

consisting of VP16, mitoxanlrone and prednimusine (VMP) in patients with relapsed amiretroviral therapy (AZT or DDI), and G-CSF.

or resistant HIV related non-Hodgkin's lymphoma (HIV-NHL). Methods: Up lo October 1995,27(23 M / 4 F ) consecutive previously untreated pu (median age

Patients and methods: Twenty-one patients were consecutively treated. Thirteen 33,range2M9 years) with HD-HIV were enrolled. Median performance status was 1 (range 1-3).

At diagnosis of HD, 7 (26%) of pts had AIDS, 3 (11 %) ARC, and 17 (63%) were asymptomatic.

patients were resistant to primary chemotherapy and eight patients had relapsed after Eigroy-Tivepcrcemc/puhadBsyraptcnsaiHDrjre9eniaiion.PunsxrWdE70rng/rn2Lv. on day

first complete remission (CR). VP16 and prednimustinc were both given orally at dote* l,B10mgAn2Lv.ondayl,V6mgfatf Lv.ondaylarKlP40mgAn2p.afrornday ltodayS.Counci

of 80 mg/m 2 daily for 5 days, and mitoxantrone was given i.v. at a dose of 10 mgAn2 were repealed every 21 days for six cycles. ACT (250 mg x 2/day) or DDI (200 or 300 m j x 2/day),

on day 1; cycles were repeated every three weeks. when AZT was previously used, were given orally from the heginning of CT. G-CSF was given m

the dose of 5 mcg/xgMay i c from day 6 to day 20 in all cycle*.

Results: Nineteen out of 21 patients are evaluate forresponse.The median number of

Remits: ClinJco-pmhologJc characteristics of pu and response to thenpy are shown in the labtc

cycles actually administered was 2 (range 1-5). A CR occurred in 5 out of 19 patients

(26*; exact 95% confidence interval: 9% to 51%). Four of these CRs were observed • pu CD4+ at diagnosis Subtype Stage Response DFS

in the 7 evaluaWe relapsed patients. Out of 45 cycles evaluaWe for toxicity, severe cmercdVfevoluablc median I (range) MC&LD III & IV Oft CR ot2vn

ncutropenia (<500Anl) occurred in 19 (42%) cycles and severe Ihrombocytopcnia 27/21* 187(6*12) 18(66*) 22(82%) 90% 71% 4 3 %



(<25.000Anl) In 6 (13%) cycles. One toxic death occurred due to a sepsis during • 6 pts are still on treatment.

neutropenia. The overall median survival was 2 months (range, <1-13); the median Toxicity was moderate with grade 3-4 leukopenia and thrombocytopenia in 7 (33%) and in 2 (10%)

survival time for the 5 patients with CR (13 months, range 6-13) was statistically pu respectively. Fifteen out of 21 pts received AZT and 2 pu received DDL Only 6 (28%) pu had

signi fleam longer (pMtSXH) than that observed in patients without CR (2 months, range oppenuniaic infection* (01). duringor after CT (median follow-up, 14 months). No change of


therapy. Six out of 15 (40%) pu who achieved a CR relapsed. Overall, HD progression alone and

Conclusion: Although the overall prognosis of patients with resistant and relapsed in association with 01 was the cause of death in 46% andfa15% of pu respectively. The median

HI V-NHL is very poor, palliative therapy with VMPcan be effective and relatively safe. survival was 14 months with an actuarial survival me of 33% at 24 months.

Prolonged survivals have been observed in some patients who had relapsed after initial i-nmhinM mftrryo wm frftiht* Hnwrvrr, ^llh^igh \hr fT» M f rinujnrri mm

chemotherapy. rm

satisfactory, the number of rebmjed pts was Wjh and overall median survival was not different C a

our previous experience or from literature. Taking in constdemion the moderate toxicity, we are

Supported by AIRC *95 grants. cunentlyaiaskien^higherdcaesc/CTatsrjorteriniervabwuhthesupponofG-CSF. Supported

by AIRC "95 and ISS "95.









9. Aggressive lymphoma 165

ASSOCIATION O F ZIDOVUDINE (AZT) AND METHOTREXATE (MTX) EVALUATION O F RECOMBTNANT GRANULOCYTE COLONY-STIMULATING

FOR TREATMENT OF HIV-RELATED NON-HODGKIN'S LYMPHOMAS FACTOR (G-CSF) AS PRIMARY PROPHYLAXIS IN HIV-RELATED NON-

HODGKIN'S LYMPHOMA (NHL) TREATED WITH PROMACE-CYTABOM (PR-CY)

(NHL).

F Ghcrlinzcni, P Tori, P Mazza*, 0 Corooado", P Cos»iglioU°, MC Miggiano, PL G. Rossi, A Ro, A. Donisl, S. Casari, R. Stellini, G.P. Cadeo , G.P. Carosi. Sezione

Zinzani, M Magagnoli, F Chiodo", S Tura. Ematologia, Clinics Malattte Infettiva, I Divisions Malame Irrfetttve, Spedali Crvili,

Inst Hematology "Seragnoli", University of Bologna; •Dept. Hematology SS. UnJversita degli Studi, 25100 Brescia .Italy.

Annunziata Hospital, Taranto; "Inst. Infectious Disease, Univeoity of Bologna, Italy.

Growth factor support in HIV-related NHL is generally recommended, but its cost-

effecUveness has not been clearly established. W e compared the effects of G-CSF

Zidovudinc (AZT) can possess a significant anliprolifcrative activity when support In two consecutive groups of patients (pts) with HIV-related NHL treated at a

combined with drags that disrupt "de novo" thymidylate synthesis, such as 5- sixjto institution with the same chemotherapy (CT) program PR-CY, +/- G-CSF.

fluorouracil or MTX. HIV-related NHL are constantly associated with a very poor PR-CY was given to 33 HIV+ pts with aggressive N H L The 21 ptt diagnosed after

prognosis irrespectively of the regimen of conventional chemotherapy. 01/01/92 also received G-CSF at the fixed dose of 300ug/sc daily from day 9 to day

In January 1993 we started a clinical trial with the objective to evaluate both 20 of each cycle. The 12 earlier pts never received G-CSF and acted as controls. At

NHL (flagnosls, G-CSF treated pts had significantly more advanced HIV disease

efficacy and tolerance of the combination AZT + MTX m HIV-relatcd NHL. 29

compared to controls ( CD4+ oount <0.100/ml in 7 1 % vs 17%; P O . 0 0 3 ) and lower

patients (pti) (22 males, 7 females) have so far been enrolled in the trial, mean age is WBC counts (3.84 vs 6.66/ml; P<0.03).CIWcopathotoglcal features of NHL were

34.5 years (range 23-49); 13 (32%) are drug abusers, 19 (65%) had less than 100 comparable. A total of 137 cycles were evaluaWe, 85 with and 52 without G-CSF G-

CD4/mmc. The most represented hiitoJogic subtypes are: diffuse large cell lymphoma CSF support significantly decreased the frequency of day-1 drug dose reductions

(14 pts), Burkitt (7 pti); stage IV was present in 21 pU (72%). 13 pu (45%) had from 3 7 % In controls to 8% in treated pts (P
bulky disease. Treatment plan includes three consecutive courses of MTX, 1 g/m 3 from 56% of cycles in controls to 9% in treated p u ( P O . 0 0 1 ) ; it improved the actually

delivered total doses of adrtamydn, cydophosphamide and etoposide (P<0.02), but

(days 1, 8 and 15) + oral AZT 2 g/m 3 (days 1. 2 and 3), 4 g/m3 (days 8, 9 and 10)

did not affect the frequency and duration of fever and of hospttalizatton. CR rate and

and 6 g/m 1 (days 15, 16 and 17). Folinic acid is administered 12 hours after each survival were worse among G-CSF treated pts. However, this result was related to

dose of MTX. From the 11th patient on, the treatment was continued with three more their more advanced HIV disease and differences were not significant when pts were

courses in case of complete or partial response. Out of 25 evaluable pu, 11 (44%) stratified according to their CD4+ count Among pts with a CD4+ count O.IOXVml,

achieved a complete remission (CR) and 9 (36%) a partial response (PR) (overall day-1 drug dose reductions and CT delay occurred in 10% of cycles with G-CSF

response rate •» 80%). In 3 pts CR duration is lasting more than 24 months. Median compared to 8 0 % without G-CSF (P<.001).

follow-up of the responden is 12 months. Grade I [1-1V neutropenia was observed in In conclusion, G-CSF support significantly improved dose-intensity In patients









Downloaded from http://annonc.oxfordjournals.org/ by guest on January 2, 2012

23/112 courses (21%). G-CSF was administered in 65 courses (58%). No patient treated with aggressive CT for HIV-related NHL, particularly in those with a CD4+

showed grade III-1V extrahematological toxiaty; only 1 patient developed an count O.100/ml, a subset which can hardly tolerate full-dose CT. However the

opportunistic infection. increased dose intensity achieved with G-CSF did not result in a better clinical

outcome and the major determinant of prognosis remained the stage of the

In conclusion, these data show that AZT + MTX combination is effective and underlying HIV disease.

safe in HIV-related NHL.









CUNICO-PATHOLOGIC CORRELATIONS IN 120 PATIENTS (PTS) WITH I11V- VlTAMlN-D-MEDtATED HYPERCALCEMIA A3 THE PRESENTING SYMPTOM OF

RELATED-SYSTEMIC-NON-HODGKIN'S LYMPHOMA (HIV-NHL): A T-CELL IMMUNOBLASTK LYMPHOMA.

MONOINSTrrUTIONAL STUDY.

G. Nasti*, E. Vaccber*. D. Emmte •, M. Spina', M. Tavio*. C. Snoonelli •, S. Saniarossa*. G. J.F. Seymour and D. Manor, Department of Clinical Haematology and Medical

Di Gennaro*. A. Carbone ° and U. TireUi*. • Division of Medical Oncology and AIDS; • Oncology, The Royal Mefooume Hospttal, Parkvflle 3050, Australia.

Division of Pathology - Avlano Cancer Center - (PN) - Italy.

HypercaJcemla Is an uncommon complication of non-Hodfjkin's tymphomas

HTV-NHLicpmtiuan betei uyo icons group of diseases, characterised by the presence of distinct (NHL), occurring in up to 15% of patients with Intermediate- or high-grade

molecular-paihological entities, but overall considered associated with a poor outcome. Few dteease. However, In patients with HTLV-1-related adult T-cell leukemia /

extensive studies on clinico-paihological correlations have been conducted. In this suidy we

lymphoma (ATLL) hypercafcemla ts present in more than 80% of cases and is

ouun ined the relationship between pathological and cli nical data obtained from aroonoisilutional

series of 120 pu with HI V-NHL, diagnosed and treated with chemotherapy from September 1987 known to be mediated through the systemic actions of parathyroid hormone-

toJuly 1995. AD cases were intermediate-high grade NHL according u the WP. and the updated related protein (PTHrP) which also mediates the humoral hypercalcemla of

Kiel classification. The main clinico-paihological correlations are reported in uble. most soBd tumours. In contrast, recent studies suggest that ectoptc production

Histo Ionic OTHIW of activated vltamln-0 (calcltrlol) by non-maBgnant macrophages within

G H J KiALC* Miscell P rymphomatous Ussue Isresponsttrfefor the hypercalcemla compficating B-cen

(n»18) (n*23) (n*39) (n'14) (n°27) value NHL Few patients wtth HTLV-1 negative T-cell NHL and hypercafcemia have

(*) (*) (*) (*) (*) been evaluated, and the mechanism of the hypercalcemla In these cases Is

unknown. A 71 y.o. woman presented with symptomatic hypercatcemla and

AGE yrs (mean, ± SD) 36113 38±8 34±g 38±12 37±11 NS

PRIOR AIDS 17 35 26 39 30 NS diffuse bone pain; serum C a ~ 3.1 mmol/l [normal: 2.1 - 2.6]. Nuclear bone

CD4 COUNT <100/mm3 69 0.009 scan had multiple areas of abnormal uptake suggestive of "metastases". She

33+ 59 rt* 63*

had no adenopathy or organ enlargement cfinicaOy or wtth Imaging, and no

STAGE m-IV 67 48 82 69 63 0.08

CR~RATE 56 24 41 25 16 NS evidence of a primary tumour sKe. Bone marrow biopsy was normal. Serum

SURVIVAL mo (median) 16.4* 6\6 8 9.8 6.7" 0.08 PTH was suppressed and PTHrP was undetectable. Serum caJcttrtol was

A

markedly elevated: 150 pmoM [expected: < 75). HfV and HTLV-1 serology were

KiALC » Ki 1 - anaplastic large celt + G vs ALC p • 0.05; * G vs Mis p - 0,05; • J vs H p « 0.02; negative. A biopsy of a rib lesion revealed T-ceD immurtoblastic NHL, stage

** Complete remission; • GviH p • 0.01 ;**Gvs Mis. p - 0.01.

tVA, International Index score « 3. The hypercalcemla promptly normalised

Our preliminary results show: 1) a relationship between histologic subtypes of NHL and degree following CHOP therapy and CR was attained after 7 cycles. Cafcftrlol levels

of immunodeficiency, with Burtdtfl and G (centroblastic) NHL »««nrijii-H wiili a significantly returned to normal. The patient remains In ongoing CR 12 months after

higher CD4 level; 2) a significant better outcome for NHL of the G subgroup than H subgroup. presentation. This case iustrates that the hypercateemia of T-cell NHL may be

This finding sujgestthat G NHL should not be included with H (immunobUstic) in the same vtamkvO-medSateu, as in B-NHL, rather than PTHrP-mediated as In HTLV-1+

category of diffuse large cell NHL, (Harris eta], 1994), since H NHL is attoriited with poor ATLL. Further, rymphoma should be considered as a cause In patients

outcome. presenting wtth hypercafceirda and muSiple bone "metastases".

Supported by ISS and ADtC grams.









166 9. Aggressive lymphoma

621



GLOMERULONEPHRITIS IN NON-HODGKIN-LYMPHOMA

REPORT OF 3 CASES AND LITERARY REVIEW

J. Zahner, D. Bach, B. Grabensee, W. Schneider. Medizinische

Klinik, Heinrich-Heine-Universitat, D - 40225 Dusseldorf

The association of Non-Hodgkin-Lymphoma (NHL) and glome-

rulonephritis (GN) is well-known for many years. Whereas in

Hodgkin's disease, minimal change GN is mainly observed, in

NHL various forms of GN are found.

We describe 3 cases of NHL-associated GN: 2 patients, one with

chronic lymphatic leukemia (CLL) and one with cutaneous T-cell

lymphoma, developed IgA nephritis, in a third patient with CLL

membranous GN was observed. 2 cases showed impaired renal

function and 2 had nephrotic syndromes, which improved after

treatment. In 2 cases GN and NHL developed simultaneously, in a

third lymphoma was followed by GN after 3 years.

Although the pathogenetic relationship between NHL and GN is

not fully understood, an immunological link has been postulated.

The high number of cases reported in the literature suggest inter-

dependence. Including our 3 patients we found 71 cases of NHL-

associated GN in the literature. NHL-associated GN is observed

more often in men than in women and more frequently in low-









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grade NHL - especially in CLL - than in high-grade NHL.

Compared to GN in M. Hodgkin, NHL-associated GN shows

higher rates of impaired renal function and nephrotic syndrome.

The majority of NHL do not show any predisposition to a special

type of GN, only in cutaneous lymphoma IgA nephropathy

predominates. Apart from simultaneous manifestation, sometimes

GN precedes NHL or develops after the onset of NHL.









9. Aggressive lymphoma 167



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