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AIDS AND HIV

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AIDS AND HIV





INTRODUCTION

 First cases in 1981 after PCP cases in homosexuals

 Defect in cell mediated immunity (CD4 T helper cells)

 AIDS = CD4 99%

 False -ve testing: “window-period” where infection occurs but antibodies

aren’t formed yet (may be months): 95% +ve by 3 months

 False -ves: seroreversion, atypical strains

 False +ves: blood transfusion from someone with antibodies to HIV, children

35 (prednisone 40 mg po

bid with a taper over 3 weeks)

 Treatment generally for 3 weeks

 Many do not clear the infection

 Adverse effects of septra 20Xs more common in AIDS patients: N/V, rash,

neutropenia, thrombocytopenia, hyponatremia, hepatitis

 PCP prophylaxis: recommended when CD4 200 and patient

is alert with no focal findings but most will do CT first anyways

 See Figure 126-1

 CT typical for toxo: treat and no LP

 CT with lesions but not typical for toxo: LP, may need stereotactic bx

 CT with no lesion: LP

 Note: CT head without contrast may miss subtle lesions (go on to CT with

contrast or MRI)

 Lumbar puncture: DO NOT forget to send cytology (CNS lymphoma),

cryptococcal antigen latex agglutination, coccidioidomyocosis titer,

viral/fungal/TB cultures (phone lab and ask how much volume needed for

these tests), opening pressure can help





 HIV Encephalopathy

 AIDS dementia complex

 Occurs in 1/3

 Progressive process cuased by direct HIV infection

 Initial presentation with loss of recent memeory, subtle cognitive defects

 Early presentation may be confused with depression, anxiety, etc

 Later: frank mental status changes, seizure, frontal release, incr. DTRs

 Must r/o other CNS pathology

 Neuroimaging shows atrophy and diffuse deep mater hyperintensities

 LP is typically norma;

 Cryptococcus neoformans

 10% of AIDS patients

 Focal cerebral lesions or diffuse meningoencephalitis

 Fever, headache, nausea, vomiting is the MC initial symptoms

 Can present with visual changes, seizures, focal deficits

 Dx = CSF identification

 Cryptococcal antigen latex agglutination is nearly 100% sensitive and specific

(India ink staining is 70% sensitive, fungal culture 90%, serum cryptococcal

antigen 90%)

 Treatment: amphotericin B +/- flucytosine X 10 weeks (high relapse rate)

 Chronic suppression therapy with fluconazole after initial treatment

 Bone marrow suppression is a problem with treatment regimen

 Toxoplasma gondii

 MC focal encephalitis in AIDS patient

 Headache, fever, altered LOC, seizures, focal deficits in up to 80%

 PEARL: HIV/AIDS and looks like a stroke; infection is acutally more

common

 Serology useless as most people have been exposed to it

 CSF antibody is helpful but there are many false +ves

 Dx: MULTIPLE SUBCORTICAL LESIONS on CT head

 May not be seen on non-contrast CT head; repeat if highly suspicious (or

MR)

 Brain lesion ddx: toxo, lymphoma, TB, fungi, progressive multifocal

leukoencephalopathy, CMV, KS, hemorrhage

 Suggestive of toxo: multiple lesion, predominace for BG and corticomedullary

area

 Lymphoma: usually single slesion in periverntricular matter or corpus

callosum

 Treatment: Pyrimethamine po + sulfadiazine po + folinic acid to reduce the

incidence of pancytopenia

 Dexamethasone for significant brain edema

 Dilantin for seizure prophylaxix for significan edema

 Chronic suppression therapy with pyrimethamine, sulfadiazine, and folinic

acid

 Prophylaxis: indicated for +ve seriology and CD4 5000

but significant variation exists (some treat all with measurable viral loads)

 Drug Regimens

 No universal concensus

 First line regimen: two NRTIs + one PI or two NRTIs + one NNRTI

 Prophylaxis mostly based on the CD4 count which is the best predictor of

opportunistic infections

 Prophylaxis: PCP, TB, toxo, MAC, others

 Immunizations

 NO LIVE VIRUS or BACTERIA vaccines

 Pneumococcal vaccination for all > 2yo

 Influenza generally recommended

 HBV immunization if at risk

 HAV immunization b/c of suceptibility to liver insult

 Td booster: some recent evidence of increased viral expression following

administration of Td but clinical significance unknown





 Disposition

 Admission

- FUO

- Hypoxemia

- PCP pneumonia

- TB

- New CNS symptoms

- Intractable diarrhea

- Suicidal

- CMV retinitis

- Zoster involving the eye

- Unable to care for self

- No follow up

 Discharge

- Normal vitals

- Stable condition

- Able to take meds and self care

- Good follow-up

- Caregivers to help



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