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Vibrio cholerae in Calcutta

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Archives of Disease in Childhood 1994; 71: 161-162 161





Vibrio cholerae 0139 in Calcutta

S K Bhattacharya, M K Bhattacharya, D Dutta, S Garg, A K Mukhopadhyay, M Deb,

A Moitra, G B Nair





Abstract Table 1 Clinicalfeatures of 28 V cholerae 0139 cases

Vibrio cholerae 0139 was recovered

from 28 of 79 children with acute watery No (%)

diarrhoea. Clinically, they presented with Median (range) age in months 48 (2-96)

watery diarrhoea (100%), vomiting (79%), 95% CI 36 to 66

Male:female 13:15

abdominal cramps (61%), anorexia (61%), Watery stools 28 (100)

dehydration (100%)/) and absence of fever. Median stool frequency/day 8

95%/CI 5to 10

Both clinical and blood biochemical para- Vomiting 22 (79)

meters of these cases were similar to the Median preadmission duration of diarrhoea (hours) 9

95%CI 7to12

illness caused by the new strain in adults. Abdominal cramps 17 (61)

Hypoglycaemia was seen in 40% of those Fever (>38°C) 0 (0)

Anorexia 17 (61)

screened. Dehydration:

(Arch Dis Child 1994; 71: 161-162) None 0

Mild to moderate 10 (36)

Severe 18 (64)



The aetiological agent of several cholera-like CI=confidence interval.

outbreaks in the Indian subcontinentl 2 has

been identified as Vibrio cholerae that does

not agglutinate with 0 1 antiserum and Before any antibiotic treatment a faecal

hence was categorised as non-0 1. Serological sample was collected from each child using a

studies have established the clonal origin of sterile rectal catheter into a sterile MaCartney

the V cholerae non-0 1 strains from diverse bottle and immediately sent to the laboratory.

geographical areas and a new serogroup 0139 V cholerae was isolated following a standard

with the synonym Bengal was created to procedure and serogrouped using the slide

accommodate these strains. V cholerae 0139 agglutination technique as described previ-

Bengal, much like the 01 serogroup, produces ously. Production of cholera toxin was

large amount of cholera toxin which is unusual determined by a highly sensitive bead-enzyme

for a non-0 1 serovar. During a large epidemic linked immunosorbent assay (ELISA).' A

that occurred in Calcutta, 84% of the cases blood sample was also obtained on admission

were adults and the clinical profile of the cases for estimation of concentrations of sodium,

infected with the new epidemic strain led us potassium, chloride, total carbon dioxide,

to designate the disease as cholera.3 While urea, and glucose from a representative

cholera has been thought not to be a problem number of cases.

in children below 2 years of age, we recently

documented that as many as 31% of

Results

hospitalised children with watery diarrhoea in

an endemic area had cholera.4 During A total of 79 cases of acute watery diarrhoea

September to November 1993 we isolated were screened of which 28 cases were bacterio-

V cholerae 0139 from 28 cases in children logically and serologically confirmed for

and the present report deals with the clinicalV cholerae 0139 and this group constituted

presentation of 0139 cholera in children in the study population. All the 28 strains of

Calcutta. V cholerae 0139 recovered from the same

National Institute of number of cases were found to produce

Cholera and Enteric cholera toxin by bead-ELISA. The children

Diseases, Calcutta, Subjects and methods had watery stools (100%)/, vomiting (79%),

India

S K Bhattacharya Children below the age of 12 years suffering abdominal cramps (61%)/, anorexia (61%),

M K Bhattacharya from acute diarrhoea with no history of dehydration (100%), and absence of fever

D Dutta prior antibiotic treatment, admitted to the (table 1).

S Garg

A K Mukhopadhyay Infectious Diseases Hospital, Calcutta, The results of estimation of serum elec-

M Deb between 0600 and 1300 hours, during the trolytes in 13 cases and the same of blood

G B Nair period September to November 1993, were urea and glucose in the peripheral blood of 10

Infectious Diseases selected and included in the study. Upon

Hospital, Calcutta, admission, history was obtained from the Table 2 Biochemistry results of V cholerae 0139 cases

India patients or their accompanying mothers or (serum/blood)

A Moitra other relatives, followed by a thorough

Correspondence to: physical examination, and the findings Mean (SD) Normal range

Dr S K Bhattacharya,

National Institute of Cholera were recorded in a specially designed form. Sodium (mmol/l) 140-8 (5 3) 138-145

and Enteric Diseases, P-33, The children were assessed and treated Potassium(mmol/l)

Chloride

(mmol/l) 4-7 (1-1)

106-3 (4-2)

3-4-4-7

98-106

CIT Road, Scheme XM,

Beliaghata, Calcutta 700010, according to World Health Organisation Total carbon dioxide (mmol/A) 14-6 (5 3) 20-28

India. (WHO) guidelines for management of Urea (mmolIl)

Glucose (mmol/l)

6-2 (4-1)

4-4 (1 9)

1-8-6-4

3-3-5-5

Accepted 16 May 1994 cholera.

Downloaded from adc.bmj.com on December 24, 2011 - Published by group.bmj.com





162 Bhattacharya, Bhattacharya, Dutta, Garg, Mukhopadhyay, Deb, Moitra, Nair



cases are shown in table 2. Four patients had feature of cholera-like illness caused by the

hypoglycaemia. non-0 1 serogroups of V cholerae and of cholera

All the children studied were successfully caused by V cholerae 0139 in adults as

rehydrated with oral (oral rehydration salt previously documented by us.3 It is, therefore,

solution using the WHO formula) and/or suggested that the differential diagnosis of

intravenous fluid and electrolyte (Ringer's children presenting with cholera-like illness

lactate) solution. Eighteen children (64%) were should include cholera caused by V cholerae

given intravenous fluids. None of the cases died. 0139. Further, presence of abdominal cramps

The average hospital stay was 40 hours. should not exclude cholera caused by the new

serogroup. Hypoglycaemia, a well recognised

complication of acute diarrhoea including

Discussion cholera in children,6 was seen in 40°/ of cases

Since September 1993, when the 0139 cholera screened.

epidemic was waning, we isolated

BT/TF/9/63/9

V cholerae 0139 from 28 cases of 79 children This work was partly supported by grants (NoDelhi, India. 1)

from the Department of Biotechnology, New

screened. Lindenbaum et al reported that

cholera in children is similar to that in adults.5

The clinical features and biochemical changes 1 Ramamurthy of Vibrio cholerae with epidemic potential of

novel strain

T, Garg S, Sharma R, et al. Emergence

in

in the blood of children infected with Vcholerae southem and eastern India. Lancet 1993; 341: 703-4.

2 Albert MJ, Ansaruzzaman M, Bardhan PK, et al. Large

0139 were similar to that already reported by epidemic of cholera-like disease in Bangladesh caused by

us for adults infected by the same serogroup3 Vibrio cholerae 0139 synonym Bengal. Lancet 1993; 342:

387-90.

and were indistinguishable from cholera caused 3 Bhattacharya SK, Bhattacharya MK, Nair GB, et al.

by V cholerae belonging to the 01 serogroup. A Clinical profile of acute diarrhoea cases infected with the

new epidemic strain of Vibrio cholerae 0139: designation

notable exception, however, was the presence of the disease as cholera. Jl Infect 1993; 27: 11-5.

of abdominal pain in 61% of the cases which 4 Bhattacharya SK, Bhattacharya MK, Ramamurthy T, et al.

was higher than that observed in 0139 cholera Cholera in young children in an endemic area. Lancet

1992; 340: 1549.

in adults (440/o).3 Abdominal cramps observed 5 Lindenbaum J, Gordon RS Jr, Hirschhron N, Akbar R,

in these cases could not be attributed to Greenough WB III, Islam MR. Cholera in children. Lancet

1966; i: 1066-8.

hypokalaemia because even after correction 6 Bennish ML, during diarrhoea in childhood: prevalence,

Hypoglycemia

Azad AK, Rahman 0, Phillips RE.

of electrolyte imbalances abdominal cramps pathophysiology, and outcome. N Engl J7 Med 1990; 322:

persisted. Abdominal cramp is not an unusual 1357-63.

Downloaded from adc.bmj.com on December 24, 2011 - Published by group.bmj.com









Vibrio cholerae O139 in Calcutta.

S K Bhattacharya, M K Bhattacharya, D Dutta, et al.



Arch Dis Child 1994 71: 161-162

doi: 10.1136/adc.71.2.161





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