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Applied Sciences Lecture Course





Diuretics



Dr Cathy Armstrong

SpR In Anaesthesia & Clinical Fellow in Undergraduate

Medical Education

Manchester Royal Infirmary





March 2011

Aims & Objectives

• Define the term diuretic



• Classify diuretics into groups

– Describe mechanisms of action

– Describe relevant pharmacokinetic aspects





• Discuss common clinical uses

What is a diuretic?

What is a diuretic?







An agent that promotes excretion

of urine

In the glomerulus where is the

majority of sodium reabsorbed?

1. Proximal 25% 25% 25% 25%

convoluted tubule

2. Loop of Henle

3. Distal convoluted

tubule

4. Collecting duct







1 2 3 4

Cvpharmacology.com

Which of these is NOT a class of

diuretic?

1. Loop diuretic 20% 20% 20% 20% 20%



2. Carbonic anhydrase

inhibitor

3. Sodium-sparing

diuretic

4. Potassium-sparing

diuretic

5. Thiazide diuretic

1 2 3 4 5

Cvpharmacology.com

Thiazide diuretics

• Main action on distal convoluted tubule

– Bendroflumethazide

– hydrochlorothiazide

• Inhibit sodium & chloride reabsorption









www.mc.uky.edu

Thiazide diuretics

• Maximal effects produced by small doses

• Duration of action 12-18 hours

• Side effect profile

– Hypokalaemia

• Particular caution with digoxin

– Increased uric acid levels

– Impaired glucose tolerance

– Increased cholesterol levels

Cvpharmacology.com

What is the commonest clinical use

of thiazide diuretics?

1. Heart failure 25% 25% 25% 25%

2. Hypertension

3. Cerebral oedema

4. Renal failure









1 2 3 4

Hypertension



• Thiazide diuretics



– ↓ systolic BP by 10-15 mmHg



– ↓ diastolic BP by 5-10 mmHg

Hypertension



MAP = CO x SVR

~ 1

r4

CO = HR x SV



Preload Contractility Afterload

Hypertension



MAP = CO x SVR

~ 1

r4

CO = HR x SV



Preload Contractility Afterload

Hypertension



MAP = CO x SVR

~ 1

r4

CO = HR x SV



Preload Contractility Afterload

Hypertension – NICE guidance









A = ACE inhibitor, C = calcium channel blocker, D = thiazide

diuretic

Loop diuretics

• Act on thick ascending limb of loop of Henle

• Sodium-potassium-chloride cotransporter

– Handles 25% of sodium reabsorption









www.mc.uky.edu

Loop diuretics

• Induce renal synthesis of prostaglandins

– ↑ renal blood flow

– Redistribution of renal cortical blood flow





• Quick onset, shorter duration of action

Loop diuretics

• Side effects

– Hypokalaemia

– Hyperglycaemia & hyperuricaemia

– Transient damage to CN VIII (esp with gentamycin)

• Uses

– Pulmonary oedema (Acute LVF)

– Chronic heart failure

– Hypertension

– Peripheral oedema

Cvpharmacology.com

Pulmonary oedema

• Presentation

– Dyspnoea

– Hypoxia

– Sweating

– Tachycardia (gallop rhythm)

– Elevated JVP

– Pink frothy sputum

– Crepitations on auscultation of lung fields

Pulmonary oedema

• Immediate management

– Oxygen

– Sit patient up

– IV access

– Furosemide (40 – 80mg IV)

– Diamorphine

– GTN infusion (if BP will tolerate)

– Consider CPAP

Pulmonary oedema









Cvpharmacology.com

Chronic Heart Failure – NICE

Guidance

Potasium sparing diuretics

• Block sodium channels distal end of DCT









www.mc.uky.edu

Potasium sparing diuretics

• Weak diuretics

• Examples

– Amiloride, Triamterene

• Often co-administered to reduce

hypokalaemic effects of loop / thiazides

– Co-amilofruse

– Co-triamterzide

• Side effects

– hyperkalaemia

Aldosterone Antagonists

• Spironolactone



• Aldosterone enhances expression of sodium

channel in distal end of DCT

Cvpharmacology.com

Aldosterone Antagonists

• Uses

– Oedema & ascites in cirrhosis of the liver

– Chronic heart failure

– Primary hyperaldosteronism

• Conn’s syndrome

• side effects

– Hyperkalaemia

– N&V

– gynaecomastia

Carbonic anhydrase inhibitors

• Prevent reabsorption of bicarbonate in proximal

convoluted tubule









www.mc.uky.edu

Carbonic anhydrase inhibitors

• Weak diuretics

• Not used for diuretic properties



• Uses

– Prophylaxis against altitude sickness

– glaucoma

Osmotic diuretics

• Mannitol

– Draws water from the extracellular & intracellular

spaces into the vascular compartment

– Easily filtered but not reabsorbed

– Continues to be osmotically active in tubules

causing diuresis

• Main use

– Cerebral oedema

Diuretics in acute renal failure?

Summary









Cvpharmacology.com

?



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