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Protein Structure

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Chapter 5: Outline

Amino Acids

Amino acid classes Stereoisomers

Bioactive AA Titration of AA

Modified AA AA reactions

Peptides

Proteins (We are here)

Protein structure

Fibrous proteins

Globular proteins

5P2-1

Protein Function

1. Catalysis

2. Structure

3. Movement

4. Defense

5. Regulation

6. Transport

7. Storage

8. Stress Response



5P2-2

Proteins by Shape-1

Fibrous proteins exist as long stranded molecules:

Eg. Silk, collagen, wool. A collagen segment in

space-filling mode illustrates this point.









Red spheres represent oxygen,

grey carbon, and blue nitrogen

5P2-3

Proteins by Shape-2

Globular proteins

have somewhat

spherical shapes.

Most enzymes are

globular. Eg.

myoglobin,

hemoglobin.

Myoglobin in

space-filling mode

is the chosen

example.

5P2-4

Proteins by Composition

Simple

Contain only amino acids

Conjugated

simple protein (apoprotein)

prostetic group (nonprotein)

glycoproteins Holo-

lipoproteins protein

metaloproteins

etc.

5P2-5

Four Levels of Protein Structure

Primary, 1o

the amino acid sequence

Secondary, 2o

3-D arrangement of backbone atoms in

space

Tertiary, 3o

3-D arrangement of all the atoms in

space

Quaternary, 4o

3-D arrangement of subunit chains

5P2-6

Determining Primary Structure

1. Hydrolyze protein with hot 6M HCl.

Identify AA and % of each.

Usually done by chromatography

2. Identify the N-term and C-term AAs

C-term via carboxypeptidase

N-term via Sanger’s Reagent, DNFB

2,4-dinitrofluorobenzene

Often step 2 can be skipped today.



5P2-7

Det. Primary Structure: 2

3. Selectively fragment large proteins

into smaller ones.

Eg. Tripsin: cleave to leave Arg or Lys

as C-term AA

Eg. Chymotrypsin: cleave to leave Tyr

or Trp or Phe as C-term AA

Eg. Cyanogen bromide cleaves at

internal Met leaving Met as C-term

homoserine lactone

5P2-8

Det. Primary Structure: 3

4. Determine AA sequence of peptides

with AA sequencer using Edman’s

reagent:

phenyl isothiocyanate which reacts

with the N-term AA

See the next slide









5P2-9

Det. Primary Structure: 3b

+ -

H3N 1 2 3 COO

protein

N C S Edman’s reagent

S

-

NH C NH 1 2 3 COO



N

Thiazoline derivative

NH C CH R1 + -

+ H3N 2 3 COO

C

S O

aqueous acid

O RAR

C

N

CH R1 Phenylthiohydantoin (PTH)

C NH derivative of N-term AA

S 5P2-10

Det. Primary Structure: 4

5. Reassemble peptide fragments from

step 3 to give protein.

An example follows on the next slide.









5P2-11

Det. Primary Structure: 4b

A twelve AA peptide was hydrolyzed.

Trypsin hydrolysis:

Leu-Ser-Tyr-Gly-Ile-Arg

One is

Thr-Ala-Met-Phe-Val-Lys C-term

Chymotrypsin hydrolysis

Val-Lys-Leu-Ser-Tyr Lys is internal!

Gly-Ile-Arg

Thr-Ala-Met-Phe

Deduce the AA sequence

5P2-12

Det. Primary Structure: 4c

Keeping in mind the N-term AA and overlaping the

sequences properly gives:

Tr Leu-Ser-Tyr-Gly-Ile-Arg

Ct Gly-Ile-Arg

Ct Val-Lys-Leu-Ser-Tyr

Tr Thr-Ala-Met-Phe-Val-Lys

Ct Thr-Ala-Met-Phe

The complete sequence is:

Thr-Ala-Met-Phe-Val-Lys-Leu-Ser-Tyr-Gly-Ile-Arg





5P2-13

Secondary Structure

The two very important secondary

structures of proteins are:

a-helix

b-pleated sheet

Both depend on hydrogen bonding

between the amide H and the

carbonyl O further down the chain

or on a parallel chain.

5P2-14

a Helix: Peptide w Hbonds

First six C=O to N hydrogen bonds shown









5P2-15

b Sheet: stick form Protein G

H bonds in dotted red-blue

H bonds shown in dotted red-blue

Chain

segment 1







Seg 2





Seg 3

Chain 1

Seg 4



5P2-16

B Sheet: Lewis Structure

N-term C-term N-term N-term

CH HC CH CH

C O H N C O C O

H N C O H N H N

HC CH HC HC

O C N H O C O C

N H O C N H N H

CH HC CH CH

C O H N C O C O

H N C O H N H N

C-term N-term C-term C-term

Antiparallel sheet Parallel sheet

5P2-17

Supersecondary Structure

Reverse turns in a protein chain allow

helices and sheets to align side-by-side

Common AA found at turns are:

glycine: small size allows a turn

proline: geometry favors a turn









5P2-18

Supersecondary Structure: 2

Combinations of a helix and b sheet.









bab aa b meander

5P2-19

Tertiary Structure

The configuration of all the atoms in the

protein chain:

side chains

prosthetic groups

helical and pleated sheet regions









5P2-20

Tertiary Structure: 2

Protein folding attractions:

1. Noncovalent forces

a. Inter and intrachain H bonding

b. Hydrophobic interactions

c. Electrostatic attractions

+ to - ionic attraction

d. Complexation with metal ions

e. Ion-dipole

2. Covalent disulfide bridges 5P2-21

Tertiary interactions: diag.

disulfide Polypeptide Chain



+

NH3 O CH2 S CH3 HO CH2

O C S CH2 CH3 CH2 OH

metal coord’n

+

H3C CH CH3 NH3 O

Mg2+ O O

O H C H3C CH CH3 O C





ionic H bonds

Ion-dipole hydrophobic or dipole

5P2-22

Domains

Domains are common structural units

within the protein that bind an ion or

small molecule.









5P2-23

Quaternary Structure-1

Quaternary structure is the result of

noncovalent interactions between two

or more protein chains.

Oligomers are multisubunit proteins with

all or some identical subunits.

The subunits are called protomers.

two subunits are called dimers

four subunits are called tetramers



5P2-24

Quaternary Structure-2

If a change in structure on one chain

causes changes in structure at another

site, the protein is said to be allosteric.

Many enzymes exhibit allosteric control

features.

Hemoglobin is a classic example of an

allosteric protein.







5P2-25

Denaturation

-loss of protein structure, 2 o 4 o, but

not 1o.

1. Strong acid or base

2. Organic solvents

3. Detergents

4. Reducing agents

5. Salt concentrations

6. Heavy metal ions

7. Temperature changes

8. Mechanical stress

5P2-26

Denaturation-2

Denaturing destroys the physiological

function of the protein.

Function may be restored if the correct

conditions for the protein function are

restored.

But! Cooling a hardboiled egg does not

restore protein function!!







5P2-27

Fibrous Proteins

Fibrous proteins have a high

concentration of a-helix or b-sheet.

Most are structural proteins.

Examples include:

a-keratin

collagen

silk fibroin







5P2-28

Globular Proteins

Usually bind substrates within a

hydrophobic cleft in the structure.

Myoglobin and hemoglobin are typical

examples of globular proteins.

Both are hemoproteins and each is

involved in oxygen metabolism.









5P2-29

Myoglobin: 2 o and 3 o aspects

Globular myoglobin has 153 AA arranged in

eight a-helical regions labeled A-H.

The prosthetic heme group is necessary for

its function, oxygen storage in mammalian

muscle tissue.

His E7 and F8 are important for locating the

heme group within the protein and for binding

oxygen.

A representation of myoglobin follows with

the helical regions shown as ribbons.



5P2-30

Myoglobin: 2o and 3o aspects



Some helical regions









Heme group with iron (orange)

at the center

5P2-31

The Heme Group

- -

OOC CH2CH2 CH2CH2COO

N of His

F8 binds

to H3C CH3

fifth site on N N

the iron. Fe(II)

H2C Pyrrole ring

N N

CH CH3

His E7 acts

as a ”gate”

CH3 CH CH2

for oxygen. 5P2-32

Binding Site for Heme

Lower His bonds covalently

to iron(II) N

N

Oxygen coordinates to H

sixth site on iron and the O

upper His acts as a “gate” O

for the oxygen. N N

Fe

N N

N

N

5P2-33

Hemoglobin

A tetrameric protein

two a-chains (141 AA)

two b-chains (146 AA)

four heme units, one in each chain

Oxygen binds to heme in hemoglobin

cooperatively: as one O2 is bound, it

becomes easier for the next to bind.

Lengthy segments of the a and b chains

homologous to myoglobin.

5P2-34

Hemoglobin: ribbons + hemes

Each chain

is in ribbon

form and

color coded.

The heme

groups are

in space

filling form









5P2-35

Oxygen Binding Curves

Oxygen bonds differently to hemoglobin

and myoglobin.

Myoglobin shows normal behavior while

hemoglobinn shows cooperative

behavior. Each oxygen added to a

heme makes additon of the next one

easier.

The myoglobin curve is hyperbolic.

The hemoglobin curve is sigmoidal.

5P2-36

Oxygen Binding Curves-2









5P2-37

The Bohr Effect (H+ and Hb)

Lungs:

pH higher than in actively metabolizing

tissue. (Low H+). Hb binds oxygen and

releases H +.



Muscle at Work:

pH lower (H + product of metabolism). Hb

releases oxygen and binds H+.



metabolism

HbO2 + H+ + CO2 O2 + H+-Hb-CO2

in lungs

5P2-38



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