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The Role of Phosphorylation in Mediating Cellular Responses

to DNA Damage: The ATM-Mediated DNA Damage Response

Professor Yosef Shiloh



The Role of Phosphorylation in Mediating

Cellular Responses to DNA Damage:

The ATM-Mediated DNA Damage Response









Professor Yosef Shiloh

Sackler School of Medicine

Tel Aviv University, Israel

1









The DNA molecule









2









DNA damage









3

The Role of Phosphorylation in Mediating Cellular Responses

to DNA Damage: The ATM-Mediated DNA Damage Response

Professor Yosef Shiloh

Estimated endogenous DNA damage in mammalian cells

Damage Events per cell/day

Single-strand break 55,200

Depurination 12,000

Depyrimidination 600

06-methylguanine 3,120

Cytosine deamination 192

Glucose-6-phosphate adduct 3

Thymine glycol 270

Thymidine glycol 70

Hydroxymethyluracil 620

8-oxo-G 178

Interstrand cross-link 8

Double-strand break (DSB) 8



Compiled by Dr. Vilhelm Bohr 4









DSB repair pathways









5

Nonhomologous Homologous recombination

end-joining (NHEJ) (HR)









The DNA damage response

DNA repair

Cell cycle checkpoints

Affects numerous

cellular processes

Modulates gene expression

Concerted action of numerous,

interlocked signaling pathways

creating an intricate web

Activated instantaneously

and vigorously,

particularly by DSBs



Pericentriolar material

Mother centriole





6

Daughter centriole

Replicated DNA

Chromosome

The Role of Phosphorylation in Mediating Cellular Responses

to DNA Damage: The ATM-Mediated DNA Damage Response

Professor Yosef Shiloh

Cellular responses to DNA damage

DNA Endogenous Exogenous

turnover agents agents



DNA

damage



Sensors /Activators



Transducers

activation,

signalling

Effectors









DNA Cell cycle Stress

repair arrest responses

Apoptosis

7









үH2AX foci үH2AX foci



Foci show where

the damage occurred

This is a good marker

for the induction

of double-strand breaks

and their disappearance

Sophisticated techniques

can be used to induce the DNA

damage in specific portions

of the nucleus



Many damage response proteins

are rapidly recruited to the damage sites





8









P P P P

P P

P ATM ATM P

RAP80BRCC36 RAP80BRCC36

P

NBS1 NBS1 P

UBC13

P

MDC1

P P P P RNF8

P

P BRCA1 P P

P P MDC1 P BRCA1 P P

P P

P

P ATM P P P

RNF20

P P ATM P P

RNF20

P 53BP1 53BP1 RNF40 P

RNF40 P



Ub Ub Ub Ub Ub Ub Ub

Ub Ub Ub Ub

Ub Ub Ub Ub

Ub Ub Ub Ub

Ub Ub Ub Ub

Ub Ub Ub Ub









9

The Role of Phosphorylation in Mediating Cellular Responses

to DNA Damage: The ATM-Mediated DNA Damage Response

Professor Yosef Shiloh

Defects in maintenance of genome stability

Monogenic disorders combining sensitivity

to genotoxic stresses, tissue degeneration,

cancer predisposition

Chromosomal and genomic aberrations

The DNA damage response as a barrier

to cancer development

Bloom syndrome

Milder mutations or heterozygosity for severe

Fanconi’s anemia

mutations may contribute to chronic conditions:

Sensitivity to acute doses of genotoxic stress

Premature aging

Immunodeficiency

Neurodegeneration

Nijmegen breakage Cancer predisposition Xeroderma pigmentosum

syndrome

Reduced ability to cope with environmental stresses

Load on public health caused by mutations

affecting genomic stability may be heavier

than previously estimated



10

Disorders flag important players Ataxia-telangiectasia



Werner syndrome

in the DNA damage response









Ataxia-telangiectasia (A-T)

1926: Syllaba and Henner described 3 siblings

with progressive ataxia and ocular telangiectasias

1941: Louis-Bar described a Belgian girl

with a similar syndrome

1957: Sedgwick and Boder reported a series

of American patients and called the disease

ataxia-telangiectasia (A-T)









11









Ataxia-telangiectasia (A-T) (2)

Autosomal recessive

Cerebellar ataxia (loss of Purkinje cells),

severe neuromotor dysfunction

Telangiectasia

Immunodeficiency (B&T cells);

recurrent infections

Thymic and gonadal dysgenesis

Retarded growth

Chromosomal instability

Cancer predisposition

Acute sensitivity to ionizing radiation

A profound defect in cellular responses

to double strand breaks (DSBs) in the DNA

Responsible gene: ATM



12

The Role of Phosphorylation in Mediating Cellular Responses

to DNA Damage: The ATM-Mediated DNA Damage Response

Professor Yosef Shiloh

Cellular responses to DNA damage

DNA Endogenous Exogenous

turnover agents agents



DNA

damage



ATM Sensors /Activators



Transducers

activation,

signalling

Effectors









DNA Cell cycle Stress

13

Apoptosis

repair arrest responses









The ATM protein



Completely missing or inactivated in A-T

patients (reduced levels in “mild A-T”)

A large (370 kDa), nuclear phosphoprotein

A carboxy-terminal PI3-kinase-like motif

Protein kinase activity (serine-threonine)

ATM targets: preference for SQ or TQ

The primary activator of the cellular response

to double strand breaks (DSBs)

Activated by DSBs

Phosphorylates key

DDR players

14









ATM activation



DSBs

Inactive dimers turn

into active monomers;

ATM ATM autophosphorylation

Recruitment (Bakkenist & Kastan, 2003)



to DSB sites Several autophosphorylations,

(Andegeko et al., 2001) Tip60-mediated acetylation

Activated (Kozlov et al., 2006, Sun et al., 2007)

ATM

Role for autophosphorylations

Phosphorylation in ATM retention at DSB sites

(So et al., 2009)

Additional post-translational Effectors

modifications?





DSB Cell cycle Stress

15

Apoptosis

repair checkpoints responses

The Role of Phosphorylation in Mediating Cellular Responses

to DNA Damage: The ATM-Mediated DNA Damage Response

Professor Yosef Shiloh



ATM-mediated DNA damage responses

There are two levels

of phosphorylations









16









PI3-kinase-related protein kinases (PIKKs)

1 3056

ATM



1 2644

ATR



1 3031



hSMG-1

1 4128

DNA-PKcs

1 2549



mTOR/FRAP

1 3830



TRRAP



DOMAINS: FAT PI3K FATC 17









p53 activation and stabilization



ATM



S395 26S

Additional T68

modifications

Mdm2

Chk2

S342

S403S367 ATM has a very strong grip

S15 S20 Mdmx HAUSP of this pathway

S387 p53 T42 S337

COP1 ATM can fine tune this pathway

USP10

S342

*p53 *p53 S403S367



*p53 *p53 *p53 Mdmx 14-3-3

26S

HAUSP

poly-Ub

26S

Apoptosis Cell cycle

checkpoints







18

R.I.P

The Role of Phosphorylation in Mediating Cellular Responses

to DNA Damage: The ATM-Mediated DNA Damage Response

Professor Yosef Shiloh



Research directions

ATM

ATM activation

Identification of novel ATM



ATM-mediated pathways

Function of target proteins:

Cellular level

Organismal level Other Protein Protein Gene Cell DNA

stress degradationsynthesis regulation cycle repair

Overview of the DNA response checkpoints



damage response using

systems biology approach

Search for new treatment

modalities for A-T





19









New branches of the ATM-mediated

DNA damage response

Substrate identification

Proteomic methods:

ATM

• ATM-associated proteins

• Proteomic screens for ATM substrates

• Damage-induced dynamics

of the cellular phosphoproteome

Candidate approach

Functional analysis of specific substrates and the corresponding pathways

Function of a single protein, single phosphorylation,

in a complex signaling network

Identification of the “proteomic environment”

surrounding the substrate in undamaged

and damaged cells

“Protein replacement” followed

by analysis of cellular phenotype

Gene targeting in mice



20









ATM functional links: a SPIKE map, September 2010

http://www.cs.tau.ac.il/~spike/









ATM









21

The Role of Phosphorylation in Mediating Cellular Responses

to DNA Damage: The ATM-Mediated DNA Damage Response

Professor Yosef Shiloh

High-throughput identification of ATM/ATR/DNA-PK substrates









Most of these

phosphorylations

are real and play

a role in the DNA

damage response 22









Insights into the ATM-mediated DDR



Control of specific processes via many

ATM-mediated pathways

Recruitment to damage sites of proteins that function

in damage processing/signaling, in their “regular”

or different capacities

Broad interface between the DDR and processes driven

by the ubiquitin family of proteins

Intimate relationships with telomere maintenance, meiotic

and V(D)J recombination and RNA metabolizing systems







23









Understanding A-T









Defective repair of DNA double strand breaks

24

The Role of Phosphorylation in Mediating Cellular Responses

to DNA Damage: The ATM-Mediated DNA Damage Response

Professor Yosef Shiloh



ATM-mediated signaling facilitating DSB repair



Direct targeting of repair proteins

Creating appropriate microenvironment for repair

within chromatin









25









PNKP – polynucleotide kinase 3’-phosphatase



Dual activity enables

restoration of “legitimate” ends









26









ATM-mediated phosphorylation of PNKP



FHA Phosphatase Kinase

5 114126 521



Segal-Raz et al., 2011

Targets: S114 and S126

Induced by DSB-inducing agents

ATM-dependent

Cells in which endogenous

Phosphatase assay

PNKP is replaced by ectopic, 0.6

0.5

non-phosphorylatable PNKP

% Activity









0.4 wt

show intermediate 0.3 x2

radiosensitivity between 0.2



those of PNKP-/- and wild 0.1

0.0



27

type cells 0 15 30 60

Time (min)

The Role of Phosphorylation in Mediating Cellular Responses

to DNA Damage: The ATM-Mediated DNA Damage Response

Professor Yosef Shiloh









Allowing repair to happen within chromatin









28









ATM-dependent phosphorylation of KAP-1 mediates

global chromatin relaxation following DNA damage



B B Coiled- HP1BD

RING box box PHD Bromo

coil





Ziv et al., 2006

S824

Known as transcription corepressor

Rapidly phosphorylated by ATM at DSB sites

Quickly migrates throughout chromatin

and mediates chromatin decondensation

Facilitation of DSB repair









29









Chromatin reorganization

Prerequisite for all DNA transactions

Major mediator: changes in histone

post-translational modifications

Examples for histone PTMs associated

with the DDR:

H2AX phosphorylation/dephosphorylation

H2A monoubiquitination mediated by RNF8 and RNF168

Monoubiquitination of histone H2B

– an ongoing, dynamic PTM









30

The Role of Phosphorylation in Mediating Cellular Responses

to DNA Damage: The ATM-Mediated DNA Damage Response

Professor Yosef Shiloh



RNF20/40- and ATM-mediated H2B monoubiquitination

Upon DSB induction RNF20/40 is recruited Moyal et al., 2011

P P

to damaged sites and phosphorylated by ATM RNF

20

H2B monoubiquitination at damaged ATM RNF

sites is important for timely recruitment 40

P

of both NHEJ and HR repair proteins

and subsequent DSB repair NHEJ



Formation of appropriate chromatin

microenvironment for DNA repair

Example of a protein machinery that is normally

active in a non-DDR context and is recruited

HR

to the damaged sites to carry out the same

function at the damaged sites







31









Exploration of the interface between the ubiquitin

family and the DNA damage response









32









Players in ubiquitin-related processes









33

~1600 proteins

The Role of Phosphorylation in Mediating Cellular Responses

to DNA Damage: The ATM-Mediated DNA Damage Response

Professor Yosef Shiloh



Overlap between the ubiquitin arena

and ATM substrates



Ub arena ATM substrates









1600 145 900



34









Phosphoproteome dynamics after DNA damage

Untreated DNA damage

A. Bensimon et al., 2010





Prepare nuclei





Lyse nuclei, digest p

p p

p

p

p

p

p



Phosphopeptide p p

p p

enrichment p

p

p

p









LC-MS/MS







Label-free

quantification Computational



35

analysis









Damage-induced protein phosphorylation

Hundreds of proteins phosphorylated

or dephosphorylated in response to DSB induction

Many new phosphorylations, not on SQ/TQ

Only 60% of the phosphorylations are ATM-dependent

ATM “switch off” after damage induction switches off

many ATM-dependent phosphorylations -> ATM

sustained activity is required to maintain them







No ATM inhibitor



With ATM inhibitor



36

The Role of Phosphorylation in Mediating Cellular Responses

to DNA Damage: The ATM-Mediated DNA Damage Response

Professor Yosef Shiloh

Functional networks of phosphorylated

and dephosphorylated proteins









Phosphorylated

Dephosphorylated





37

Both









Ub ATM









1600 145 900

88 63



299





“General”

phosphorylation

38









Functional screens for novel DDR players



Screening of siRNA

libraries using assays

based on DDR readouts









Nuclear foci of 53BP1







KAP-1 phosphorylation







39

The Role of Phosphorylation in Mediating Cellular Responses

to DNA Damage: The ATM-Mediated DNA Damage Response

Professor Yosef Shiloh



Ub ATM









1600

186 15 900

8



299





“General”

phosphorylation

40









The role of phosphorylation in mediating

cellular responses to DNA damage:

the ATM-mediated DNA damage response







ATM









41









42



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