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The Other Side of the

HIV/AIDS Debate: Evaluating

Scientific Evidence Hidden in

Plain Sight

The public face of HIV is well-known



 Although everybody is at risk



 HIV is a sexually-transmitted virus that “selectively” preys on



 Gay men

 African-Americans

 Drug users

 Just about all of Africa



 We are encouraged to be tested



 We have been told that the AIDS drugs are the salvation of

the entire African continent



 HIV is not required to get AIDS

The public face of HIV is well-known



 The journals that review HIV tests, drugs, and patients

 As well as the instructional materials from



 Medical schools

 Centers for Disease Control (CDC)

 HIV-test manufacturers



 Will all agree with the public’s perception in the large print

 A different story emerges if you look at the fine print

 This talk will focus on an analysis of that fine print

Analyze the statistics from the CDC



 Mark Twain: There are three types of lies

1) Lies



2) Damned lies



3) Statistics



 Will this theory prove true when we examine the

statistics from the CDC’s website?

Analyze the statistics from the CDC

 From 1981 - 2005, the CDC “estimated” the number of

HIV/AIDS cases (diagnoses, deaths, and persons living with

AIDS) to be ~1 million (956,666)

 This is a 0% increase



 At the end of 2003, the CDC revised their estimates – the

number of HIV/AIDS cases (diagnoses, deaths, and persons

living with AIDS) is estimated to be between 1,039,000 to

1,185,000

 On the surface, this is a 23.9% increase in the number of HIV/AIDS cases



 The actual estimated increase or decrease rate of HIV infection

 The 1981 US population (229,465,714)

 The current US population (298,444,215)

 The actual estimated HIV/AIDS cases decreased by 23.1%





http://www.cdc.gov/hiv/resources/factsheets/At-A-Glance.htm

Analyze the statistics from the CDC:

Where are we today?

 What made HIV a Black world epidemic and how do we

account for the following predicament?

 HIV has gone from a disease that infects white homosexual

males in the U.S.



 To that of a disease that traveled across the Atlantic Ocean

and infected heterosexual Africans



 Then, it crossed the Atlantic Ocean again to infect African

American males in prison



 Now, HIV has somehow evolved with the intelligence that it

should skip the majority of the American population and become

synonymous with African Americans and women in particular





http://www.cdc.gov/hiv/resources/factsheets/At-A-Glance.htm

Analyze the statistics from the CDC:

HIV/AIDS epidemic?

 Depending on how you present the statistics, African Americans

are facing a serious epidemic

 In 2005, the CDC said that African Americans (12–13% of the

American population) make up 49% of the estimated number of

HIV/AIDS cases diagnosed



 Let’s analyze the same information a different way



 In 2005, the CDC estimated that 38,096 people were diagnosed with

HIV/AIDS



 The “estimated” percentage of people diagnosed with HIV/AIDS

 African Americans (18,667/38,797,748) is 0.048%

 U.S. population (38,096/298,444,215) is 0.013%









http://www.cdc.gov/hiv/topics/aa/resources/factsheets/aa.htm

Analyze the statistics from the CDC:

HIV/AIDS deaths?



 In 2004, the CDC “estimated” that 17,453 people died from

AIDS



 If these estimated AIDS deaths are “real”, then these deaths can

be verified by looking at the actual number of deaths for that year









http://www.cdc.gov/hiv/topics/aa/resources/factsheets/aa.htm

Leading causes of death in the U.S., 2004

Total Deaths 2,398,365

1 Diseases of the heart 654,092

2 Malignant neoplasms (cancer) 550,270

3 Cerebrovascular diseases (stroke) 150,147

4 Chronic lower respiratory diseases (emphysema,

chronic bronchitis) 123,884

5 Unintentional injuries (accidents) 108,694

6 Diabetes mellitus 72,815

7 Alzheimer’s disease 65,829

8 Influenza and pneumonia 61,472

9 Nephritis and nephrosis (Kidney disease) 42,762

10 Septicemia (systemic infection) 33,464

11 Intentional self-harm (suicide) 31,647

12 Chronic liver/cirrhosis (liver disease) 26,549

13 Essential (primary) hypo and hypertension renal disease 22,953

14 Parkinson’s disease 18,018

15 Pneumonitis due to solids and liquids 16,959



All other causes 418,810



http://www.cdc.gov/nchs/data/nvsr/nvsr54/nvsr54_19.pdf

High public policy priority: Rethinking/revising



 Although 62% of all

deaths recorded in the

U.S. in 2004 are caused

by heart disease, cancer,

strokes, and lower

respiratory diseases



 If you follow the money

budgeted per death, it

becomes apparent, the

amount of NIH funds

allocated for HIV/AIDS

research (72.6%) are

excessive









http://www.fairfoundation.org/update.htm

Sexual transmission of HIV

 The CDC says HIV is sexually transmitted



 Padian NS et al., Heterosexual transmission of Human

Immunodeficiency Virus (HIV) in Northern California: Results

from a ten-year study

 American Journal of Epidemiology. 1997;146(4):350-7



 Followed 175 HIV-discordant couples

 Although 25% of the couples had unprotected sex

 No seroconversion after entry into the study was observed

 None of the unprotected individuals became HIV+



 Sex has nothing to do with HIV/AIDS

 David W. Rasnick, PhD, member of the Scientific Group for

the Reappraisal of AIDS, wrote a letter to the British

Medical Journal entitled ”Sex has nothing to do with AIDS”

 (http://www.bmj.com/cgi/eletters/326/7381/126/e)

Sexual transmission of HIV: The prostitute

paradox

 If HIV is sexually transmitted, it should be found in sex-

trade workers

 Six additional studies published in prestigious scientific

journals that demonstrate HIV can not be sexually

transmitted

1. Potterat J J et al. Mortality in a Long-term Open Cohort of Prostitute Women. Am J

Epidemiol 2004;159:778-785.

2. Modan, B et al. Prevalence of HIV antibodies in transsexual and female prostitutes,

American Journal of Public Health. 1992;82(4):590-592.

3. Hyams KC et al. HIV infection in a non-drug abusing prostitute population. Scandinavian

Journal of Infectious Diseases. 1989;21(3):353-4.

4. Seidlin M et al. Prevalence of HIV infection in New York call girls. Journal of acquired

immune deficiency syndromes. JAIDS, 1988;1(2):150-4

5. Smith GL, Smith KF. Lack of HIV infection and condom use in licensed prostitutes.

Lancet. 1986;1392.

6. Brenky-Faudeux D, Fribourg-Blanc A. HTLV-III antibody in prostitutes. Lancet.

1985;2:1424.

Prominent scientists on the HIV=AIDS

hypothesis

 David Rasnick, PhD

 Earned his living as a designer of protease inhibitors (more on

this later)

 It has taken me 15 years of curiosity, acceptance, doubt, study,

understanding, new doubt, followed by new understanding, to

come to terms with HIV/AIDS--and I'm a scientist, I’m able to

plow through the intimidating technical literature

 No wonder the public has bought the contagious AIDS theory

 The truth is guarded by experts and hidden by a thick forest of

jargon, credentials, and all those papers

 The fraud, incompetence, and outright lies produced by the cult

of HIV have already been documented

 But holding the perpetrators accountable will not be easy

Prominent scientists on the HIV=AIDS

hypothesis

 Eleni Papadopulos-Eleopulos, PhD and her group in Perth,

Australia published articles concluding that there is no evidence

for the existence of HI viruses

 Papadopulos-Eleopulos E, Turner VF, Papadimitriou JM. IS a positive

western blot proof of HIV infection? Biotechnology NY 1 993;1 1:696-707



 Papadopulos-Eleopulos E: Is HIV the cause of AlDS? Continuum 1997;5:8-

19.



 Stefan Lanka, PhD – has experience in molecular biology,

molecular genetics, marine biology, and virology says all

retroviruses, including HIV, are biologically inexistent and their

phenomenology is based on laboratory artifacts

 Lanka S. Fehldiagnose AIDS. Wechselwirkung l994;16:48-53.



 Lanka S. HIV-Realität oder Artefakt? Raum und Zeit 1 995;77:1 7-27.



 Lanka S. HIV - reality or artifact? Continuum 1995;3/1 :4-9

Prominent scientists on the HIV=AIDS

hypothesis

 Roberto A. Giraldo, MD

 Most serologic tests that look for the presence of antibodies against

germs use neat serum [undiluted]

 Tests that look for antibodies to hepatitis A and B viruses, rubella virus,

syphilis, hystoplasma and cryptococus, etc are just a few examples



 To prevent false positive reactions, some serologic tests use diluted

serum

 Measles, varicelia, and mumps viruses use a dilution of 1:16

 Cytomegalovirus uses a dilution of 1:20

 Epstein-Barr Virus uses a dilution of 1:10

 ELISA test for HIV uses a dilution of 1:400



 If undiluted or neat serum is used, every human being on planet earth

will react positive to the ELISA test for HIV

 Tested his own serum (HIV+ at less dilute concentrations)



(http://www.virusmyth.net/aids/data/rgelisa.htm) - 1998

Prominent scientists on the HIV=AIDS

hypothesis

 Rebecca Culshaw, PhD

 Assistant Professor of Mathematics, Univ Texas at Tyler,

BS, MS, PhD

Research Interests: Mathematical Biology

Five peer reviewed publications and seven conference papers in

ten years



 Created quite a stir by announcing “Why I quit HIV” in March

2006, after having devoted ten years to mathematical modeling

of how HIV causes AIDS



 The entire basis for this theory is wrong



 AIDS is not a disease so much as it is a sociopolitical construct

that few people understand and even fewer question



http://www.lewrockwell.com/orig7/culshaw1.html

Understand the HIV/AID debate: Decouple

HIV from AIDS

 HIV: Human immunodeficiency virus

 Retroviruses

 Contain the genetic material RNA rather than DNA

 Contain genes that encode the proteins

 gag, pol, env, and (often) pro



 AIDS: Acquired immune deficiency syndrome

 As an illness, AIDS originated in the search by the CDC for sick

homosexual men, also suffering from Kaposi's Sarcoma (KS) and/or

Pneumocystis carinii pneumonia (PCP)



 KS was named for Dr. Moritz Kaposi who first described it in 1872

 Sarcoma is a cancer

 Pneumocystis carinii is a common microorganism (fungus) that exists in

mammals (rats, guinea pigs, monkeys, dogs, sheep, humans, etc.)

 First described around World War II in severely malnourished and

premature infants

Clinical conditions redefined as HIV/AIDS



 Candidiasis of bronchi, trachea, or lungs (fungal infection)

 Candidiasis esophageal

 Cervical cancer (invasive)

 Coccidioidomycosis, disseminated or extrapulmonary (fungal

disease)

 Cryptococcosis, extrapulmonary (fungal infection)

 Cryptosporidiosis, chronic intestinal for longer than 1 month

(protozoan parasite)

 Cytomegalovirus disease (other than liver, spleen or lymph nodes

(Herpes)

 Encephalopathy

 Herpes simplex: chronic ulcer(s) (for more than 1 month); or

bronchitis, pneumonitis, or esophagitis

 Histoplasmosis, disseminated or extrapulmonary (fungal infection)

 Isosporiasis, chronic intestinal (for more than 1 month) (parasitic

infection)

 Kaposi's sarcoma (human herpesvirus 8 )

 Lymphoma Burkitt's, immunoblastic or primary brain (variety of

cancers)

Clinical conditions redefined as HIV/AIDS



 Mycobacterium avium complex

 Mycobacterium, other species, disseminated or extrapulmonary

 Pneumocystis jiroveci pneumonia (formerly Pneumocystis carinii)

(fungal infection)

 Pneumonia (recurrent)

 Progressive multifocal leukoencephalopathy

 Salmonella septicemia (bacterial infection)

 Toxoplasmosis of the brain (protozoan Toxoplasma gondii)

 Tuberculosis (Mycobacterium tuberculosis)

 Wasting syndrome

 Malaria

 Dysentery

 Leprosy

 Vaccine and antibiotic damage

 Amyl nitrate damage (poppers, used by homosexual males)

 Malnutrition

HIV antibody tests: Housekeeping items



 Can not detect the actual virus



 There is no such thing as an AIDS test

 They test for non-specific antibodies in the body

 Antibodies are proteins, so please keep this in mind

throughout the rest of the presentation because this

designation will be used

 Three types of molecules in the body (DNA is transcribed into RNA

and RNA is translated into Proteins)



 There are inherent problems using antibody tests to

diagnose any disease

 People do not necessarily have the virus that their

antibodies may appear to suggest they have

HIV antibody tests: Housekeeping items

 Examples of how misleading antibody tests are

 People can have positive antibody responses to certain

laboratory chemicals, but this does not mean they are infected

with laboratory chemicals

 People vaccinated for polio will test positive for antibodies to

polio even though they don’t have polio

 People exposed to TB will test antibody positive for TB but

this does not necessarily mean they are currently infected

with TB

 The test for glandular fever measures antibody response to

red blood cells of sheep and horses, but a positive test does

not mean that someone is infected with sheep or horse blood,

or that animal blood causes glandular fever



 These examples are shown to demonstrate why

antibody responses alone cannot determine if someone

is infected with a particular virus

HIV antibody tests: Arbitrarily interpreted



 1988, the Mayo Clinic reported that “the Western Blot (WB)

method lacks standardization, is cumbersome, and is

subjective in interpretation of banding patterns”



 1988, the Journal of the American Medical Association

published an article stating that 19 different labs, testing

ONE blood sample got 19 different WB results (JAMA,

260, 1988)



 1993, a review in Bio/Technology reported that the FDA, the

CDC, the Department of Defense, and the Red Cross

 All interpret “WB” differently

 All the other major US labs for HIV testing also have

their own criteria for interpreting WB

HIV antibody tests: Arbitrarily interpreted

HIV status depends on where you live

 11 interpretations of what

constitute HIV+

 Africa is the easiest place

to be deemed HIV+

 Australia is the hardest

 6 different U.S.

interpretations

 People can literally move

to a different location and

they will no longer be

HIV+

 What other contagious

disease do you know of

that behave in this

manner?



AFR = Africa; AUS = Australia; FDA = US Food and Drug Administration; RCX = US Red Cross;

CDC = US Center for Disease Control; CON = US Consortium for Retrovirus Serology Standardization;

GER = Germany; UK = United Kingdom; FRA = France; MACS = US Multicenter AIDS Cohort Study 1983-1992

HIV antibody tests: No virologic gold

standard

 The medical literature adds something truly astounding!

 1987, the New England Journal of Medicine reported that the meaning

of positive tests will depend on the joint (ELISA/WB) false positive

rate

 The real rate is unknown because there is no recognized gold standard



 1996, Journal of American Medical Association reported: The diagnosis

of HIV infection in infants is particularly difficult

 Because there is no reference or “gold standard”



 1997, Abbott laboratories, the world leader in HIV-test production

stated

 At present, there is no recognized standard for establishing the “presence

or absence” of HIV antibody in human blood

 2000, the Journal AIDS reported that "2.9% - 12.3%" of women in a

study tested positive, depending on the test used

 Since there is no established gold standard test, it is unclear which of these

two proportions is the “best estimate” of the real prevalence rate

No virologic gold standard: Insert from

Abbott Laboratory









http://www.healtoronto.com/testkits.html

High false positive rate with HIV tests



 1985, at the beginning of HIV testing, it was reported in

the New England Journal of Medicine

 "68% to 89%” of all repeatedly reactive ELISA (HIV antibody) tests were

likely to represent false positive results



 1992, the Lancet reported ("HIV Screening in Russia")

 For 66 true positives, there were 30,000 false positives

 In pregnant women, "there were 8,000 false positives for 6 confirmations"



 2000, the Archives of Family Medicine reported

 The more women we test, the greater "the proportion of false positive and

ambiguous (indeterminate) test results"



 The tests described above are standard HIV tests, the

kind promoted in the ads

 ELISA or EIA (Enzyme-linked immuno-sorbant assay)

HIV tests are non-specific: ELISA & WB



 The ELISA is the first test that is used for HIV detection

 If you test HIV+, a second ELISA is recommended, if you test HIV+

again, then a Western Blot (WB) is performed

 ELISA - a mixture of proteins, which are said to come only from

HIV, is exposed to a blood sample and any antibodies in the blood

that can bind to these proteins are allowed to do so

 If all of the proteins in the mixture come from HIV, “and” if all of

the antibodies recognize only HIV proteins, a positive reading means

that a person has been exposed to HIV



 1993, Eleni Papadopulos-Eleopulos, PhD

 The proteins in the mixture are not unique to HIV for the ELISA or WB

 The antibodies in the blood samples are not specific only to HIV

proteins





Papadopulos-Eleopulos, E., Turner, V.F., Papadimitriou, J.M. 1993.

"Is a positive Western Blot proof of HIV infection?" Bio/Technology. 11:696-707

HIV tests are non-specific: Example of a

Western Blot

 All retroviruses

 Contain genes that encode

for the proteins (gag, pol,

env)

 In the U.S., there are 5

major proteins used to

determine a person’s HIV

status





env gp160

gp120

gp 41

gag p55

p18

p24

pol p65

p51

p31



Image reproduced from Commercial Methods in Clinical Microbiology, 2000. ASM Press.

HIV tests are non-specific: Analyzing the

important protein bands in the WB

 Analysis of the five proteins that determines a person’s HIV

status

 p24: Found in all endogenous retroviruses (HTLV-1, HTLV-II, HIV-2)

 p31: The amino-acid sequences of the "purified HIV (p30-p32)” are

identical to that of a normal protein found in the human immune system

called "Class II histocompatibility DR proteins"

 Henderson, L.E., Sowder, R., Copeland, T.D., et. al. 1987. "Direct identification of Class II

histocompatibility DR proteins in preparations of human T-cell lymphotrophic virus type III." J.

Virol. 61:629-632.



 p41: Protein called actin - the most abundant protein in human cells

 In some cells, actin accounts for 15% of the total cellular content

 Actin filaments drives shape changes, cell locomotion, chemotactic migration

and participate in muscle contraction



 p120, p160 - oligomers of p41, for instance (p 120 = p40 *3 and p160 =

p40*4)

66 factors known to generate a false

positive on HIV tests

1. Anti-carbohydrate antibodies

2. Naturally-occurring antibodies

3. Passive immunization: receipt of gamma or immune globulin

4. Leprosy

5. Tuberculosis

6. Mycobacterium avium

7. Systemic lupus erythematosus

8. Renal (kidney) failure

9. Hemodialysis/renal failure

10. Alpha interferon therapy in hemodialysis patients

11. Flu

12. Flu vaccination

13. Herpes simplex I

14. Herpes simplex II

15. Upper respiratory tract infection (cold or flu)

16. Recent viral infection or exposure to viral vaccines

17. Pregnancy in multiparous women

18. Malaria

19. High levels of circulating immune complexes

66 factors known to generate a false

positive on HIV tests

20. Hypergammaglobulinemia (high levels of antibodies)

21. False positives on other tests, including RPR (rapid plasma

reagent) test for syphilis

22. Rheumatoid arthritis

23. Hepatitis B vaccination

24. Tetanus vaccination

25. Organ transplantation

26. Renal transplantation

27. Anti-lymphocyte antibodies

28. Anti-collagen antibodies (found in gay men, haemophiliacs,

Africans of both sexes and people with leprosy)

29. Serum-positive for rheumatoid factor, antinuclear antibody

(both found in rheumatoid arthritis and other autoantibodies)

30. Autoimmune diseases: Systemic lupus erythematosus,

scleroderma, connective tissue disease, dermatomyositis

31. Acute viral infections, DNA viral infections

32. Malignant neoplasms (cancers)

33. Alcoholic hepatitis/alcoholic liver disease

34. Primary sclerosing cholangitis

66 factors known to generate a false

positive on HIV tests

35. Hepatitis

36. "Sticky" blood (in Africans)

37. Antibodies with a high affinity for polystyrene (used in

the test kits)

38. Blood transfusions, multiple blood transfusions

39. Multiple myeloma

40. HLA antibodies (to Class I and II leukocyte antigens)

41. Anti-smooth muscle antibody

42. Anti-parietal cell antibody

43. Anti-hepatitis A IgM (antibody)

44. Anti-Hbc IgM

45. Administration of human immunoglobulin preparations

pooled before 1985

46. Hemophilia

47. Hematologic malignant disorders/lymphoma

48. Primary biliary cirrhosis

49. Stevens-Johnson syndrome

50. Q-fever with associated hepatitis

51. Heat-treated specimens

66 factors known to generate a false

positive on HIV tests

52. Lipemic serum (blood with high levels of fat or lipids)

53. Hemolyzed serum (blood where haemoglobin is

separated from the red cells)

54. Hyperbilirubinemia

55. Globulins produced during polyclonal gammopathies

(which are seen in AIDS risk groups)

56. Healthy individuals as a result of poorly-understood

cross-reactions

57. Normal human ribonucleoproteins

58. Other retroviruses

59. Anti-mitochondrial antibodies

60. Anti-nuclear antibodies

61. Anti-microsomal antibodies

62. T-cell leukocyte antigen antibodies

63. Proteins on the filter paper

64. Epstein-Barr virus

65. Visceral leishmaniasis

66. Receptive anal sex

References - factors known to cause a false

positive on HIV tests

1. Agbalika F, Ferchal F, Garnier J-P, et al. 1992. False-positive antigens related to emergence of a 25-30 kD

protein detected in organ recipients. AIDS. 6:959-962.



2. Andrade V, Avelleira JC, Marques A, et al. 1991. Leprosy as a cause of false-positive results in serological assays

for the detection of antibodies to HIV-1. Intl. J. Leprosy. 9:125.



3. Arnold NL, Slade RA, Jones MM, et al. 1994. Donor follow up of influenza vaccine-related multiple viral enzyme

immunoassay reactivity. Vox Sanguinis. 67:191.



4. Ascher D, Roberts C. 1993. Determination of the etiology of seroreversals in HIV testing by antibody

fingerprinting. AIDS. 6:241.



5. Barbacid M, Bolgnesi D, Aaronson S. 1980. Humans have antibodies capable of recognizing oncoviral glycoproteins:

Demonstration that these antibodies are formed in response to cellular modification of glycoproteins rather than

as consequence of exposure to virus. Proc. Natl. Acad. Sci. 77:1617-1621.



6. Biggar R, Melbye M, Sarin P, et al. 1985. ELISA HTLV retrovirus antibody reactivity associated with malaria and

immune complexes in healthy Africans. Lancet. ii:520-543.



7. Blanton M, Balakrishnan K, Dumaswala U, et al. 1987. HLA antibodies in blood donors with reactive screening tests

for antibody to the immunodeficiency virus. Transfusion. 27(1):118.



8. Blomberg J, Vincic E, Jonsson C, et al. 1990. Identification of regions of HIV-1 p24 reactive with sera which

give "indeterminate" results in electrophoretic immunoblots with the help of long synthetic peptides. AIDS Res.

Hum. Retro. 6:1363.



9. Burkhardt U, Mertens T, Eggers H. 1987. Comparison of two commercially available anti-HIV ELISA's: Abbott

HTLV-III ELA and DuPont HTLV-III ELISA. J. Med. Vir. 23:217.



10. Bylund D, Ziegner U, Hooper D. 1992 Review of testing for human immunodeficiency virus. Clin. Lab. Med.

12:305-333.

References - factors known to cause a false

positive on HIV tests

11. Challakere K, Rapaport M. 1993. False-positive human immunodeficiency virus type 1 ELISA results in low-risk

subjects. West. J. Med. 159(2):214-215.



12. Charmot G, Simon F. 1990. HIV infection and malaria. Revue du practicien. 40:2141.



13. Cordes R, Ryan M. 1995. Pitfalls in HIV testing. Postgraduate Medicine. 98:177.



14. Dock N, Lamberson H, O'Brien T, et al. 1988. Evaluation of atypical human immunodeficiency virus immunoblot

reactivity in blood donors. Transfusion. 28:142.



15. Esteva M, Blasini A, Ogly D, et al. 1992. False positive results for antibody to HIV in two men with systemic

lupus erythematosus. Ann. Rheum. Dis. 51:1071-1073.



16. Fassbinder W, Kuhni P, Neumayer H. et al. 1986. Prevalence of antibodies against LAV/HTLV-III [HIV] in

patients with terminal renal insufficiency treated with hemodialysis and following renal transplantation. Deutsche

Medizinische Wochenschrift. 111:1087.



17. Fleming D, Cochi S, Steece R. et al. 1987. Acquired immunodeficiency syndrome in low-incidence areas. JAMA.

258(6):785.



18. Gill MJ, Rachlis A, Anand C. 1991. Five cases of erroneously diagnosed HIV infection. Can. Med. Asso. J.

145(12):1593.



19. Healey D, Bolton W. 1993. Apparent HIV-1 glycoprotein reactivity on Western blot in uninfected blood donors.

AIDS. 7:655-658.



20. Hisa J. 1993. False-positive ELISA for human immunodeficiency virus after influenza vaccination. JID. 167:989.



21. Isaacman S. 1989. Positive HIV antibody test results after treatment with hepatitis B immune globulin. JAMA.

262:209.

References - factors known to cause a false

positive on HIV tests

22. Jackson G, Rubenis M, Knigge M, et al. 1988. Passive immunoneutralisation of human immunodeficiency virus in

patients with advanced AIDS. Lancet, Sept. 17:647.



23. Jindal R, Solomon M, Burrows L. 1993. False positive tests for HIV in a woman with lupus and renal failure.

NEJM. 328:1281-1282.



24. Jungkind D, DiRenzo S, Young S. 1986. Effect of using heat-inactivated serum with the Abbott human T-cell

lymphotropic virus type III [HIV] antibody test. J. Clin. Micro. 23:381.



25. Kashala O, Marlink R, Ilunga M. et al. 1994. Infection with human immunodeficiency virus type 1 (HIV-1) and

human T-cell lymphotropic viruses among leprosy patients and contacts: correlation between HIV-1 cross-

reactivity and antibodies to lipoarabionomanna. J. Infect. Dis. 169:296-304.



26. Lai-Goldman M, McBride J, Howanitz P, et al. 1987. Presence of HTLV-III [HIV] antibodies in immune serum

globulin preparations. Am. J. Clin. Path. 87:635.



27. Langedijk J, Vos W, Doornum G, et al. 1992. Identification of cross-reactive epitopes recognized by HIV-1

false-positive sera. AIDS. 6:1547-1548.



28. Lee D, Eby W, Molinaro G. 1992. HIV false positivity after hepatitis B vaccination. Lancet. 339:1060.



29. Leo-Amador G, Ramirez-Rodriguez J, Galvan-Villegas F, et al. 1990. Antibodies against human immunodeficiency

virus in generalized lupus erythematosus. Salud Publica de Mexico. 32:15.



30. Mackenzie W, Davis J, Peterson D. et al. 1992. Multiple false-positive serologic tests for HIV, HTLV-1 and

hepatitis C following influenza vaccination, 1991. JAMA. 268:1015-1017.



31. Mathe G. 1992. Is the AIDS virus responsible for the disease? Biomed & Pharmacother. 46:1-2.



32. Mendenhall C, Roselle G, Grossman C, et al. 1986. False-positive tests for HTLV-III [HIV] antibodies in

alcoholic patients with hepatitis. NEJM. 314:921.

References - factors known to cause a false

positive on HIV tests

33. Moore J, Cone E, Alexander S. 1986. HTLV-III [HIV] seropositivity in 1971-1972 parenteral drug abusers - a

case of false-positives or evidence of viral exposure? NEJM. 314:1387-1388.



34. Mortimer P, Mortimer J, Parry J. 1985. Which anti-HTLV-III/LAV [HIV] assays for screening and comfirmatory

testing? Lancet. Oct. 19, p873.



35. Neale T, Dagger J, Fong R, et al. 1985. False-positive anti-HTLV-III [HIV] serology. New Zealand Med. J.

October 23.



36. Ng V. 1991. Serological diagnosis with recombinant peptides/proteins. Clin. Chem. 37:1667-1668.



37. Ozanne G, Fauvel M. 1988. Perfomance and reliability of five commercial enzyme-linked immunosorbent assay kits

in screening for anti-human immunodeficiency virus antibody in high-risk subjects. J. Clin. Micro. 26:1496.



38. Papadopulos-Eleopulos E. 1988. Reappraisal of AIDS - Is the oxidation induced by the risk factors the primary

cause? Med. Hypo. 25:151.



39. Papadopulos-Eleopulos E, Turner V, and Papadimitriou J. 1993. Is a positive Western blot proof of HIV

infection? Bio/Technology. June 11:696-707.



40. Pearlman ES, Ballas SK. 1994. False-positive human immunodeficiency virus screening test related to rabies

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41. Peternan T, Lang G, Mikos N, et al. Hemodialysis/renal failure. 1986. JAMA. 255:2324.



42. Piszkewicz D. 1987. HTLV-III [HIV] antibodies after immune globulin. JAMA. 257:316.



43. Profitt MR, Yen-Lieberman B. 1993. Laboratory diagnosis of human immunodeficiency virus infection. Inf. Dis.

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44. Ranki A, Kurki P, Reipponen S, et al. 1992. Antibodies to retroviral proteins in autoimmune connective tissue

disease. Arthritis and Rheumatism. 35:1483.

References - factors known to cause a false

positive on HIV tests

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46. Sayers M, Beatty P, Hansen J. 1986. HLA antibodies as a cause of false-positive reactions in screening enzyme

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47. Sayre KR, Dodd RY, Tegtmeier G, et al. 1996. False-positive human immunodeficiency virus type 1 Western blot

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48. Schleupner CJ. Detection of HIV-1 infection. In: (Mandell GI, Douglas RG, Bennett JE, eds.) Principles and

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49. Schochetman G, George J. 1992. Serologic tests for the detection of human immunodeficiency virus infection. In

AIDS Testing Methodology and Management Issues, Springer-Verlag, New York.



50. Simonsen L, Buffington J, Shapiro C, et al. 1995. Multiple false reactions in viral antibody screening assays after

influenza vaccination. Am. J. Epidem. 141-1089.



51. Smith D, Dewhurst S, Shepherd S, et al. 1987. False-positive enzyme-linked immunosorbent assay reactions for

antibody to human immunodeficiency virus in a population of midwestern patients with congenital bleeding

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52. Snyder H, Fleissner E. 1980. Specificity of human antibodies to oncovirus glycoproteins; Recognition of antigen by

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53. Steckelberg JM, Cockerill F. 1988. Serologic testing for human immunodeficiency virus antibodies. Mayo Clin.

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54. Sungar C, Akpolat T, Ozkuyumcu C, et al. Alpha interferon therapy in hemodialysis patients. Nephron. 67:251.

References - factors known to cause a false

positive on HIV tests

55. Tribe D, Reed D, Lindell P, et al. 1988. Antibodies reactive with human immunodeficiency virus gag-coated

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56. Ujhelyi E, Fust G, Illei G, et al. 1989. Different types of false positive anti-HIV reactions in patients on

hemodialysis. Immun. Let. 22:35-40.



57. Van Beers D, Duys M, Maes M, et al. Heat inactivation of serum may interfere with tests for antibodies to

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58. Voevodin A. 1992. HIV screening in Russia. Lancet. 339:1548.



59. Weber B, Moshtaghi-Borojeni M, Brunner M, et al. 1995. Evaluation of the reliability of six current anti-HIV-

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60. Wood C, Williams A, McNamara J, et al. 1986. Antibody against the human immunodeficiency virus in commercial

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61. Yale S, Degroen P, Tooson J, et al. 1994. Unusual aspects of acute Q fever-associated hepatitis. Mayo Clin.

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62. Yoshida T, Matsui T, Kobayashi M, et al. 1987. Evaluation of passive particle agglutination test for antibody to

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63. Yu S, Fong C, Landry M, et al. 1989. A false positive HIV antibody reaction due to transfusion-induced HLA-

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64. National Institue of Justice, AIDS Bulletin. Oct. 1988.

Low CD4 T-cell count: Non-HIV/AIDS

diagnosis

 Prior to 1993, the definition of AIDS required clinical symptoms

of serious disease

 According to the 1993 redefinition of AIDS, clinically healthy

but HIV+ people in the US have "AIDS" when their CD4 cell

count drops below 200.

 This redefinition is absurd - a variety of physical and even

psychological conditions have been shown to cause very low CD4

cell counts in "HIV negative" individuals

 Literally overnight, this change of definition caused the number

of people with "AIDS" in the United States to double

 From 1993 to 1997, the CDC disclosed the percentage of AIDS

patients that had AIDS'93, but not AIDS'87

 Starting in 1998, the CDC would no longer disclose which percentage

of AIDS cases was "AIDS'93" but not "AIDS'87", and stonewalled

all attempts of AIDS rethinkers to acquire it

Low CD4 T-cell count: A Variety of causes

account for this phenomenon

 Many viral infections

 Bacterial infections

 Parasitic infections

 Sepsis

 Tuberculosis

 Coccidioidomycosis (acquired from inhalation of spores)

 Burns

 Trauma

 Intravenous injections of foreign proteins

 Malnutrition

 Over-exercising

 Intravenous drug users

 Pregnancy

 Normal daily variation

 Psychological stress and social isolation

 Malaria

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The viral load test: Polymerase chain

reaction (PCR)

 PCR is method of rapidly synthesizing many copies of a specific

segment of DNA

 PCR is the biotechnology version of the Xerox machine

 The amount of DNA you have to study increases exponentially



 Viral load tests suppose to measure the amount of HIV RNA

present in the blood stream, but, instead they measure genetic

fragments, not levels of active virus in the body



 The viral load hypothesis fails to answer two important

questions

1. If billions of HIV are present, why is PCR necessary to find them?

2. If PCR is the only way HIV can be detected, how is it possible for

scientists to verify the results of PCR?

The viral load test: Invalid and not

reproducible

 Nobel Laureate Kary Mullis, the inventor of the PCR method

has stated publicly that "viral load" tests are invalid

 The “so-called viral load numbers” are not reproducible, not

even when the same technology is used

 A nationwide team of orthodox AIDS researchers led by

doctors Benigno Rodriguez and Michael Lederman of Case

Western Reserve University in Cleveland

 Disputed the value of viral load tests standard used since 1996

to assess health, predict progression to disease, and grant

approval to new AIDS drugs after their study of 2,800 HIV

positives concluded viral load measures failed in more than

90% of cases to predict or explain immune status









Published in the September 27, 2006 issue of the Journal of the American Medical Association

The viral load test: Invalid and not

reproducible

 In a study by French

researchers

 15 HIV-1 strains using 3 viral load tests

were analyzed

 The samples contained the same

load of this alleged “HIV” as

quantified by p24 measurements

 p24: Found in all endogenous

retroviruses (HTLV-1, HTLV-II, HIV-2)

 If the tests were true

measurement of HIV RNA the

results should have been the

same for all strains in a given

test and all tests for a specific

strain

 Every number to the right of the first

column should be identical







Journal of Acquired Immune Deficiency Syndromes and Human Retrovirology 15:174.

Another analysis of the HIV/AIDS

statistics from the CDC’s website

 In 2005, the CDC estimated that 38,096 people were

diagnosed with HIV/AIDS



 African Americans (12–13% of the American population)

make up 49% of the estimated number of HIV/AIDS cases

diagnosed

 (38,096 *0.49) 18,667



 If HIV exits, the actual HIV diagnoses that should have

occurred can be calculated by subtracting

 The diagnoses that are a direct result of a low CD4 cell count

 The false positives generated from the joint ELISA/WB test

Another analysis of the HIV/AIDS

statistics from the CDC’s website

 1/2 (38,096) of those diagnosed with HIV/AIDS are from a

low CD4 count (19,048)

 Assuming a 90% false positive rate based upon the joint WB

and ELISA antibody tests 19,048 *0.90 (17,143)

 The HIV/AIDS cases should have been 38,096 – 19,048-

17,143 (1,905 people in the entire U.S.)



 Since 49% of those estimated to be infected with HIV are

African Americans

 This means that 933 out of ~39 million African Americans may

have this thing that is called “HIV”



 Since antibodies are really not a good measure to detect any

disease, then this number should be reduced to “Zero”

If commerce laws were applied equally

 HIV tests would have to bear a disclaimer just like

cigarettes



 “WARNING"

 This test will not tell you if you are infected with a virus

 It may confirm that you are pregnant

 It may confirm that you have used drugs or alcohol

 It may confirm that you have been vaccinated

 It may confirm that you have a cold, liver disease, arthritis

 It may confirm that you are stressed, poor, hungry, or tired

 It may confirm that you are an African

 It will not tell you if you are going to live or die

 In fact, we really do not know what testing “positive or

negative” means at all



Liam Scheff is an investigative journalist whose research was the basis for the 2004 BBC documentary,

"Guinea Pig Kids," about the forced use of experimental AIDS drugs.

The fine print: Summary of scientific

evidence

 They tell you, unabashedly

 HIV tests are arbitrarily interpreted



 HIV tests are not standardized (no gold standard)



 The term HIV does not describe a single entity



 HIV describes a collection of non-specific, cross-reactive

cellular material



 HIV can not be sexually transmitted



 HIV is not required for AIDS



 What is causing people to become sick?

The Drugs: HIV therapy - AZT



 Liquid Plummer

 Developed in the 1960s as a chemotherapy for leukemia

 A "nucleoside analog" drug, or DNA chain terminator

 Stops the DNA molecule from duplicating

 Kills cells that try to reproduce

 Chemotherapies are notoriously immunosuppressive

 The idea for cancer treatment is that a short shock program of maybe two or three

weeks will kill the tumor while only half-killing the patient

 Then you get the person off the therapy as quickly as possible

 Then build up the person’s immune system



 Officially acknowledged side effects

 Diarrhea, dementia, lymphoma (cancer), muscle wasting, and T-cell depletion, which are

also AIDS-defining conditions

The Drugs: HIV therapy – protease

inhibitors

 Proteases

 Are some of the most important enzymes (proteins) that we

have

 They aid in the breakdown of proteins in the body (digestion

of protein)



 Protease inhibitors

 Throw your body out of homeostasis

 Inhibit the body's natural proteases

 Prevent the digestion of proteins

 If the digestive process is incomplete, undigested proteins can

wind up in a person’s circulatory system, as well as in other parts

of the body

 Will cause an autoimmune response

Side effects - protease inhibitors

 Invirase (Hoffmann-LaRoche inserts)

 Body as a whole: allergic reaction, chest pain, edema, fever, intoxication, parasites

external, retrosternal pain, shivering, wasting syndrome, weight decrease



 Cardiovascular: Cyanosis, heart murmur, heart valve disorder, hypertension, hypotension,

syncope, vein distended



 Endocrine/Metabolic: Dehydration, dry eye syndrome, hyperglycemia, xerophthalmia



 Gastrointestinal: Cheilitis, constipation, dysphagia, eructation, feces bloodstained, feces

discolorred, gastralgia, gastritis, gastrointestinal inflammation, gingivitis, glossitis,

hemorrhage rectum, hemorrhoids, hepatomegaly, melena, pain pelvic, painful defecation,

pancreatitis, parotic disorder, salivary glands disorder, stomatitis, tooth disorder,

vomiting



 Hematlogic: Anemia, microhemorrhages, pancytopenia, splenomegaly, thrombocytopenia



 Musculoskeletal: Arthralgia, arthritis, back pain, cramps muscle, musculoskeletal

disorders, stiffness, tissue changes, trauma



 Neurological: Ataxia, bowel movements frequent, confusion, convulsions, dysarthria,

dysesthesia, heart rate disorder, hyperesthesia, hyperreflexia, hyporeflexia, mouth dry,

numbness face, pain facial, paresis, poliomyelitis, progressive multifocal

leukoencephalopathy, spasms, tremor

Side effects - protease inhibitors

 Invirase (continued)

 Psychological: Agitation, amnesia, anxiety, depression, dream excessive, euphoria,

hallucination, insomnia, intellectual ability reduced, irritability, lethargy, libido

disorder, overdose effect, psychotic disorder, somnolence, speech disorder



 Reproductive System: Prostate enlarged, vaginal discharge



 Resistance Mechanism: Abscess, angina tonsillaris, candidiasis, hepatitis, herpes

simplex, herpes zoster, infection bacterial, infection mycotic, infection

staphylococcal, influenza, lymphadenopathy, tumor



 Respiratory: Bronchitis, cough, dyspnea, epistaxis, hemoptysis, laryngitis,

pharyngitis, pneumonia, respiratory disorder, rhinitis, sinusitis, upper respiratory

tract infection



 Skin and Appendages: Acne, dermatitis, dermatitis seborrheic, eczema, erythema,

folliculitis, furunculosis, hair changes, hot flushes, photosensitivity reaction,

pigment changes skin, rash maculopapular, skin disorder, skin nodule, skin

ulceration, sweating increased, urticaria, verruca, xeroderma



 Special Senses: Blepharitis, earache, ear pressure, eye irritation, hearing

decreased, otitis, taste alteration, tinnitus, visual disturbance



 Urinary system: Micturition disorder, urinary tract infection

Side effects - protease inhibitors

 Crixivan (Merck, Sharp & Dohme inserts)



 Body as a whole/site unspecified: Abdominal distention, chest pain,

chills, fever, flank pain, flu-like illness, fungal infection, malaise, pain,

syncope



 Cardiovascular system: Cardiovascular disorder, palpitation



 Digestive system: Acid regurgitation, anorexia, aphthous stomatitis,

cheilitis, cholecystitis, cholestasis, constipation, dry mouth, dyspepsia,

eructation, flatulence, gastritis, gingivitis, glossodynia, gingival

hemorrhage, increased appetite, infectious gastroenteritis, jaundice,

liver cirrhosis



 Hemic and Lymphatic System: Anemia, lymphadenopathy, spleen disorder



 Metabolic/Nutritional/Immune: Food allergy



 Musculoskeletal system: Arthralgia, back pain, leg pain, myalgia, muscle

cramps, muscle weakness, musculoskeletal pain, shoulder pain, stiffness

Side effects - protease inhibitors

 Crixivan (continued)

 Nervous system and psychiatric: Agitation, anxiety, anxiety disorder, bruxism,

decreased mental acuity, depression, dizziness, dream abnormality, dysesthesia,

excitement, fasciculation, hypesthesia, nervousness, neuralgia, neurotic disorder,

paresthesia, peripheral neuropathy, sleep disorder, somnolence, tremor, vertigo



 Respiratory system: Cough, dyspnea, halitosis, pharyngeal hyperemia, pharyngitis,

pneumonia, rales/rhonchi, respiratory failure, sinus disorder, sinusitis, upper

respiratory infection



 Skin and skin Appendage: Body odor, contact dermatitis, dermatitis, dry skin,

flushing, folliculitis, herpes simplex, herpes zoster, night sweats, pruritus,

seborrhea, skin disorder, skin infection, sweating, urticaria



 Special senses: Accommodation disorder, blurred vision, eye pain, eye swelling,

orbital edema, taste disorder



 Urogenital system: Dysuria, hematuria, hydronephrosis, nocturia, premenstrual

syndrome, proteinuria, renal colic, urinary frequency, urinary tract infection, urine

abnormality, urine sediment abnormality, urolithiasis

Side effects – AIDS drugs





Photos of an infant with Stevens-Johnson Syndrome, a

blistering, peeling, potentially fatal skin rash. It is one of the

known side-effects of the AIDS drug Nevirapine (Viramune).

Viramune is one of the primary drugs being readied for

distribution in Africa.









“Viramune is not a cure for HIV-1 infection.”

Side effects – Protease inhibitor effects



BUFFALO HUMPS" between the shoulders and protruding

abdomen

Confidential name-based HIV infection

reporting



 There are 33 States and 4 Dependent Areas that will not

release your name if you test HIV +

 Alabama, Alaska, Arizona, Arkansas, Colorado, Florida, Idaho,

Indiana, Iowa, Kansas, Louisiana, Michigan, Minnesota,

Mississippi, Missouri, Nebraska, Nevada, New Jersey, New

Mexico, New York, North Carolina, North Dakota, Ohio,

Oklahoma, South Carolina, South Dakota, Tennessee, Texas,

Utah, Virginia, West Virginia, Wisconsin, Wyoming



 American Samoa, Guam, the Northern Mariana Islands, and

the U.S. Virgin Islands



 Illinois is not on that list

AIDS in Africa: The Bangui definition



 In 1985, the WHO called a meeting in Bangui, the capital

of the Central African Republic, to define African AIDS

 The meeting was presided over by CDC official Joseph

McCormick

 McCormick wrote about it in his book "Level 4 Virus

hunters of the CDC," saying…

 If I could get everyone at the WHO meeting in Bangui to agree on

a single, simple definition of what an AIDS case was in Africa,

then, imperfect as the definition might be, we could actually start

counting the cases



 The result was - African AIDS would be defined by physical

symptoms: fever, diarrhea, weight loss, and coughing or itching



AIDS in Africa: an epidemiological paradigm, Science, 1986

In Africa, HIV status is irrelevant



 Even if you test negative, you can be called an AIDS

patient

 In 1992, a study in Ghana: 59% of the seronegative (HIV-

negative) group were clinically diagnosed as having AIDS

 All the patients had three major signs: weight loss, prolonged

diarrhea, and chronic fever

 Lancet, October, 1992



 Across Africa: 50% (2215 out of 4383) African AIDS

patients from Abidjan, Ivory Coast, Lusaka, Zambia, and

Kinshasa, Zaire, were HIV-antibody negative

 British Medical Journal, 1991

In Sub-Saharan Africa

 ~60% of the population lives and dies without safe drinking water,

adequate food, or basic sanitation

 Sep, 2003 report in the Ugandan Daily "New Vision" outlined the

situation in Kampala, a city of ~ 1.3 million inhabitants, which, like

most tropical countries, experience seasonal flooding

 In the flood zone

 Heaps of unclaimed garbage among the crowded houses

 Countless pools of water that provide a breeding ground for mosquitoes

and create a dirty environment that favors cholera

 Latrines are built above water streams

 During rain - residents open a hole to release the feces from the latrines

 The rain then washes away the feces to the streams

 The residents fetch water from the streams



 Some defecate in polythene bags, which they throw into the

stream (flying toilets)

False positive rate in South Africa





 Dec 2002 – an article published by a fifth-year medical

student at Bristol University in Britain



 Mukai Chimuterngwende-Gordon



 83% chance that the HIV test mechanism in Africa -

called Enzyme-Linked Immuno-Sorbent Assay (Elisa)

would produce false results









http://new.hst.org.za/news/index.php/20030118/


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