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ANTIBODY MEDIATED IMMUNE RESPONSES

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LECTURE OUTLINE

ANTIBODY MEDIATED IMMUNE

RESPONSES



LEARNING OBJECTIVES

At the end of the lecture students should be able to know ,

• Various types of antibodies

• Different functions of antibodies

• Different Mechanisms of antibody mediated immune

responses.

Antibodies or Immunoglobulins

* Definition:

Glycoprotein in serum and tissue fluid



* Produced by:

B-lymphocytes in response to exposure to antigen



* React specifically with antigen



* Five classes of Antibodies:

IgG

IgM

IgA

IgD

IgE

IgG

Properties

– Major serum Ig

– Major Ig in extravascular spaces

– The only Placental transfer Ig

– Fixes complement

– Phagocytes - opsonization

IgM

Properties

– First Ig made by fetus and B cells

– Present in colostrum and mother milk protect newly born.

– Fixes complement



IgA

-Found in serum and body secretion:

Tears, saliva, gastric and pulmonary

secretions

– Major secretory Ig on Mucous surfaces give Local Immunity by

coating m.o, bacteria or viruses preventing their adherence to

mucosal cells

– Does not fix complement (unless aggregated)

– Present in colostrum and mother milk protect newly born.



IgE

– Least common serum Ig

• Binds to basophils and mast cells (Does not require Ag binding)

– Allergic and hypersensitivity reactions

– Parasitic infections (Helminths)

• Binds to Fc receptor on eosinophils

– Does not fix complement

IgD

– Present in very small amount in serum

– B cell surface Ig

– Does not bind complement

Antibody Production



Clonal selection theory:



*B-cells display immunoglobulin molecules on surfaces

*Immunoglobulin serve as receptors for specific antigen

*The antigen binds to immunoglobulin receptor of B-cells

*B-cells is stimulated to divide and form a clone

*B-cells become plasma cells and secrete antibodies

*Some stimulated B-cells revert to small lymphocyte (memory cells)

*Memory cells proliferate on re-exposure to same antigen





Activation of B-cells

Two mechanisms:

1) T-dependent antigen:

. Most antigen require T-helper cells to activate B-cells

. Antigen is phagocytosed by macrophages or B-cells

. Macrophages or B process present Ag to T-cells

. These activate T-cells to produce lymphokines

. lymphokines (IL-2,IL-4,IL,5) stimulate B-cells to

divide and differentiate into plasma cells



specific antibody

. Plasma cells form or

differentiate into memory cells



. All classes of antibody (IgG,IgM,IgA,IgD,IgE) are T-cell dependant



2) T-independent antigens:

. Activation of B-cells directly without help of T-cells(e.g. bacterial

capsular polysaccharides)

. IgM antibody is primarily produced

Antibody Structure



Immunoglobulins are glycoproteins made up of

- Four polypeptid chains (IgG):

a- Two light (L) polypeptide chains

b- Two heavy (H) polypeptide chains







- The four chains are linked by disulfide bonds

- Terminal portion of L-chain contains part of antigen binding site

- H-chains are distinct for each of the five immunoglobulins

- Terminal portion of H-chain participate in antigen binding site

- The other (Carboxyl) terminal portion forms Fc fragment

Variable(V) and Constant (C) Regions

- Each H-chain and each L-chain has V-region and C-region

- V-region lies in terminal portion of molecule

- V-region shows wide variation in

amino a. sequences

- Hypervariable region form region

complementary to Ag determinant

- It is responsible for antigen binding

- C-region lies in carboxyl or terminal portion of molecule

- C-region shows an unvarying amino acid sequence

- It is responsible for biologic function







Antibody Fragments

Fab fragment: antigen binding

site









Fc (crystallizable fragment):

a- Complement fixation (IgM and IgG)

b- Opsonization (IgG)

C- Placental attachment (IgG)

d- Mucosal attachment (IgA)

e- Binding to mast cells (IgE)

Properties of Immunoglobulins





Antibody Diversity

*Immunoglobulins are protein (antigenic)



*Immunoglobulins subdivided into :



a- Isotypes: Antigenic difference in C-region

five immunogl. classes are different isotypes



b- Idiotypes: Ag difference in V-region of immunogl.



c- Allotypes: Antigenic feature of immunogl. that vary

among individual under genetic control

Ag difference in C-region of H and L chain









Primary and Secondary antibody response

Primary antibody respone Secondary antibody response



* first exposure to antigen * Subsequent exposure

* lag period: days or weeks * Lag period: hours

(slow onset) (rapid onset)

* Small amount immunogl. * large amount immunogl.

low Ab level with gradual increase high Ab with rapid increas

* Ab Persist for short duration * Persist for long periods Weeks then

decline rapidly (monthes or years)

* Antibody is IgM * Antibody is IgG

Primary and Secondary antibody response



Primary and Secondary antibody response





Secondary response

to Ag









Amount of

Primary response

antibodies to Ag

in serum









1 2 3 4 5 6

Time (months)

1st injection of Ag 2nd injection of Ag





Mechanism of antibody mediated immune

response

• Antibodies can work in several ways, depending on the nature of the

antigen.

• Antibodies that interlock with toxins produced by certain bacteria can

disable them directly (and are known as antitoxins).

• by coating (or opsonizing) bacteria, make the microbes highly palatable

to scavenger cells equipped to engulf and destroy them.

• More often an antigen-antibody combination unleashes a group of lethal

enzymes known as Complement.

• Yet other antibodies block viruses from entering into cells (a quality that

is exploited in making vaccines). And, in a phenomenon known as

antibody-dependent cell-mediated cytotoxicity (ADCC), cells coated with

antibody become vulnerable to attack by several types of white blood

cells.

Antibody Targets

• Antibodies themselves do not destroy antigen; they inactivate

and tag it for destruction

• All antibodies form an antigen-antibody (immune) complex

• Defensive mechanisms used by antibodies are neutralization,

agglutination, precipitation, and complement fixation



Complement Fixation and Activation

• Complement fixation is the main mechanism used against cellular

antigens

• Antibodies bound to cells change shape and expose complement binding

sites

• This triggers complement fixation and cell lysis

• Complement activation:

– Enhances the inflammatory response

– Uses a positive feedback cycle to promote phagocytosis

– Enlists more and more defensive elements

Other Mechanisms of Antibody Action

• Neutralization –

antibodies bind to and block specific sites on viruses or exotoxins,

thus preventing these antigens from binding to receptors on

tissue cells



Other Mechanisms of Antibody Action

• Agglutination – antibodies bind the same determinant on more

than one antigen

– Makes antigen-antibody complexes that are cross-linked into large

lattices

– Cell-bound antigens are cross-linked, causing clumping (agglutination)

• Precipitation – soluble molecules are cross-linked into large

insoluble complexes



Mechanisms of Antibody Action



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