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Assumptions and Methods in Cognitive Neuropsychology

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Assumptions and Methods in Cognitive Neuropsychology



 Caitlin Turkiewicz and Darrick Chow

 What is cognitive neuropsychology

o Not concerned with treatments; can’t differentiate good treatments from bad

o About mind, but looking at the brain as a lens  about understanding mind

o Cognitive psychology is about the brain

o PALPA – assesses language disorders

 The case of AC

o Lesions in both hemispheres, history of cardiovascular difficulties

o Lost ability to write anything but name and address

o Could copy things, but not pictures

o Had difficulty with perceptual properties (eg: how many legs an animal had)

 Semantic information is not all or none

 Different parts of brain; processed differently (modular)

 What are the pieces that make up ‘mind’? How do they talk to each other?

 Modularity

o Fodor’s theory on modules (assumptions, not data)

 Hardwired: specific and localized

 Not all modules have all of the characteristics

 Not assembled: not made up of smaller sub-processes (minimum size for that

particular process)

 Informationally encapsulated: not penetrable from other kinds of information,

eg: representation of a word in terms of its spelling  semantic knowledge

doesn’t directly affect the unit (but can indirectly affect it)

 Look up what a ‘precis’ is

 Assumptions of Cognitive Neuropsychology

o These assumptions are required for neuropsychologists to conduct research

 Functional modularity: input modular system and central system (not

functionally modular), the modular systems are the easiest to study and thus

the ones we aught to pay attention to [what is in the mind]

 Anatomical Modularity – must exist in one specific area of the brain, not

multiple area, a simplifying assumption

 Uniformity of Functional Architecture Across People – like a flow chart (the

boxes of arrows of it), if not true, won’t be able to generalize across people

(note that he doesn’t assume it’s uniform across anatomical architecture)

 If false – if two people have very different results, cognitive professors

would know something was amiss (eg: reading nonwords faster than

words)

 Subtractivity – assume that brain damage can only subtract modules, not

introduce new ones (possible for patients to cope and use their normal

architecture abnormally, but uncommon)

o A question about Oliver Sacks will be on the quiz, google the crap out of him

o The definition of ‘harbinger’ will be on the quiz for a bonus point

o Ronald Fisher  father of anova, made a major impact

 Inferences from Data to Theory

o Use data to support or refute a theory

o 3 types of data abrain damaged person can provide

 Associations

o Could have different modules, but both might be affected

o Associations are not as important as disassociation

 Eg: impairment on one task but not another

 Dissociations

o Might not be good evidence

 One task might be harder for another

 Implausible, so not a serious issue

 Double dissociations

o The holy grail, best data you can have

o Rare to find perfect performance on one task and impairment on another

o May have double dissociations in the same patient

 Misunderstanding

o Nothing wrong with post-hoc analyseses, as long as leads to something new

 All science is originally post-hoc

o Cogneuropsychologists don’t think their theories are the only correct idea; welcome to

suggestions

 Syndromes

o An argument by association: if I have X, then I must have U

 Fine theories, but weak  if someone does not have U, there goes the theory

 Other Concerns with Cognitive Neuropsychology

o Need more than one patient with that pattern (over the years)

o Theories are too powerful: attempt to explain everything that is possible, however, they

are just trying to explain all observed data







Looking at groups of patients can get different results  slides, prof



Gave vs. have  need to know ‘have’ to know how to pronounce it



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